UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Plasminogen Activator Inhibitor-1 Is a Marker and a Mediator of Senescence

Arteriosclerosis, thrombosis, and vascular biology, 2017-08, Vol.37 (8), p.1446-1452

2017

Volltextzugriff (PDF)

MiR-125b Is Critical for Fibroblast-to-Myofibroblast Transition and Cardiac Fibrosis

Circulation (New York, N.Y.), 2016-01, Vol.133 (3), p.291-301

2016

Volltextzugriff (PDF)

Molecular basis of organ fibrosis: Potential therapeutic approaches

Experimental biology and medicine (Maywood, N.J.), 2013-05, Vol.238 (5), p.461-481

2013

Volltextzugriff (PDF)

Acetyltransferase p300 Is a Putative Epidrug Target for Amelioration of Cellular Aging-Related Cardiovascular Disease

Cells (Basel, Switzerland), 2021-10, Vol.10 (11), p.2839

Ghosh, Asish K

2021

Volltextzugriff (PDF)

The transcriptional coactivator and acetyltransferase p300 in fibroblast biology and fibrosis

Journal of cellular physiology, 2007-12, Vol.213 (3), p.663-671

2007

Volltextzugriff (PDF)

Acetyltransferase p300 inhibitor reverses hypertension‐induced cardiac fibrosis

Journal of cellular and molecular medicine, 2019-04, Vol.23 (4), p.3026-3031

2019

Volltextzugriff (PDF)

Context-dependent functions of angiopoietin 2 are determined by the endothelial phosphatase VEPTP

Proceedings of the National Academy of Sciences - PNAS, 2018-02, Vol.115 (6), p.1298-1303

2018

Volltextzugriff (PDF)

Molecular basis of cardiac endothelial-to-mesenchymal transition (EndMT): Differential expression of microRNAs during EndMT

Cellular signalling, 2012-05, Vol.24 (5), p.1031-1036

2012

Volltextzugriff (PDF)

A novel acetyltransferase p300 inhibitor ameliorates hypertension-associated cardio-renal fibrosis

Epigenetics, 2017-11, Vol.12 (11), p.1004-1013

2017

Volltextzugriff (PDF)

Global gene expression profiling in PAI-1 knockout murine heart and kidney: molecular basis of cardiac-selective fibrosis

PloS one, 2013-05, Vol.8 (5), p.e63825

2013

Volltextzugriff (PDF)

Downregulation of the Apelinergic Axis Accelerates Aging, whereas Its Systemic Restoration Improves the Mammalian Healthspan

Cell reports (Cambridge), 2017-11, Vol.21 (6), p.1471-1480

2017

Volltextzugriff (PDF)

Factors Involved in the Regulation of Type I Collagen Gene Expression: Implication in Fibrosis

Experimental biology and medicine (Maywood, N.J.), 2002-05, Vol.227 (5), p.301-314

Ghosh, Asish K.

2002

Volltextzugriff (PDF)

Use of Atrial Fibrillation Electrograms and T1/T2 Magnetic Resonance Imaging to Define the Progressive Nature of Molecular and Structural Remodeling: A New Paradigm Underlying the Emergence of Persistent Atrial Fibrillation

Journal of the American Heart Association, 2024-03, Vol.13 (5), p.e032514

2024

Volltextzugriff (PDF)

Curcumin prevents and reverses murine cardiac hypertrophy

The Journal of clinical investigation, 2008-03, Vol.118 (3), p.879-893

2008

Volltextzugriff (PDF)

Response to Letter Regarding Article, “MiR-125b Is Critical for Fibroblast-to-Myofibroblast Transition and Cardiac Fibrosis”

Circulation (New York, N.Y.), 2016-06, Vol.133 (24), p.e714-e714

2016

Volltextzugriff (PDF)

Epigenetics in Reactive and Reparative Cardiac Fibrogenesis: The Promise of Epigenetic Therapy

Journal of cellular physiology, 2017-08, Vol.232 (8), p.1941-1956

2017

Volltextzugriff (PDF)

p300 Is Elevated in Systemic Sclerosis and Its Expression Is Positively Regulated by TGF-β: Epigenetic Feed-Forward Amplification of Fibrosis

Journal of investigative dermatology, 2013-05, Vol.133 (5), p.1302-1310

2013

Volltextzugriff (PDF)

Interleukin-10 Treatment Attenuates Pressure Overload―Induced Hypertrophic Remodeling and Improves Heart Function via Signal Transducers and Activators of Transcription 3―Dependent Inhibition of Nuclear Factor-κB

Circulation (New York, N.Y.), 2012-07, Vol.126 (4), p.418-429

2012

Volltextzugriff (PDF)

FAT-Free p300 Is Good for Scar-Free Tissue Repair

Journal of cellular biochemistry, 2014-09, Vol.115 (9), p.1486-1489

Ghosh, Asish K.

2014

Volltextzugriff (PDF)

A lymphatic defect causes ocular hypertension and glaucoma in mice

The Journal of clinical investigation, 2014-10, Vol.124 (10), p.4320-4324

2014

Volltextzugriff (PDF)

The Tumor Suppressor p53 Abrogates Smad-dependent Collagen Gene Induction in Mesenchymal Cells

The Journal of biological chemistry, 2004-11, Vol.279 (46), p.47455-47463

2004

Volltextzugriff (PDF)

A small molecule inhibitor of PAI-1 protects against doxorubicin-induced cellular senescence

Oncotarget, 2016-11, Vol.7 (45), p.72443-72457

2016

Volltextzugriff (PDF)

Plasminogen Activator Inhibitor Type I Controls Cardiomyocyte Transforming Growth Factor-β and Cardiac Fibrosis

Circulation (New York, N.Y.), 2017-08, Vol.136 (7), p.664-679

2017

Volltextzugriff (PDF)

Rosiglitazone Abrogates Bleomycin-Induced Scleroderma and Blocks Profibrotic Responses Through Peroxisome Proliferator-Activated Receptor-γ

The American journal of pathology, 2009-02, Vol.174 (2), p.519-533

2009

Volltextzugriff (PDF)

Cardiomyocyte PAI-1 influences the cardiac transcriptome and limits the extent of cardiac fibrosis in response to left ventricular pressure overload

Cellular signalling, 2023-04, Vol.104, p.110555-110555, Article 110555

2023

Volltextzugriff (PDF)

1 – 25 von 63

Suche als RSS-Feed

Aktive Filter

ThemaAnimals

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(61)
Mit Volltextzugriff
(61)

Publikationsform

Artikel
(63)

Sprache

Englisch
(63)

Erscheinungsjahr

n.n

Thema

14 weitere anzeigen...

Weniger anzeigen...

Zeitschrift / Serie

5 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

Plasminogen Activator Inhibitor-1 Is a Marker and a Mediator of Senescence

MiR-125b Is Critical for Fibroblast-to-Myofibroblast Transition and Cardiac Fibrosis

Molecular basis of organ fibrosis: Potential therapeutic approaches

Acetyltransferase p300 Is a Putative Epidrug Target for Amelioration of Cellular Aging-Related Cardiovascular Disease

The transcriptional coactivator and acetyltransferase p300 in fibroblast biology and fibrosis

Acetyltransferase p300 inhibitor reverses hypertension‐induced cardiac fibrosis

Context-dependent functions of angiopoietin 2 are determined by the endothelial phosphatase VEPTP

Molecular basis of cardiac endothelial-to-mesenchymal transition (EndMT): Differential expression of microRNAs during EndMT

A novel acetyltransferase p300 inhibitor ameliorates hypertension-associated cardio-renal fibrosis

Global gene expression profiling in PAI-1 knockout murine heart and kidney: molecular basis of cardiac-selective fibrosis

Downregulation of the Apelinergic Axis Accelerates Aging, whereas Its Systemic Restoration Improves the Mammalian Healthspan

Factors Involved in the Regulation of Type I Collagen Gene Expression: Implication in Fibrosis

Use of Atrial Fibrillation Electrograms and T1/T2 Magnetic Resonance Imaging to Define the Progressive Nature of Molecular and Structural Remodeling: A New Paradigm Underlying the Emergence of Persistent Atrial Fibrillation

Curcumin prevents and reverses murine cardiac hypertrophy

Response to Letter Regarding Article, “MiR-125b Is Critical for Fibroblast-to-Myofibroblast Transition and Cardiac Fibrosis”

Epigenetics in Reactive and Reparative Cardiac Fibrogenesis: The Promise of Epigenetic Therapy

p300 Is Elevated in Systemic Sclerosis and Its Expression Is Positively Regulated by TGF-β: Epigenetic Feed-Forward Amplification of Fibrosis

Interleukin-10 Treatment Attenuates Pressure Overload―Induced Hypertrophic Remodeling and Improves Heart Function via Signal Transducers and Activators of Transcription 3―Dependent Inhibition of Nuclear Factor-κB

FAT-Free p300 Is Good for Scar-Free Tissue Repair

A lymphatic defect causes ocular hypertension and glaucoma in mice

The Tumor Suppressor p53 Abrogates Smad-dependent Collagen Gene Induction in Mesenchymal Cells

A small molecule inhibitor of PAI-1 protects against doxorubicin-induced cellular senescence

Plasminogen Activator Inhibitor Type I Controls Cardiomyocyte Transforming Growth Factor-β and Cardiac Fibrosis

Rosiglitazone Abrogates Bleomycin-Induced Scleroderma and Blocks Profibrotic Responses Through Peroxisome Proliferator-Activated Receptor-γ

Cardiomyocyte PAI-1 influences the cardiac transcriptome and limits the extent of cardiac fibrosis in response to left ventricular pressure overload

Aktive Filter

Suchergebnisse filtern