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CD4 T cell help prevents CD8 T cell exhaustion and promotes control of Mycobacterium tuberculosis infection

Cell reports (Cambridge), 2021-09, Vol.36 (11), p.109696-109696, Article 109696

2021

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Evasion of innate immunity by Mycobacterium tuberculosis: is death an exit strategy?

Nature reviews. Microbiology, 2010-09, Vol.8 (9), p.668-674

2010

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TIM3 Mediates T Cell Exhaustion during Mycobacterium tuberculosis Infection

PLoS pathogens, 2016-03, Vol.12 (3), p.e1005490-e1005490

2016

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Efferocytosis Is an Innate Antibacterial Mechanism

Cell host & microbe, 2012-09, Vol.12 (3), p.289-300

2012

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Emerging Tim-3 functions in antimicrobial and tumor immunity

Trends in immunology, 2011-08, Vol.32 (8), p.345-349

2011

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iNKT cell production of GM-CSF controls Mycobacterium tuberculosis

PLoS pathogens, 2014-01, Vol.10 (1), p.e1003805-e1003805

2014

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Eicosanoid pathways regulate adaptive immunity to Mycobacterium tuberculosis

Nature immunology, 2010-08, Vol.11 (8), p.751-758

2010

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Mycobacterium tuberculosis-specific CD4+ and CD8+ T cells differ in their capacity to recognize infected macrophages

PLoS pathogens, 2018-05, Vol.14 (5), p.e1007060-e1007060

2018

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Tuberculosis Susceptibility and Vaccine Protection Are Independently Controlled by Host Genotype

mBio, 2016-09, Vol.7 (5)

2016

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Host genetic background is a barrier to broadly effective vaccine-mediated protection against tuberculosis

The Journal of clinical investigation, 2023-07, Vol.133 (13), p.1-16

2023

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A natural polymorphism of Mycobacterium tuberculosis in the esxH gene disrupts immunodomination by the TB10.4-specific CD8 T cell response

PLoS pathogens, 2020-10, Vol.16 (10), p.e1009000-e1009000

2020

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Tim3 binding to galectin-9 stimulates antimicrobial immunity

The Journal of experimental medicine, 2010-10, Vol.207 (11), p.2343-2354

2010

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Innate invariant NKT cells recognize Mycobacterium tuberculosis-infected macrophages, produce interferon-gamma, and kill intracellular bacteria

PLoS pathogens, 2008-12, Vol.4 (12), p.e1000239-e1000239

2008

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A Higher Activation Threshold of Memory CD8+ T Cells Has a Fitness Cost That Is Modified by TCR Affinity during Tuberculosis

PLoS pathogens, 2016-01, Vol.12 (1), p.e1005380-e1005380

2016

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CD11cHi monocyte-derived macrophages are a major cellular compartment infected by Mycobacterium tuberculosis

PLoS pathogens, 2020-06, Vol.16 (6), p.e1008621-e1008621

2020

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Regulation of neutrophils by interferon-γ limits lung inflammation during tuberculosis infection

The Journal of experimental medicine, 2011-10, Vol.208 (11), p.2251-2262

2011

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Recognition of microbial and mammalian phospholipid antigens by NKT cells with diverse TCRs

Proceedings of the National Academy of Sciences - PNAS, 2013-01, Vol.110 (5), p.1827-1832

2013

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Tuberculosis Triggers a Tissue-Dependent Program of Differentiation and Acquisition of Effector Functions by Circulating Monocytes

The Journal of immunology (1950), 2008-11, Vol.181 (9), p.6349-6360

2008

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Vaccine-elicited memory CD4+ T cell expansion is impaired in the lungs during tuberculosis

PLoS pathogens, 2017-11, Vol.13 (11), p.e1006704-e1006704

2017

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Mitochondrial respiration contributes to the interferon gamma response in antigen-presenting cells

eLife, 2021-11, Vol.10

2021

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Identification and characterization of the T cell receptor (TCR) repertoire of the cynomolgus macaque (Macaca Fascicularis)

BMC genomics, 2022-09, Vol.23 (1), p.647-647, Article 647

2022

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Functionally Overlapping Variants Control Tuberculosis Susceptibility in Collaborative Cross Mice

mBio, 2019-11, Vol.10 (6)

2019

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IFNγ and iNOS-Mediated Alterations in the Bone Marrow and Thymus and Its Impact on Mycobacterium avium -Induced Thymic Atrophy

Frontiers in immunology, 2021-12, Vol.12, p.696415-696415

2021

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Role of CD1d-restricted NKT cells in microbial immunity

Infection and immunity, 2003-10, Vol.71 (10), p.5447-5455

2003

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Human and Murine Clonal CD8+ T Cell Expansions Arise during Tuberculosis Because of TCR Selection

PLoS pathogens, 2015-05, Vol.11 (5), p.e1004849-e1004849

2015

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CD4 T cell help prevents CD8 T cell exhaustion and promotes control of Mycobacterium tuberculosis infection

Evasion of innate immunity by Mycobacterium tuberculosis: is death an exit strategy?

TIM3 Mediates T Cell Exhaustion during Mycobacterium tuberculosis Infection

Efferocytosis Is an Innate Antibacterial Mechanism

Emerging Tim-3 functions in antimicrobial and tumor immunity

iNKT cell production of GM-CSF controls Mycobacterium tuberculosis

Eicosanoid pathways regulate adaptive immunity to Mycobacterium tuberculosis

Mycobacterium tuberculosis-specific CD4+ and CD8+ T cells differ in their capacity to recognize infected macrophages

Tuberculosis Susceptibility and Vaccine Protection Are Independently Controlled by Host Genotype

Host genetic background is a barrier to broadly effective vaccine-mediated protection against tuberculosis

A natural polymorphism of Mycobacterium tuberculosis in the esxH gene disrupts immunodomination by the TB10.4-specific CD8 T cell response

Tim3 binding to galectin-9 stimulates antimicrobial immunity

Innate invariant NKT cells recognize Mycobacterium tuberculosis-infected macrophages, produce interferon-gamma, and kill intracellular bacteria

A Higher Activation Threshold of Memory CD8+ T Cells Has a Fitness Cost That Is Modified by TCR Affinity during Tuberculosis

CD11cHi monocyte-derived macrophages are a major cellular compartment infected by Mycobacterium tuberculosis

Regulation of neutrophils by interferon-γ limits lung inflammation during tuberculosis infection

Recognition of microbial and mammalian phospholipid antigens by NKT cells with diverse TCRs

Tuberculosis Triggers a Tissue-Dependent Program of Differentiation and Acquisition of Effector Functions by Circulating Monocytes

Vaccine-elicited memory CD4+ T cell expansion is impaired in the lungs during tuberculosis

Mitochondrial respiration contributes to the interferon gamma response in antigen-presenting cells

Identification and characterization of the T cell receptor (TCR) repertoire of the cynomolgus macaque (Macaca Fascicularis)

Functionally Overlapping Variants Control Tuberculosis Susceptibility in Collaborative Cross Mice

IFNγ and iNOS-Mediated Alterations in the Bone Marrow and Thymus and Its Impact on Mycobacterium avium -Induced Thymic Atrophy

Role of CD1d-restricted NKT cells in microbial immunity

Human and Murine Clonal CD8+ T Cell Expansions Arise during Tuberculosis Because of TCR Selection

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