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STING Recognition of Cytoplasmic DNA Instigates Cellular Defense

Molecular cell, 2013-04, Vol.50 (1), p.5-15

2013

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Deregulation of STING Signaling in Colorectal Carcinoma Constrains DNA Damage Responses and Correlates With Tumorigenesis

Cell reports (Cambridge), 2016-01, Vol.14 (2), p.282-297

2016

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Extrinsic Phagocyte-Dependent STING Signaling Dictates the Immunogenicity of Dying Cells

Cancer cell, 2018-05, Vol.33 (5), p.862-873.e5

2018

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STING-Dependent Signaling Underlies IL-10 Controlled Inflammatory Colitis

Cell reports (Cambridge), 2017-12, Vol.21 (13), p.3873-3884

2017

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Suppression of STING signaling through epigenetic silencing and missense mutation impedes DNA damage mediated cytokine production

Oncogene, 2018-04, Vol.37 (15), p.2037-2051

2018

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Ovarian Cancer Cells Commonly Exhibit Defective STING Signaling Which Affects Sensitivity to Viral Oncolysis

Molecular cancer research, 2019-04, Vol.17 (4), p.974-986

2019

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STING pathway and regulation of innate immune signaling in response to DNA pathogens

Cellular and molecular life sciences : CMLS, 2011-04, Vol.68 (7), p.1157-1165

2011

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STING signalling is terminated through ESCRT-dependent microautophagy of vesicles originating from recycling endosomes

Nature cell biology, 2023-03, Vol.25 (3), p.453-466

2023

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Pro-inflammation Associated with a Gain-of-Function Mutation (R284S) in the Innate Immune Sensor STING

Cell reports (Cambridge), 2018-04, Vol.23 (4), p.1112-1123

2018

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Phosphorylation of the NFAR proteins by the dsRNA-dependent protein kinase PKR constitutes a novel mechanism of translational regulation and cellular defense

Genes & development, 2010-12, Vol.24 (23), p.2640-2653

2010

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Bacterial c-di-GMP Affects Hematopoietic Stem/Progenitors and Their Niches through STING

Cell reports (Cambridge), 2015-04, Vol.11 (1), p.71-84

2015

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VSV Disrupts the Rae1/mrnp41 mRNA Nuclear Export Pathway

Molecular cell, 2005-01, Vol.17 (1), p.93-102

2005

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Exogenous non-coding dsDNA-dependent trans-activation of phagocytes augments anti-tumor immunity

Cell reports. Medicine, 2024-05, Vol.5 (5), p.101528-101528, Article 101528

2024

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The dsRNA binding protein family: critical roles, diverse cellular functions

The FASEB journal, 2003-06, Vol.17 (9), p.961-983

2003

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Defects in Translational Regulation Mediated by the α Subunit of Eukaryotic Initiation Factor 2 Inhibit Antiviral Activity and Facilitate the Malignant Transformation of Human Fibroblasts

Molecular and Cellular Biology, 2004-03, Vol.24 (5), p.2025-2040

2004

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Vesicular stomatitis virus inhibits mitotic progression and triggers cell death

EMBO reports, 2009-10, Vol.10 (10), p.1154-1160

2009

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Defective translational control facilitates vesicular stomatitis virus oncolysis

Cancer cell, 2004, Vol.5 (1), p.51-65

2004

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Purging metastases in lymphoid organs using a combination of antigen-nonspecific adoptive T cell therapy, oncolytic virotherapy and immunotherapy

Nature medicine, 2008-01, Vol.14 (1), p.37-44

2008

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Activation of the dsRNA-dependent protein kinase, PKR, induces apoptosis through FADD-mediated death signaling

The EMBO journal, 1998-12, Vol.17 (23), p.6888-6902

1998

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PKR, apoptosis and cancer

International Journal of Biochemistry and Cell Biology, 1999, Vol.31 (1), p.123-138

1999

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Vesicular stomatitis virus (VSV) therapy of tumors

IUBMB life, 2000-08, Vol.50 (2), p.135-138

2000

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Mutants of the RNA-dependent protein kinase (PKR) lacking double-stranded RNA binding domain I can act as transdominant inhibitors and induce malignant transformation

Molecular and Cellular Biology, 1995-06, Vol.15 (6), p.3138-3146

1995

Volltextzugriff (PDF)

Functional expression and RNA binding analysis of the interferon-induced, double-stranded RNA-activated, 68,000-Mr protein kinase in a cell-free system

Molecular and Cellular Biology, 1991-11, Vol.11 (11), p.5497-5505

1991

Volltextzugriff (PDF)

The Murine PKR Tumor Suppressor Gene Is Rearranged in a Lymphocytic Leukemia

Experimental cell research, 1998-11, Vol.244 (2), p.394-404

1998

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STING Recognition of Cytoplasmic DNA Instigates Cellular Defense

Deregulation of STING Signaling in Colorectal Carcinoma Constrains DNA Damage Responses and Correlates With Tumorigenesis

Extrinsic Phagocyte-Dependent STING Signaling Dictates the Immunogenicity of Dying Cells

STING-Dependent Signaling Underlies IL-10 Controlled Inflammatory Colitis

Suppression of STING signaling through epigenetic silencing and missense mutation impedes DNA damage mediated cytokine production

Ovarian Cancer Cells Commonly Exhibit Defective STING Signaling Which Affects Sensitivity to Viral Oncolysis

STING pathway and regulation of innate immune signaling in response to DNA pathogens

STING signalling is terminated through ESCRT-dependent microautophagy of vesicles originating from recycling endosomes

Pro-inflammation Associated with a Gain-of-Function Mutation (R284S) in the Innate Immune Sensor STING

Phosphorylation of the NFAR proteins by the dsRNA-dependent protein kinase PKR constitutes a novel mechanism of translational regulation and cellular defense

Bacterial c-di-GMP Affects Hematopoietic Stem/Progenitors and Their Niches through STING

VSV Disrupts the Rae1/mrnp41 mRNA Nuclear Export Pathway

Exogenous non-coding dsDNA-dependent trans-activation of phagocytes augments anti-tumor immunity

The dsRNA binding protein family: critical roles, diverse cellular functions

Defects in Translational Regulation Mediated by the α Subunit of Eukaryotic Initiation Factor 2 Inhibit Antiviral Activity and Facilitate the Malignant Transformation of Human Fibroblasts

Vesicular stomatitis virus inhibits mitotic progression and triggers cell death

Defective translational control facilitates vesicular stomatitis virus oncolysis

Purging metastases in lymphoid organs using a combination of antigen-nonspecific adoptive T cell therapy, oncolytic virotherapy and immunotherapy

Activation of the dsRNA-dependent protein kinase, PKR, induces apoptosis through FADD-mediated death signaling

PKR, apoptosis and cancer

Vesicular stomatitis virus (VSV) therapy of tumors

Mutants of the RNA-dependent protein kinase (PKR) lacking double-stranded RNA binding domain I can act as transdominant inhibitors and induce malignant transformation

Functional expression and RNA binding analysis of the interferon-induced, double-stranded RNA-activated, 68,000-Mr protein kinase in a cell-free system

The Murine PKR Tumor Suppressor Gene Is Rearranged in a Lymphocytic Leukemia

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