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Dephosphorylation of receptor tyrosine kinases as target of regulation by radiation, oxidants or alkylating agents

The EMBO journal, 1996-10, Vol.15 (19), p.5314-5325

1996

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A distinct modulating domain in glucocorticoid receptor monomers in the repression of activity of the transcription factor AP‐1

The EMBO journal, 1994-09, Vol.13 (17), p.4087-4095

1994

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DNA damage induced p53 stabilization : no indication for an involvement of p53 phosphorylation

Oncogene, 1999-03, Vol.18 (9), p.1723-1732

1999

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UV-induced DNA damage is an intermediate step in UV-induced expression of human immunodeficiency virus type 1, collagenase, c-fos, and metallothionein

Molecular and Cellular Biology, 1989-11, Vol.9 (11), p.5169-5181

1989

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CREB is activated by UVC through a p38/HOG-1-dependent protein kinase

The EMBO journal, 1997-03, Vol.16 (5), p.1009-1022

1997

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Antitumor promotion and antiinflammation: Down-modulation of AP-1 (Fos/Jun) activity by glucocorticoid hormone

Cell, 1990-09, Vol.62 (6), p.1189-1204

1990

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12-O-tetradecanoyl-phorbol-13-acetate induction of the human collagenase gene is mediated by an inducible enhancer element located in the 5'-flanking region

Molecular and Cellular Biology, 1987-06, Vol.7 (6), p.2256-2266

1987

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UV-induced signal transduction

Journal of Photochemistry & Photobiology, B: Biology, 1997, Vol.37 (1), p.1-17

1997

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UV‐induced activation of AP‐1 involves obligatory extranuclear steps including Raf‐1 kinase

The EMBO journal, 1993-03, Vol.12 (3), p.1005-1012

1993

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Interference between pathway‐specific transcription factors: glucocorticoids antagonize phorbol ester‐induced AP‐1 activity without altering AP‐1 site occupation in vivo

The EMBO journal, 1992-06, Vol.11 (6), p.2241-2246

1992

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UV irradiation-induced interleukin-1 and basic fibroblast growth factor synthesis and release mediate part of the UV response

The Journal of biological chemistry, 1993-03, Vol.268 (9), p.6734-6741

1993

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Phorbol ester-inducible genes contain a common cis element recognized by a TPA-modulated trans-acting factor

Cell, 1987-06, Vol.49 (6), p.729-739

1987

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Requirement for fos gene expression in the transcriptional activation of collagenase by other oncogenes and phorbol esters

Cell, 1988-07, Vol.54 (3), p.325-334

1988

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Comparison of human stromelysin and collagenase by cloning and sequence analysis

Biochemical journal, 1986-12, Vol.240 (3), p.913-916

1986

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Autoregulation of fos: the dyad symmetry element as the major target of repression

The EMBO journal, 1989-09, Vol.8 (9), p.2559-2566

1989

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Photoproducts in transcriptionally active DNA induce signal transduction to the delayed U.V.-responsive genes for collagenase and metallothionein

Oncogene, 1998-06, Vol.16 (22), p.2827-2834

1998

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Activation of the c-fos gene by UV and phorbol ester: different signal transduction pathways converge to the same enhancer element

Oncogene, 1988-09, Vol.3 (3), p.301

1988

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UV-induced transcription from the human immunodeficiency virus type 1 (HIV-1) long terminal repeat and UV-induced secretion of an extracellular factor that induces HIV-1 transcription in nonirradiated cells

Journal of Virology, 1989-11, Vol.63 (11), p.4540-4544

1989

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Involvement of growth factor receptors in the mammalian UVC response

Cell, 1994-09, Vol.78 (6), p.963-972

1994

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UV-Induced Stabilization of c-fos and Other Short-Lived mRNAs

Molecular and Cellular Biology, 2000-05, Vol.20 (10), p.3616-3625

2000

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Induction of metallothionein and other mRNA species by carcinogens and tumor promoters in primary human skin fibroblasts

Molecular and Cellular Biology, 1986-05, Vol.6 (5), p.1760-1766

1986

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Damage to DNA by UV light and activation of transcription factors

Biochemical pharmacology, 1994-01, Vol.47 (1), p.129-136

1994

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Regulation of gene expression by tumor promoters

Pharmacology & therapeutics (Oxford), 1990, Vol.48 (2), p.157-188

1990

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Overexpression of c-fos increases recombination frequency in human osteosarcoma cells

Carcinogenesis (New York), 1993-05, Vol.14 (5), p.925-928

1993

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The Fos and Jun/AP-1 proteins are involved in the downregulation of Fos transcription

Oncogene, 1989-05, Vol.4 (5), p.629-636

1989

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Dephosphorylation of receptor tyrosine kinases as target of regulation by radiation, oxidants or alkylating agents

A distinct modulating domain in glucocorticoid receptor monomers in the repression of activity of the transcription factor AP‐1

DNA damage induced p53 stabilization : no indication for an involvement of p53 phosphorylation

UV-induced DNA damage is an intermediate step in UV-induced expression of human immunodeficiency virus type 1, collagenase, c-fos, and metallothionein

CREB is activated by UVC through a p38/HOG-1-dependent protein kinase

Antitumor promotion and antiinflammation: Down-modulation of AP-1 (Fos/Jun) activity by glucocorticoid hormone

12-O-tetradecanoyl-phorbol-13-acetate induction of the human collagenase gene is mediated by an inducible enhancer element located in the 5'-flanking region

UV-induced signal transduction

UV‐induced activation of AP‐1 involves obligatory extranuclear steps including Raf‐1 kinase

Interference between pathway‐specific transcription factors: glucocorticoids antagonize phorbol ester‐induced AP‐1 activity without altering AP‐1 site occupation in vivo

UV irradiation-induced interleukin-1 and basic fibroblast growth factor synthesis and release mediate part of the UV response

Phorbol ester-inducible genes contain a common cis element recognized by a TPA-modulated trans-acting factor

Requirement for fos gene expression in the transcriptional activation of collagenase by other oncogenes and phorbol esters

Comparison of human stromelysin and collagenase by cloning and sequence analysis

Autoregulation of fos: the dyad symmetry element as the major target of repression

Photoproducts in transcriptionally active DNA induce signal transduction to the delayed U.V.-responsive genes for collagenase and metallothionein

Activation of the c-fos gene by UV and phorbol ester: different signal transduction pathways converge to the same enhancer element

UV-induced transcription from the human immunodeficiency virus type 1 (HIV-1) long terminal repeat and UV-induced secretion of an extracellular factor that induces HIV-1 transcription in nonirradiated cells

Involvement of growth factor receptors in the mammalian UVC response

UV-Induced Stabilization of c-fos and Other Short-Lived mRNAs

Induction of metallothionein and other mRNA species by carcinogens and tumor promoters in primary human skin fibroblasts

Damage to DNA by UV light and activation of transcription factors

Regulation of gene expression by tumor promoters

Overexpression of c-fos increases recombination frequency in human osteosarcoma cells

The Fos and Jun/AP-1 proteins are involved in the downregulation of Fos transcription

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