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STING-dependent paracriny shapes apoptotic priming of breast tumors in response to anti-mitotic treatment

Nature communications, 2020-01, Vol.11 (1), p.259-259, Article 259

2020

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Interactions between cancer-associated fibroblasts and tumor cells promote MCL-1 dependency in estrogen receptor-positive breast cancers

Oncogene, 2019-04, Vol.38 (17), p.3261-3273

2019

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Low BCL-xL expression in triple-negative breast cancer cells favors chemotherapy efficacy, and this effect is limited by cancer-associated fibroblasts

Scientific reports, 2024-06, Vol.14 (1), p.14177-10, Article 14177

2024

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Stress in the metastatic journey - the role of cell communication and clustering in breast cancer progression and treatment resistance

Disease models & mechanisms, 2024-03, Vol.17 (3)

2024

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Targeting of MCL-1 in breast cancer-associated fibroblasts reverses their myofibroblastic phenotype and pro-invasive properties

Cell death & disease, 2022-09, Vol.13 (9), p.787-787, Article 787

2022

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miR-491-5p-induced apoptosis in ovarian carcinoma depends on the direct inhibition of both BCL-XL and EGFR leading to BIM activation

Cell death & disease, 2014-10, Vol.5 (10), p.e1445-e1445

2014

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Targeting of BCL-2 Family Members during Anticancer Treatment: A Necessary Compromise between Individual Cell and Ecosystemic Responses?

Biomolecules (Basel, Switzerland), 2020-07, Vol.10 (8), p.1109

2020

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Tight Sequestration of BH3 Proteins by BCL-xL at Subcellular Membranes Contributes to Apoptotic Resistance

Cell reports (Cambridge), 2016-12, Vol.17 (12), p.3347-3358

2016

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bc-GenExMiner 4.5: new mining module computes breast cancer differential gene expression analyses

Database : the journal of biological databases and curation, 2021-02, Vol.2021

2021

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DNA methylation signal has a major role in the response of human breast cancer cells to the microenvironment

Oncogenesis (New York, NY), 2017-10, Vol.6 (10), p.e390-e390

2017

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Constitutive p53 heightens mitochondrial apoptotic priming and favors cell death induction by BH3 mimetic inhibitors of BCL-xL

Cell death & disease, 2016-02, Vol.7 (2), p.e2083-e2083

2016

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An EMT-primary cilium-GLIS2 signaling axis regulates mammogenesis and claudin-low breast tumorigenesis

Science advances, 2021-10, Vol.7 (44), p.eabf6063

2021

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Mitotic stress-induced secretome primes cancer cells to apoptosis and maximizes paclitaxel response in breast tumors when combined with BCL-xL-targeting BH3 mimetics

Molecular & cellular oncology, 2020-05, Vol.7 (3), p.1735912-1735912

2020

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TOM22, a core component of the mitochondria outer membrane protein translocation pore, is a mitochondrial receptor for the proapoptotic protein Bax

Cell death and differentiation, 2007-04, Vol.14 (4), p.785-794

2007

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Bcl-xL controls a switch between cell death modes during mitotic arrest

Cell death & disease, 2014-06, Vol.5 (6), p.e1291-e1291

2014

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c-Myc-induced sensitization to apoptosis is mediated through cytochrome c release

Genes & development, 1999-06, Vol.13 (11), p.1367-1381

1999

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Bax Activation by Engagement with, Then Release from, the BH3 Binding Site of Bcl-xL

Molecular and Cellular Biology, 2011-02, Vol.31 (4), p.832-844

2011

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Mitochondria at Center of Exchanges between Cancer Cells and Cancer-Associated Fibroblasts during Tumor Progression

Cancers, 2020-10, Vol.12 (10), p.3017

2020

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RE: Immune Checkpoint Profiles in Luminal B Breast Cancer (Alliance)

JNCI : Journal of the National Cancer Institute, 2020-08, Vol.112 (8), p.863-864

2020

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pRb/E2F-1-mediated caspase-dependent induction of Noxa amplifies the apoptotic effects of the Bcl-2/Bcl-xL inhibitor ABT-737

Cell death and differentiation, 2013-05, Vol.20 (5), p.755-764

2013

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Sensitization of osteosarcoma cells to apoptosis by oncostatin M depends on STAT5 and p53

Oncogene, 2007-10, Vol.26 (46), p.6653-6664

2007

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Mitochondrial targeting by use of lipid nanocapsules loaded with SV30, an analogue of the small-molecule Bcl-2 inhibitor HA14-1

Journal of controlled release, 2011-04, Vol.151 (1), p.74-82

2011

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EMT and primary ciliogenesis: For better or worse in sickness and in health

Genesis (New York, N.Y. : 2000), 2024-02, Vol.62 (1), p.e23568-n/a

2024

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Constitutive p53 heightens mitochondrial apoptotic priming and favors cell death induction by BH3 mimetic inhibitors of BCL-xL

Cell death & disease, 2017-02, Vol.8 (2), p.e2596-e2596

2017

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STING-dependent paracriny shapes apoptotic priming of breast tumors in response to anti-mitotic treatment

Interactions between cancer-associated fibroblasts and tumor cells promote MCL-1 dependency in estrogen receptor-positive breast cancers

Low BCL-xL expression in triple-negative breast cancer cells favors chemotherapy efficacy, and this effect is limited by cancer-associated fibroblasts

Stress in the metastatic journey - the role of cell communication and clustering in breast cancer progression and treatment resistance

Targeting of MCL-1 in breast cancer-associated fibroblasts reverses their myofibroblastic phenotype and pro-invasive properties

miR-491-5p-induced apoptosis in ovarian carcinoma depends on the direct inhibition of both BCL-XL and EGFR leading to BIM activation

Targeting of BCL-2 Family Members during Anticancer Treatment: A Necessary Compromise between Individual Cell and Ecosystemic Responses?

Tight Sequestration of BH3 Proteins by BCL-xL at Subcellular Membranes Contributes to Apoptotic Resistance

bc-GenExMiner 4.5: new mining module computes breast cancer differential gene expression analyses

DNA methylation signal has a major role in the response of human breast cancer cells to the microenvironment

Constitutive p53 heightens mitochondrial apoptotic priming and favors cell death induction by BH3 mimetic inhibitors of BCL-xL

An EMT-primary cilium-GLIS2 signaling axis regulates mammogenesis and claudin-low breast tumorigenesis

Mitotic stress-induced secretome primes cancer cells to apoptosis and maximizes paclitaxel response in breast tumors when combined with BCL-xL-targeting BH3 mimetics

TOM22, a core component of the mitochondria outer membrane protein translocation pore, is a mitochondrial receptor for the proapoptotic protein Bax

Bcl-xL controls a switch between cell death modes during mitotic arrest

Keeping Cell Death Alive: An Introduction into the French Cell Death Research Network

c-Myc-induced sensitization to apoptosis is mediated through cytochrome c release

Bax Activation by Engagement with, Then Release from, the BH3 Binding Site of Bcl-xL

Mitochondria at Center of Exchanges between Cancer Cells and Cancer-Associated Fibroblasts during Tumor Progression

RE: Immune Checkpoint Profiles in Luminal B Breast Cancer (Alliance)

pRb/E2F-1-mediated caspase-dependent induction of Noxa amplifies the apoptotic effects of the Bcl-2/Bcl-xL inhibitor ABT-737

Sensitization of osteosarcoma cells to apoptosis by oncostatin M depends on STAT5 and p53

Mitochondrial targeting by use of lipid nanocapsules loaded with SV30, an analogue of the small-molecule Bcl-2 inhibitor HA14-1

EMT and primary ciliogenesis: For better or worse in sickness and in health

Constitutive p53 heightens mitochondrial apoptotic priming and favors cell death induction by BH3 mimetic inhibitors of BCL-xL

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