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The Two Faces of Receptor Interacting Protein Kinase-1

Molecular cell, 2014-11, Vol.56 (4), p.469-480

2014

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Pattern Recognition Receptors and the Host Cell Death Molecular Machinery

Frontiers in immunology, 2018-10, Vol.9, p.2379-2379

2018

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Catalytic activity of the caspase―8―FLIPL complex inhibits RIPK3-dependent necrosis

Nature (London), 2011-03, Vol.471 (7338), p.363-367

2011

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FADD and caspase-8 mediate priming and activation of the canonical and noncanonical Nlrp3 inflammasomes

The Journal of immunology (1950), 2014-02, Vol.192 (4), p.1835-1846

2014

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Synchronized renal tubular cell death involves ferroptosis

Proceedings of the National Academy of Sciences - PNAS, 2014-11, Vol.111 (47), p.16836-16841

2014

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Two independent pathways of regulated necrosis mediate ischemia–reperfusion injury

Proceedings of the National Academy of Sciences - PNAS, 2013-07, Vol.110 (29), p.12024-12029

2013

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RIPK1 Blocks Early Postnatal Lethality Mediated by Caspase-8 and RIPK3

Cell, 2014-05, Vol.157 (5), p.1189-1202

2014

Volltextzugriff (PDF)

Frontline Science: Autophagy is a cell autonomous effector mechanism mediated by NLRP3 to control Trypanosoma cruzi infection

Journal of leukocyte biology, 2019-09, Vol.106 (3), p.531-540

2019

Volltextzugriff (PDF)

Higher Mixed lineage Kinase Domain-like protein (MLKL) is associated with worst overall survival in adult-type diffuse glioma patients

PloS one, 2023-08, Vol.18 (8), p.e0291019-e0291019

2023

Volltextzugriff (PDF)

The interaction between RIPK1 and FADD controls perinatal lethality and inflammation

Cell reports (Cambridge), 2024-06, Vol.43 (6), p.114335-114335, Article 114335

2024

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Protective Roles for Caspase-8 and cFLIP in Adult Homeostasis

Cell reports (Cambridge), 2013-10, Vol.5 (2), p.340-348

2013

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A Novel Cytotoxic Sequence Contributes to Influenza A Viral Protein PB1-F2 Pathogenicity and Predisposition to Secondary Bacterial Infection

Journal of Virology, 2014-01, Vol.88 (1), p.503-515

2014

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Comparison of 2D and 3D cell culture models for cell growth, gene expression and drug resistance

Materials Science & Engineering C, 2020-02, Vol.107, p.110264-110264, Article 110264

2020

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Necroptosis in development, inflammation and disease

Nature reviews. Molecular cell biology, 2017-02, Vol.18 (2), p.127

2017

Volltextzugriff (PDF)

Higher Mixed lineage Kinase Domain-like protein

PloS one, 2023-08, Vol.18 (8), p.e0291019

2023

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C11orf95-RELA fusions drive oncogenic NF-κB signalling in ependymoma

Nature (London), 2014-02, Vol.506 (7489), p.451-455

2014

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Survival function of the FADD-CASPASE-8-cFLIP(L) complex

Cell reports (Cambridge), 2012-05, Vol.1 (5), p.401

2012

Volltextzugriff (PDF)

Ex vivo gene therapy for lysosomal storage disorders: future perspectives

Expert opinion on biological therapy, 2023-04, Vol.23 (4), p.353-364

2023

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TNF-mediated alveolar macrophage necroptosis drives disease pathogenesis during respiratory syncytial virus infection

The European respiratory journal, 2021-06, Vol.57 (6), p.2003764

2021

Volltextzugriff (PDF)

RIPK-Dependent Necrosis and Its Regulation by Caspases: A Mystery in Five Acts

Molecular cell, 2011-10, Vol.44 (1), p.9-16

2011

Volltextzugriff (PDF)

A Dual Role of Caspase-8 in Triggering and Sensing Proliferation-Associated DNA Damage, a Key Determinant of Liver Cancer Development

Cancer cell, 2017-09, Vol.32 (3), p.342-359.e10

2017

Volltextzugriff (PDF)

RIPK3 is a novel prognostic marker for lower grade glioma and further enriches IDH mutational status subgrouping

Journal of neuro-oncology, 2020-05, Vol.147 (3), p.587-594

2020

Volltextzugriff (PDF)

The impairment in the NLRP3‐induced NO secretion renders astrocytes highly permissive to T. cruzi replication

Journal of leukocyte biology, 2019-07, Vol.106 (1), p.201-207

2019

Volltextzugriff (PDF)

Ripped to death

Trends in cell biology, 2011-11, Vol.21 (11), p.630-637

2011

Volltextzugriff (PDF)

Myeloid-Derived Suppressor Activity Is Mediated by Monocytic Lineages Maintained by Continuous Inhibition of Extrinsic and Intrinsic Death Pathways

Immunity (Cambridge, Mass.), 2014-12, Vol.41 (6), p.947-959

2014

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The Two Faces of Receptor Interacting Protein Kinase-1

Pattern Recognition Receptors and the Host Cell Death Molecular Machinery

Catalytic activity of the caspase―8―FLIPL complex inhibits RIPK3-dependent necrosis

FADD and caspase-8 mediate priming and activation of the canonical and noncanonical Nlrp3 inflammasomes

Synchronized renal tubular cell death involves ferroptosis

Two independent pathways of regulated necrosis mediate ischemia–reperfusion injury

RIPK1 Blocks Early Postnatal Lethality Mediated by Caspase-8 and RIPK3

Frontline Science: Autophagy is a cell autonomous effector mechanism mediated by NLRP3 to control Trypanosoma cruzi infection

Higher Mixed lineage Kinase Domain-like protein (MLKL) is associated with worst overall survival in adult-type diffuse glioma patients

The interaction between RIPK1 and FADD controls perinatal lethality and inflammation

Protective Roles for Caspase-8 and cFLIP in Adult Homeostasis

A Novel Cytotoxic Sequence Contributes to Influenza A Viral Protein PB1-F2 Pathogenicity and Predisposition to Secondary Bacterial Infection

Comparison of 2D and 3D cell culture models for cell growth, gene expression and drug resistance

Necroptosis in development, inflammation and disease

Higher Mixed lineage Kinase Domain-like protein

C11orf95-RELA fusions drive oncogenic NF-κB signalling in ependymoma

Survival function of the FADD-CASPASE-8-cFLIP(L) complex

Ex vivo gene therapy for lysosomal storage disorders: future perspectives

TNF-mediated alveolar macrophage necroptosis drives disease pathogenesis during respiratory syncytial virus infection

RIPK-Dependent Necrosis and Its Regulation by Caspases: A Mystery in Five Acts

A Dual Role of Caspase-8 in Triggering and Sensing Proliferation-Associated DNA Damage, a Key Determinant of Liver Cancer Development

RIPK3 is a novel prognostic marker for lower grade glioma and further enriches IDH mutational status subgrouping

The impairment in the NLRP3‐induced NO secretion renders astrocytes highly permissive to T. cruzi replication

Ripped to death

Myeloid-Derived Suppressor Activity Is Mediated by Monocytic Lineages Maintained by Continuous Inhibition of Extrinsic and Intrinsic Death Pathways

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