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The Oxidative DNA Lesion 8,5′-(S)-Cyclo-2′-deoxyadenosine Is Repaired by the Nucleotide Excision Repair Pathway and Blocks Gene Expression in Mammalian Cells

The Journal of biological chemistry, 2000-07, Vol.275 (29), p.22355-22362

2000

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DNA repair in human fibroblasts, as reflected by host-cell reactivation of a transfected UV-irradiated luciferase gene, is not related to donor age

Mutation research, 2004-10, Vol.554 (1), p.9-17

2004

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Xeroderma pigmentosum. Defective DNA repair causes skin cancer and neurodegeneration

JAMA : the journal of the American Medical Association, 1988-07, Vol.260 (3), p.384

Robbins, J H

1988

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A 32P-Postlabeling Assay for the Oxidative DNA Lesion 8,5′-Cyclo-2′-deoxyadenosine in Mammalian Tissues

The Journal of biological chemistry, 2001-09, Vol.276 (38), p.36051-36057

2001

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A Childhood Neurodegeneration Due to Defective DNA Repair: A Novel Concept of Disease Based on Studies of Xeroderma Pigmentosum

Journal of child neurology, 1989-04, Vol.4 (2), p.143-146

Robbins, Jay H.

1989

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Clinically asymptomatic xeroderma pigmentosum neurological disease in an adult: evidence for a neurodegeneration in later life caused by defective DNA repair

European neurology, 1993, Vol.33 (3), p.188-190

1993

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Induction by ionizing radiation of the gadd45 gene in cultured human cells: lack of mediation by protein kinase C

Molecular and Cellular Biology, 1991-02, Vol.11 (2), p.1009-1016

1991

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Cockayne Syndrome: Clinicopathologic and Tissue Culture Studies of Affected Siblings

Journal of neuropathology and experimental neurology, 1985-09, Vol.44 (5), p.507-519

1985

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EVIDENCE THAT DNA REPAIR PROCESSES PREVENT PREMATURE DEATH OF NEURONS IN HUMANS

Journal of neuropathology and experimental neurology, 1978-09, Vol.37 (5), p.680-680

Robbins, Jay H

1978

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Fluorescent Light-Induced Chromatid Breaks Distinguish Alzheimer Disease Cells from Normal Cells in Tissue Culture

Proceedings of the National Academy of Sciences - PNAS, 1996-05, Vol.93 (10), p.5146-5150

1996

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Use of Lymphoblastoid Cell Lines to Evaluate the Hypersensitivity to Ultraviolet Radiation in Cockayne Syndrome

Journal of investigative dermatology, 1984-05, Vol.82 (5), p.480-484

1984

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A 32P-Postlabeling Assay for the Oxidative DNA Lesion 8,5â²-Cyclo-2â²-deoxyadenosine in Mammalian Tissues

The Journal of biological chemistry, 2001-09, Vol.276 (38), p.36051

2001

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DNA Repair and Ultraviolet Mutagenesis in Cells From a New Patient With Xeroderma Pigmentosum Group G and Cockayne Syndrome Resemble Xeroderma Pigmentosum Cells

Journal of investigative dermatology, 1996-10, Vol.107 (4), p.647-653

1996

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Gene-specific DNA repair in xeroderma pigmentosum complementation groups A, C, D, and F. Relation to cellular survival and clinical features

The Journal of biological chemistry, 1993-03, Vol.268 (7), p.4839-4847

1993

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Human fibroblast strain with normal survival but abnormal postreplication repair after ultraviolet light irradiation

Cancer research (Chicago, Ill.), 1980-08, Vol.40 (8 Pt 1), p.2736

1980

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Neurological disease in xeroderma pigmentosum : documentation of a late onset type of the juvenile onset form

Brain (London, England : 1878), 1991-06, Vol.114, p.1335-1361

1991

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Incorrect priority claim for the DNA-damage hypothesis

Archives of neurology (Chicago), 1987-06, Vol.44 (6), p.579-580

Robbins, J H

1987

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Relationship of DNA repair to carcinogenesis in xeroderma pigmentosum

Cancer research (Chicago, Ill.), 1973-05, Vol.33 (5), p.929

1973

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Antigen-induced blastogenesis: the human cell determining the specificity of response in vitro

The Journal of immunology (1950), 1970-05, Vol.104 (5), p.1295-1299

1970

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The Oxidative DNA Lesion 8,5â²-(S)-Cyclo-2â²-deoxyadenosine Is Repaired by the Nucleotide Excision Repair Pathway and Blocks Gene Expression in Mammalian Cells

The Journal of biological chemistry, 2000-07, Vol.275 (29), p.22355

2000

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Xeroderma pigmentosum complementation group H is withdrawn and reassigned to group D

Human genetics, 1991-12, Vol.88 (2), p.242-242

Robbins, J H

1991

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No lack of complementation for unscheduled DNA synthesis between xeroderma pigmentosum complementation groups D and H

Human genetics, 1989-12, Vol.84 (1), p.99-100

Robbins, J H

1989

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Xeroderma Pigmentosum Neurological Abnormalities Correlate with Colony-Forming Ability after Ultraviolet Radiation

Proceedings of the National Academy of Sciences - PNAS, 1978-04, Vol.75 (4), p.1984-1988

1978

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Parkinson's disease and Alzheimer's disease: hypersensitivity to X rays in cultured cell lines

Journal of neurology, neurosurgery and psychiatry, 1985-09, Vol.48 (9), p.916-923

1985

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Parkinson's disease, twins, and the DNA-damage hypothesis

Annals of neurology, 1987-04, Vol.21 (4), p.412-412

Robbins, Jay H.

1987

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The Oxidative DNA Lesion 8,5′-(S)-Cyclo-2′-deoxyadenosine Is Repaired by the Nucleotide Excision Repair Pathway and Blocks Gene Expression in Mammalian Cells

DNA repair in human fibroblasts, as reflected by host-cell reactivation of a transfected UV-irradiated luciferase gene, is not related to donor age

Xeroderma pigmentosum. Defective DNA repair causes skin cancer and neurodegeneration

A 32P-Postlabeling Assay for the Oxidative DNA Lesion 8,5′-Cyclo-2′-deoxyadenosine in Mammalian Tissues

A Childhood Neurodegeneration Due to Defective DNA Repair: A Novel Concept of Disease Based on Studies of Xeroderma Pigmentosum

Clinically asymptomatic xeroderma pigmentosum neurological disease in an adult: evidence for a neurodegeneration in later life caused by defective DNA repair

Induction by ionizing radiation of the gadd45 gene in cultured human cells: lack of mediation by protein kinase C

Cockayne Syndrome: Clinicopathologic and Tissue Culture Studies of Affected Siblings

EVIDENCE THAT DNA REPAIR PROCESSES PREVENT PREMATURE DEATH OF NEURONS IN HUMANS

Fluorescent Light-Induced Chromatid Breaks Distinguish Alzheimer Disease Cells from Normal Cells in Tissue Culture

Use of Lymphoblastoid Cell Lines to Evaluate the Hypersensitivity to Ultraviolet Radiation in Cockayne Syndrome

A 32P-Postlabeling Assay for the Oxidative DNA Lesion 8,5â²-Cyclo-2â²-deoxyadenosine in Mammalian Tissues

DNA Repair and Ultraviolet Mutagenesis in Cells From a New Patient With Xeroderma Pigmentosum Group G and Cockayne Syndrome Resemble Xeroderma Pigmentosum Cells

Gene-specific DNA repair in xeroderma pigmentosum complementation groups A, C, D, and F. Relation to cellular survival and clinical features

Human fibroblast strain with normal survival but abnormal postreplication repair after ultraviolet light irradiation

Neurological disease in xeroderma pigmentosum : documentation of a late onset type of the juvenile onset form

Incorrect priority claim for the DNA-damage hypothesis

Relationship of DNA repair to carcinogenesis in xeroderma pigmentosum

Antigen-induced blastogenesis: the human cell determining the specificity of response in vitro

The Oxidative DNA Lesion 8,5â²-(S)-Cyclo-2â²-deoxyadenosine Is Repaired by the Nucleotide Excision Repair Pathway and Blocks Gene Expression in Mammalian Cells

Xeroderma pigmentosum complementation group H is withdrawn and reassigned to group D

No lack of complementation for unscheduled DNA synthesis between xeroderma pigmentosum complementation groups D and H

Xeroderma Pigmentosum Neurological Abnormalities Correlate with Colony-Forming Ability after Ultraviolet Radiation

Parkinson's disease and Alzheimer's disease: hypersensitivity to X rays in cultured cell lines

Parkinson's disease, twins, and the DNA-damage hypothesis

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