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The role of autophagy in neurodegenerative disease

Nature medicine, 2013-08, Vol.19 (8), p.983-997

Nixon, Ralph A

2013

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Amyloid precursor protein and endosomal‐lysosomal dysfunction in Alzheimer's disease: inseparable partners in a multifactorial disease

The FASEB journal, 2017-07, Vol.31 (7), p.2729-2743

Nixon, Ralph A.

2017

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Neurofilament Proteins as Biomarkers to Monitor Neurological Diseases and the Efficacy of Therapies

Frontiers in neuroscience, 2021-09, Vol.15, p.689938-689938

2021

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Autophagy, amyloidogenesis and Alzheimer disease

Journal of cell science, 2007-12, Vol.120 (23), p.4081-4091

Nixon, Ralph A

2007

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Disorders of lysosomal acidification—The emerging role of v-ATPase in aging and neurodegenerative disease

Ageing research reviews, 2016-12, Vol.32, p.75-88

2016

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Neurofilaments and Neurofilament Proteins in Health and Disease

Cold Spring Harbor perspectives in biology, 2017-04, Vol.9 (4), p.a018309

2017

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Lysosome trafficking and signaling in health and neurodegenerative diseases

Neurobiology of disease, 2019-02, Vol.122, p.94-105

2019

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Autophagy failure in Alzheimer's disease—locating the primary defect

Neurobiology of disease, 2011-07, Vol.43 (1), p.38-45

2011

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Lysosomal proteolysis inhibition selectively disrupts axonal transport of degradative organelles and causes an Alzheimer's-like axonal dystrophy

The Journal of neuroscience, 2011-05, Vol.31 (21), p.7817-7830

2011

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Dysfunction of autophagy and endosomal-lysosomal pathways: Roles in pathogenesis of Down syndrome and Alzheimer's Disease

Free radical biology & medicine, 2018-01, Vol.114, p.40-51

2018

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Lysosome and calcium dysregulation in Alzheimer's disease: partners in crime

Biochemical Society transactions, 2013-12, Vol.41 (6), p.1495-1502

2013

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Neurofilaments: neurobiological foundations for biomarker applications

Brain (London, England : 1878), 2020-07, Vol.143 (7), p.1975-1998

2020

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Autophagy failure in Alzheimer's disease and the role of defective lysosomal acidification

The European journal of neuroscience, 2013-06, Vol.37 (12), p.1949-1961

2013

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Endosome function and dysfunction in Alzheimer's disease and other neurodegenerative diseases

Neurobiology of aging, 2005-03, Vol.26 (3), p.373-382

Nixon, Ralph A.

2005

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A gene toolbox for monitoring autophagy transcription

Cell death & disease, 2021-11, Vol.12 (11), p.1044-1044, Article 1044

2021

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Amyloid-independent mechanisms in Alzheimer's disease pathogenesis

The Journal of neuroscience, 2010-11, Vol.30 (45), p.14946-14954

2010

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Drug development targeting degeneration of the basal forebrain cholinergic system: its time has come

Molecular neurodegeneration, 2023-10, Vol.18 (1), p.1-74, Article 74

2023

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Lysosomal Proteolysis and Autophagy Require Presenilin 1 and Are Disrupted by Alzheimer-Related PS1 Mutations

Cell, 2010-06, Vol.141 (7), p.1146-1158

2010

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Neurodegenerative lysosomal disorders: A continuum from development to late age

Autophagy, 2008-07, Vol.4 (5), p.590-599

2008

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Correction: Drug development targeting degeneration of the basal forebrain cholinergic system: its time has come

Molecular neurodegeneration, 2023-11, Vol.18 (1), p.1-81, Article 81

2023

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Presenilin 1 Maintains Lysosomal Ca2+ Homeostasis via TRPML1 by Regulating vATPase-Mediated Lysosome Acidification

Cell reports (Cambridge), 2015-09, Vol.12 (9), p.1430-1444

2015

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Endosomal Dysfunction Induced by Directly Overactivating Rab5 Recapitulates Prodromal and Neurodegenerative Features of Alzheimer’s Disease

Cell reports (Cambridge), 2020-11, Vol.33 (8), p.108420-108420, Article 108420

2020

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Reversal of autophagy dysfunction in the TgCRND8 mouse model of Alzheimer's disease ameliorates amyloid pathologies and memory deficits

Brain (London, England : 1878), 2011-01, Vol.134 (Pt 1), p.258-277

2011

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Transgenic expression of a ratiometric autophagy probe specifically in neurons enables the interrogation of brain autophagy in vivo

Autophagy, 2019-03, Vol.15 (3), p.543-557

2019

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Defective macroautophagic turnover of brain lipids in the TgCRND8 Alzheimer mouse model: prevention by correcting lysosomal proteolytic deficits

Brain (London, England : 1878), 2014-12, Vol.137 (Pt 12), p.3300-3318

2014

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The role of autophagy in neurodegenerative disease

Amyloid precursor protein and endosomal‐lysosomal dysfunction in Alzheimer's disease: inseparable partners in a multifactorial disease

Neurofilament Proteins as Biomarkers to Monitor Neurological Diseases and the Efficacy of Therapies

Autophagy, amyloidogenesis and Alzheimer disease

Disorders of lysosomal acidification—The emerging role of v-ATPase in aging and neurodegenerative disease

Neurofilaments and Neurofilament Proteins in Health and Disease

Lysosome trafficking and signaling in health and neurodegenerative diseases

Autophagy failure in Alzheimer's disease—locating the primary defect

Lysosomal proteolysis inhibition selectively disrupts axonal transport of degradative organelles and causes an Alzheimer's-like axonal dystrophy

Dysfunction of autophagy and endosomal-lysosomal pathways: Roles in pathogenesis of Down syndrome and Alzheimer's Disease

Lysosome and calcium dysregulation in Alzheimer's disease: partners in crime

Neurofilaments: neurobiological foundations for biomarker applications

Autophagy failure in Alzheimer's disease and the role of defective lysosomal acidification

Endosome function and dysfunction in Alzheimer's disease and other neurodegenerative diseases

A gene toolbox for monitoring autophagy transcription

Amyloid-independent mechanisms in Alzheimer's disease pathogenesis

Drug development targeting degeneration of the basal forebrain cholinergic system: its time has come

Lysosomal Proteolysis and Autophagy Require Presenilin 1 and Are Disrupted by Alzheimer-Related PS1 Mutations

Neurodegenerative lysosomal disorders: A continuum from development to late age

Correction: Drug development targeting degeneration of the basal forebrain cholinergic system: its time has come

Presenilin 1 Maintains Lysosomal Ca2+ Homeostasis via TRPML1 by Regulating vATPase-Mediated Lysosome Acidification

Endosomal Dysfunction Induced by Directly Overactivating Rab5 Recapitulates Prodromal and Neurodegenerative Features of Alzheimer’s Disease

Reversal of autophagy dysfunction in the TgCRND8 mouse model of Alzheimer's disease ameliorates amyloid pathologies and memory deficits

Transgenic expression of a ratiometric autophagy probe specifically in neurons enables the interrogation of brain autophagy in vivo

Defective macroautophagic turnover of brain lipids in the TgCRND8 Alzheimer mouse model: prevention by correcting lysosomal proteolytic deficits

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