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Biglycan Is Required for Adaptive Remodeling After Myocardial Infarction

Circulation (New York, N.Y.), 2008-03, Vol.117 (10), p.1269-1276

2008

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Response to Letter Regarding Article, “Biglycan Is Required for Adaptive Remodeling After Myocardial Infarction”

Circulation (New York, N.Y.), 2008-10, Vol.118 (15)

2008

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Face selectivity of the C-H bond activation of cyclohexane by the "Bare" first-row transition-metal cations Sc+-Zn

Organometallics, 1995-10, Vol.14 (10), p.4465-4470

1995

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540 Paradox effects of MMP-2 and MMP-9 are beneficial in viral induced experimental myocarditis

European journal of heart failure. Supplements, 2008-06, Vol.7 (Suppl_1), p.139-139

2008

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Paradox effects of MMP-2 and MMP-9 are beneficial in viral induced experimental myocarditis

European journal of heart failure. Supplements, 2008-06, Vol.7, p.139-139

2008

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TIR-domain-containing adaptor protein inducing IFN-beta (TRIF) is essential in coxsackievirus B3-indused myocarditis in mice

European journal of heart failure. Supplements, 2008-06, Vol.7, p.138-138

2008

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536 TIR-domain-containing adaptor protein inducing IFN-beta (TRIF) is essential in coxsackievirus B3-indused myocarditis in mice

European journal of heart failure. Supplements, 2008-06, Vol.7 (Suppl_1), p.138-138

2008

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Generation of Soluble NKG2D Ligands: Proteolytic Cleavage, Exosome Secretion and Functional Implications

Scandinavian journal of immunology, 2013-08, Vol.78 (2), p.120-129

2013

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Lysosome‐Related Effector Vesicles in T Lymphocytes and NK Cells

Scandinavian journal of immunology, 2015-09, Vol.82 (3), p.235-243

2015

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ADAM10 regulates FasL cell surface expression and modulates FasL-induced cytotoxicity and activation-induced cell death

Cell death and differentiation, 2007-05, Vol.14 (5), p.1040-1049

2007

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Secretory lysosomes and their cargo in T and NK cells

Immunology Letters, 2007-01, Vol.108 (1), p.10-19

2007

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The Adaptor Protein Nck Interacts with Fas Ligand: Guiding the Death Factor to the Cytotoxic Immunological Synapse

Proceedings of the National Academy of Sciences - PNAS, 2006-04, Vol.103 (15), p.5911-5916

2006

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FasL cross-linking inhibits activation of human peripheral T cells

International immunology, 2009-05, Vol.21 (5), p.587-598

2009

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Binding of the Intracellular Fas Ligand (FasL) Domain to the Adaptor Protein PSTPIP Results in a Cytoplasmic Localization of FasL

The Journal of biological chemistry, 2005-12, Vol.280 (48), p.40012-40024

2005

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ADAM10 regulates FasL cell surface expression and modulates FasL-induced cytotoxicity and activation-induced cell death

Cell death and differentiation, 2007-02, Vol.14 (5), p.1040-1049

2007

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Generation of Soluble NKG 2 D Ligands: Proteolytic Cleavage, Exosome Secretion and Functional Implications

Scandinavian journal of immunology, 2013-08, Vol.78 (2), p.120-129

2013

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FasL expression and reverse signalling

Results and problems in cell differentiation, 2009-01, Vol.49, p.49

2009

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Whole-body MRI and MRA for evaluation of the prevalence of atherosclerosis in a cohort of subjectively healthy individuals

Insights into imaging, 2012-10, Vol.3 (5), p.485-493

2012

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Considering Fas ligand as a target for therapy

Expert opinion on therapeutic targets, 2005-02, Vol.9 (1), p.119-134

2005

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The PCH family member CIP4 is released from T cells upon activation-induced cell death

Cell communication and signaling, 2009-02, Vol.7 (S1), p.A81-A81, Article A81

2009

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Identification of SH3 domain interaction partners of FasL using a human SH3 domain phage display library

Cell communication and signaling, 2009-02, Vol.7 (S1), p.A92-A92, Article A92

2009

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Identification of interaction partners of the adapter protein Nck in T cells

Cell communication and signaling, 2009-02, Vol.7 (S1), p.A84-A84, Article A84

2009

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Clinical diagnosis of partial duplication 7q

American journal of medical genetics, 1990-10, Vol.37 (2), p.254-257

1990

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Phenology Program of the IBP

Bioscience, 1967-10, Vol.17 (10), p.712-714

1967

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Biglycan Is Required for Adaptive Remodeling After Myocardial Infarction

Response to Letter Regarding Article, “Biglycan Is Required for Adaptive Remodeling After Myocardial Infarction”

Face selectivity of the C-H bond activation of cyclohexane by the "Bare" first-row transition-metal cations Sc+-Zn

540 Paradox effects of MMP-2 and MMP-9 are beneficial in viral induced experimental myocarditis

Paradox effects of MMP-2 and MMP-9 are beneficial in viral induced experimental myocarditis

TIR-domain-containing adaptor protein inducing IFN-beta (TRIF) is essential in coxsackievirus B3-indused myocarditis in mice

536 TIR-domain-containing adaptor protein inducing IFN-beta (TRIF) is essential in coxsackievirus B3-indused myocarditis in mice

Generation of Soluble NKG2D Ligands: Proteolytic Cleavage, Exosome Secretion and Functional Implications

Lysosome‐Related Effector Vesicles in T Lymphocytes and NK Cells

ADAM10 regulates FasL cell surface expression and modulates FasL-induced cytotoxicity and activation-induced cell death

Secretory lysosomes and their cargo in T and NK cells

The Adaptor Protein Nck Interacts with Fas Ligand: Guiding the Death Factor to the Cytotoxic Immunological Synapse

FasL cross-linking inhibits activation of human peripheral T cells

Binding of the Intracellular Fas Ligand (FasL) Domain to the Adaptor Protein PSTPIP Results in a Cytoplasmic Localization of FasL

ADAM10 regulates FasL cell surface expression and modulates FasL-induced cytotoxicity and activation-induced cell death

Generation of Soluble NKG 2 D Ligands: Proteolytic Cleavage, Exosome Secretion and Functional Implications

FasL expression and reverse signalling

Whole-body MRI and MRA for evaluation of the prevalence of atherosclerosis in a cohort of subjectively healthy individuals

Considering Fas ligand as a target for therapy

The PCH family member CIP4 is released from T cells upon activation-induced cell death

Identification of SH3 domain interaction partners of FasL using a human SH3 domain phage display library

Identification of interaction partners of the adapter protein Nck in T cells

Clinical diagnosis of partial duplication 7q

Phenology Program of the IBP

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