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Gefitinib-induced killing of NSCLC cell lines expressing mutant EGFR requires BIM and can be enhanced by BH3 mimetics

PLoS medicine, 2007-10, Vol.4 (10), p.1681-89; discussion 1690

2007

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Substantial Susceptibility of Chronic Lymphocytic Leukemia to BCL2 Inhibition: Results of a Phase I Study of Navitoclax in Patients With Relapsed or Refractory Disease

Journal of clinical oncology, 2012-02, Vol.30 (5), p.488-496

2012

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XIAP discriminates between type I and type II FAS-induced apoptosis

Nature (London), 2009-08, Vol.460 (7258), p.1035-1039

2009

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PRMT1-mediated H4R3me2a recruits SMARCA4 to promote colorectal cancer progression by enhancing EGFR signaling

Genome medicine, 2021-04, Vol.13 (1), p.58-58, Article 58

2021

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Sensitization of BCL-2–expressing breast tumors to chemotherapy by the BH3 mimetic ABT-737

Proceedings of the National Academy of Sciences - PNAS, 2012-02, Vol.109 (8), p.2766-2771

2012

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Current Advances and Future Strategies for BCL-2 Inhibitors: Potent Weapons against Cancers

Cancers, 2023-10, Vol.15 (20), p.4957

2023

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Replication stress induces mitotic death through parallel pathways regulated by WAPL and telomere deprotection

Nature communications, 2019-09, Vol.10 (1), p.4224-15, Article 4224

2019

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Key residues in the VDAC2-BAK complex can be targeted to modulate apoptosis

PLoS biology, 2024-05, Vol.22 (5), p.e3002617

2024

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Apoptosis Initiated When BH3 Ligands Engage Multiple Bcl-2 Homologs, Not Bax or Bak

Science (American Association for the Advancement of Science), 2007-02, Vol.315 (5813), p.856-859

2007

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Two distinct pathways regulate platelet phosphatidylserine exposure and procoagulant function

Blood, 2009-07, Vol.114 (3), p.663-666

2009

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BCL2 and MCL1 inhibitors for hematologic malignancies

Blood, 2021-09, Vol.138 (13), p.1120-1136

2021

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Proapoptotic Bcl-2 Relative Bim Required for Certain Apoptotic Responses, Leukocyte Homeostasis, and to Preclude Autoimmunity

Science (American Association for the Advancement of Science), 1999-11, Vol.286 (5445), p.1735-1738

1999

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Structural basis for apoptosis inhibition by Epstein-Barr virus BHRF1

PLoS pathogens, 2010-12, Vol.6 (12), p.e1001236

2010

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Synthesis of Biotinylated Episilvestrol: Highly Selective Targeting of the Translation Factors eIF4AI/II

Organic letters, 2013-03, Vol.15 (6), p.1406-1409

2013

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Mesenchymal stromal cell apoptosis is required for their therapeutic function

Nature communications, 2021-11, Vol.12 (1), p.6495-6495, Article 6495

2021

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Deubiquitinase USP9X stabilizes MCL1 and promotes tumour cell survival

Nature (London), 2010-01, Vol.463 (7277), p.103-107

2010

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Sheeppox Virus SPPV14 Encodes a Bcl-2-Like Cell Death Inhibitor That Counters a Distinct Set of Mammalian Proapoptotic Proteins

Journal of Virology, 2012-11, Vol.86 (21), p.11501-11511

2012

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In vivo Efficacy of the Bcl-2 Antagonist ABT-737 against Aggressive Myc-Driven Lymphomas

Proceedings of the National Academy of Sciences - PNAS, 2008-11, Vol.105 (46), p.17961-17966

2008

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Structures of BCL-2 in complex with venetoclax reveal the molecular basis of resistance mutations

Nature communications, 2019-06, Vol.10 (1), p.2385-2385, Article 2385

2019

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A Chemical Screening Approach to Identify Novel Key Mediators of Erythroid Enucleation

PloS one, 2015-11, Vol.10 (11), p.e0142655-e0142655

2015

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The BCL2 selective inhibitor venetoclax induces rapid onset apoptosis of CLL cells in patients via a TP53-independent mechanism

Blood, 2016-06, Vol.127 (25), p.3215-3224

2016

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Apoptosis initiated by Bcl-2-regulated caspase activation independently of the cytochrome c/Apaf-1/caspase-9 apoptosome

Nature (London), 2002-10, Vol.419 (6907), p.634-637

2002

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Enhanced stability of Mcl1, a prosurvival Bcl2 relative, blunts stress-induced apoptosis, causes male sterility, and promotes tumorigenesis

Proceedings of the National Academy of Sciences - PNAS, 2014-01, Vol.111 (1), p.261-266

2014

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BH3 mimetics antagonizing restricted prosurvival Bcl-2 proteins represent another class of selective immune modulatory drugs

Proceedings of the National Academy of Sciences - PNAS, 2010-06, Vol.107 (24), p.10967-10971

2010

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De-Novo Designed Library of Benzoylureas as Inhibitors of BCL‑XL: Synthesis, Structural and Biochemical Characterization

Journal of medicinal chemistry, 2014-02, Vol.57 (4), p.1323-1343

2014

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Gefitinib-induced killing of NSCLC cell lines expressing mutant EGFR requires BIM and can be enhanced by BH3 mimetics

Substantial Susceptibility of Chronic Lymphocytic Leukemia to BCL2 Inhibition: Results of a Phase I Study of Navitoclax in Patients With Relapsed or Refractory Disease

XIAP discriminates between type I and type II FAS-induced apoptosis

PRMT1-mediated H4R3me2a recruits SMARCA4 to promote colorectal cancer progression by enhancing EGFR signaling

Sensitization of BCL-2–expressing breast tumors to chemotherapy by the BH3 mimetic ABT-737

Current Advances and Future Strategies for BCL-2 Inhibitors: Potent Weapons against Cancers

Replication stress induces mitotic death through parallel pathways regulated by WAPL and telomere deprotection

Key residues in the VDAC2-BAK complex can be targeted to modulate apoptosis

Apoptosis Initiated When BH3 Ligands Engage Multiple Bcl-2 Homologs, Not Bax or Bak

Two distinct pathways regulate platelet phosphatidylserine exposure and procoagulant function

BCL2 and MCL1 inhibitors for hematologic malignancies

Proapoptotic Bcl-2 Relative Bim Required for Certain Apoptotic Responses, Leukocyte Homeostasis, and to Preclude Autoimmunity

Structural basis for apoptosis inhibition by Epstein-Barr virus BHRF1

Synthesis of Biotinylated Episilvestrol: Highly Selective Targeting of the Translation Factors eIF4AI/II

Mesenchymal stromal cell apoptosis is required for their therapeutic function

Deubiquitinase USP9X stabilizes MCL1 and promotes tumour cell survival

Sheeppox Virus SPPV14 Encodes a Bcl-2-Like Cell Death Inhibitor That Counters a Distinct Set of Mammalian Proapoptotic Proteins

In vivo Efficacy of the Bcl-2 Antagonist ABT-737 against Aggressive Myc-Driven Lymphomas

Structures of BCL-2 in complex with venetoclax reveal the molecular basis of resistance mutations

A Chemical Screening Approach to Identify Novel Key Mediators of Erythroid Enucleation

The BCL2 selective inhibitor venetoclax induces rapid onset apoptosis of CLL cells in patients via a TP53-independent mechanism

Apoptosis initiated by Bcl-2-regulated caspase activation independently of the cytochrome c/Apaf-1/caspase-9 apoptosome

Enhanced stability of Mcl1, a prosurvival Bcl2 relative, blunts stress-induced apoptosis, causes male sterility, and promotes tumorigenesis

BH3 mimetics antagonizing restricted prosurvival Bcl-2 proteins represent another class of selective immune modulatory drugs

De-Novo Designed Library of Benzoylureas as Inhibitors of BCL‑XL: Synthesis, Structural and Biochemical Characterization

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