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Global Expression Analysis Identified a Preferentially Nerve Growth Factor-induced Transcriptional Program Regulated by Sustained Mitogen-activated Protein Kinase/Extracellular Signal-regulated Kinase (ERK) and AP-1 Protein Activation during PC12 Cell Differentiation

The Journal of biological chemistry, 2011-12, Vol.286 (52), p.45131-45145

2011

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Role for Egr1 in the Transcriptional Program Associated with Neuronal Differentiation of PC12 Cells

PloS one, 2017-01, Vol.12 (1), p.e0170076-e0170076

2017

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Immediate-Early and Delayed Primary Response Genes Are Distinct in Function and Genomic Architecture

The Journal of biological chemistry, 2007-08, Vol.282 (33), p.23981-23995

2007

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mRNA Degradation Plays a Significant Role in the Program of Gene Expression Regulated by Phosphatidylinositol 3-Kinase Signaling

Molecular and Cellular Biology, 2010-11, Vol.30 (22), p.5295-5305

2010

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A GSK-3-mediated transcriptional network maintains repression of immediate early genes in quiescent cells

Cell cycle (Georgetown, Tex.), 2011-09, Vol.10 (18), p.3072-3077

2011

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Requirement for Phosphatidylinositol-3 Kinase in the Prevention of Apoptosis by Nerve Growth Factor

Science (American Association for the Advancement of Science), 1995-03, Vol.267 (5206), p.2003-2006

1995

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Regulation of Neuronal Survival by the Serine-Threonine Protein Kinase Akt

Science (American Association for the Advancement of Science), 1997-01, Vol.275 (5300), p.661-665

1997

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Glycogen Synthase Kinase-3 Represses Cyclic AMP Response Element-binding Protein (CREB)-targeted Immediate Early Genes in Quiescent Cells

The Journal of biological chemistry, 2007-03, Vol.282 (13), p.9482-9491

2007

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Role of mTOR in the degradation of IRS-1: Regulation of PP2A activity

Journal of cellular biochemistry, 2002, Vol.85 (2), p.304-314

2002

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AP-1 is a component of the transcriptional network regulated by GSK-3 in quiescent cells

PloS one, 2011-05, Vol.6 (5), p.e20150

2011

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Identification of Transcription Factor Binding Sites Upstream of Human Genes Regulated by the Phosphatidylinositol 3-Kinase and MEK/ERK Signaling Pathways

The Journal of biological chemistry, 2004-05, Vol.279 (19), p.20167-20177

2004

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Role of Glycogen Synthase Kinase-3 in the Phosphatidylinositol 3-Kinase/Akt Cell Survival Pathway

The Journal of biological chemistry, 1998-08, Vol.273 (32), p.19929-19932

1998

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Phosphorylation by Cyclin C/Cyclin-Dependent Kinase 2 following Mitogenic Stimulation of Murine Fibroblasts Inhibits Transcriptional Activity of LSF during G1 Progression

Molecular and Cellular Biology, 2009-05, Vol.29 (9), p.2335-2345

2009

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GSK-3 Represses Growth Factor-inducible Genes by Inhibiting NF-κB in Quiescent Cells

The Journal of biological chemistry, 2010-02, Vol.285 (7), p.4472-4480

2010

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The E-box Binding Factors Max/Mnt, MITF, and USF1 Act Coordinately with FoxO to Regulate Expression of Proapoptotic and Cell Cycle Control Genes by Phosphatidylinositol 3-Kinase/Akt/Glycogen Synthase Kinase 3 Signaling

The Journal of biological chemistry, 2011-10, Vol.286 (42), p.36215-36227

2011

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Role of Translation Initiation Factor 2B in Control of Cell Survival by the Phosphatidylinositol 3-Kinase/Akt/Glycogen Synthase Kinase 3β Signaling Pathway

Molecular and Cellular Biology, 2002-01, Vol.22 (2), p.578-586

2002

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Mammalian transcription factor LSF is a target of ERK signaling

Journal of cellular biochemistry, 2003-07, Vol.89 (4), p.733-746

2003

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B-Raf Inhibits Programmed Cell Death Downstream of Cytochrome c Release from Mitochondria by Activating the MEK/Erk Pathway

Molecular and Cellular Biology, 1999-08, Vol.19 (8), p.5308-5315

1999

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p53-dependent Induction of Apoptosis by Proteasome Inhibitors

The Journal of biological chemistry, 1997-05, Vol.272 (20), p.12893-12896

1997

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Prolyl Isomerase Pin1 Regulates Transcription Factor LSF (TFCP2) by Facilitating Dephosphorylation at Two Serine-Proline Motifs

The Journal of biological chemistry, 2010-10, Vol.285 (41), p.31139-31147

2010

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Cellular transforming genes [Retroviruses, isolated from chickens, turkeys, mice, rats, cats, and monkeys]

Science (American Association for the Advancement of Science), 1982-08, Vol.217 (4562), p.801-806

Cooper, Geoffrey M.

1982

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Mouse embryonic stem cells and preimplantation embryos require signaling through the phosphatidylinositol 3-kinase pathway to suppress apoptosis

Molecular reproduction and development, 2005-03, Vol.70 (3), p.324-332

2005

Volltextzugriff (PDF)

Identification of the MDM2 Oncoprotein as a Substrate for CPP32-like Apoptotic Proteases

The Journal of biological chemistry, 1997-06, Vol.272 (24), p.15049-15052

1997

Volltextzugriff (PDF)

Direct Binding and Activation of STAT Transcription Factors by the Herpesvirus saimiri Protein Tip

The Journal of biological chemistry, 2000-06, Vol.275 (22), p.16925-16932

2000

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Activation of a novel human transforming gene, ret, by DNA rearrangement

Cell, 1985-09, Vol.42 (2), p.581-588

1985

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Global Expression Analysis Identified a Preferentially Nerve Growth Factor-induced Transcriptional Program Regulated by Sustained Mitogen-activated Protein Kinase/Extracellular Signal-regulated Kinase (ERK) and AP-1 Protein Activation during PC12 Cell Differentiation

Role for Egr1 in the Transcriptional Program Associated with Neuronal Differentiation of PC12 Cells

Immediate-Early and Delayed Primary Response Genes Are Distinct in Function and Genomic Architecture

mRNA Degradation Plays a Significant Role in the Program of Gene Expression Regulated by Phosphatidylinositol 3-Kinase Signaling

A GSK-3-mediated transcriptional network maintains repression of immediate early genes in quiescent cells

Requirement for Phosphatidylinositol-3 Kinase in the Prevention of Apoptosis by Nerve Growth Factor

Regulation of Neuronal Survival by the Serine-Threonine Protein Kinase Akt

Glycogen Synthase Kinase-3 Represses Cyclic AMP Response Element-binding Protein (CREB)-targeted Immediate Early Genes in Quiescent Cells

Role of mTOR in the degradation of IRS-1: Regulation of PP2A activity

AP-1 is a component of the transcriptional network regulated by GSK-3 in quiescent cells

Identification of Transcription Factor Binding Sites Upstream of Human Genes Regulated by the Phosphatidylinositol 3-Kinase and MEK/ERK Signaling Pathways

Role of Glycogen Synthase Kinase-3 in the Phosphatidylinositol 3-Kinase/Akt Cell Survival Pathway

Phosphorylation by Cyclin C/Cyclin-Dependent Kinase 2 following Mitogenic Stimulation of Murine Fibroblasts Inhibits Transcriptional Activity of LSF during G1 Progression

GSK-3 Represses Growth Factor-inducible Genes by Inhibiting NF-κB in Quiescent Cells

The E-box Binding Factors Max/Mnt, MITF, and USF1 Act Coordinately with FoxO to Regulate Expression of Proapoptotic and Cell Cycle Control Genes by Phosphatidylinositol 3-Kinase/Akt/Glycogen Synthase Kinase 3 Signaling

Role of Translation Initiation Factor 2B in Control of Cell Survival by the Phosphatidylinositol 3-Kinase/Akt/Glycogen Synthase Kinase 3β Signaling Pathway

Mammalian transcription factor LSF is a target of ERK signaling

B-Raf Inhibits Programmed Cell Death Downstream of Cytochrome c Release from Mitochondria by Activating the MEK/Erk Pathway

p53-dependent Induction of Apoptosis by Proteasome Inhibitors

Prolyl Isomerase Pin1 Regulates Transcription Factor LSF (TFCP2) by Facilitating Dephosphorylation at Two Serine-Proline Motifs

Cellular transforming genes [Retroviruses, isolated from chickens, turkeys, mice, rats, cats, and monkeys]

Mouse embryonic stem cells and preimplantation embryos require signaling through the phosphatidylinositol 3-kinase pathway to suppress apoptosis

Identification of the MDM2 Oncoprotein as a Substrate for CPP32-like Apoptotic Proteases

Direct Binding and Activation of STAT Transcription Factors by the Herpesvirus saimiri Protein Tip

Activation of a novel human transforming gene, ret, by DNA rearrangement

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