UB Paderborn / Katalog / Suche / in CDI

Genetic and epigenetic features of bilateral Wilms tumor predisposition in patients from the Children's Oncology Group AREN18B5-Q

Nature communications, 2023-12, Vol.14 (1), p.8006-15, Article 8006

2023

Volltextzugriff (PDF)

Wilm's tumor 1 promotes memory flexibility

Nature communications, 2019-08, Vol.10 (1), p.3756-18, Article 3756

2019

Volltextzugriff (PDF)

A unique subset of low-risk Wilms tumors is characterized by loss of function of TRIM28 (KAP1), a gene critical in early renal development: A Children's Oncology Group study

PloS one, 2018-12, Vol.13 (12), p.e0208936-e0208936

2018

Volltextzugriff (PDF)

A unique subset of low-risk Wilms tumors is characterized by loss of function of TRIM28

PloS one, 2018-12, Vol.13 (12), p.e0208936

2018

Volltextzugriff (PDF)

Reprogramming of Sertoli cells to fetal-like Leydig cells by Wt1 ablation

Proceedings of the National Academy of Sciences - PNAS, 2015-03, Vol.112 (13), p.4003-4008

2015

Volltextzugriff (PDF)

β-Catenin Activation in a Novel Liver Progenitor Cell Type Is Sufficient to Cause Hepatocellular Carcinoma and Hepatoblastoma

Cancer research (Chicago, Ill.), 2014-08, Vol.74 (16), p.4515-4525

2014

Volltextzugriff (PDF)

Ubiquitin specific protease 18 (Usp18) is a WT1 transcriptional target

Experimental cell research, 2013-03, Vol.319 (5), p.612-622

2013

Volltextzugriff (PDF)

Rare variant detection using family-based sequencing analysis

Proceedings of the National Academy of Sciences - PNAS, 2013-03, Vol.110 (10), p.3985-3990

2013

Volltextzugriff (PDF)

WT1 Mutation and 11P15 Loss of Heterozygosity Predict Relapse in Very Low-Risk Wilms Tumors Treated With Surgery Alone: A Children's Oncology Group Study

Journal of clinical oncology, 2011-02, Vol.29 (6), p.698-703

2011

Volltextzugriff (PDF)

Wt1 ablation and Igf2 upregulation in mice result in Wilms tumors with elevated ERK1/2 phosphorylation

The Journal of clinical investigation, 2011-01, Vol.121 (1), p.174-183

2011

Volltextzugriff (PDF)

The Wilms Tumor Gene, Wt1, Is Required for Sox9 Expression and Maintenance of Tubular Architecture in the Developing Testis

Proceedings of the National Academy of Sciences - PNAS, 2006-08, Vol.103 (32), p.11987-11992

2006

Volltextzugriff (PDF)

Loss of heterozygosity for chromosomes 1p and 16q is an adverse prognostic factor in favorable-histology wilms tumor : A report from the national wilms tumor study group: Molecular oncology: genomics

Journal of clinical oncology, 2005, Vol.23 (29), p.7312-7321

2005

Volltextzugriff (PDF)

Upregulation of c-MYC in WT1-mutant tumors: assessment of WT1 putative transcriptional targets using cDNA microarray expression profiling of genetically defined Wilms' tumors

Oncogene, 2003-06, Vol.22 (24), p.3821-3826

2003

Volltextzugriff (PDF)

Absence of PPP2R1A mutations in Wilms tumor

Oncogene, 2001-04, Vol.20 (16), p.2050-2054

2001

Volltextzugriff (PDF)

Microsatellite instability in preinvasive and invasive head and neck squamous carcinoma

The American journal of pathology, 1996-06, Vol.148 (6), p.2067-2072

1996

Volltextzugriff (PDF)

Allelic loss and replication errors at microsatellite loci on chromosome 11p in head and neck squamous carcinoma: association with aggressive biological features

Clinical cancer research, 1996-05, Vol.2 (5), p.903-907

1996

Volltextzugriff (PDF)

Sequential loss of heterozygosity at microsatellite motifs in preinvasive and invasive head and neck squamous carcinoma

Cancer research (Chicago, Ill.), 1995-06, Vol.55 (12), p.2656-2659

1995

Volltextzugriff (PDF)

Menü

Weitere Dienste

Einstellungen

Genetic and epigenetic features of bilateral Wilms tumor predisposition in patients from the Children's Oncology Group AREN18B5-Q

Wilm's tumor 1 promotes memory flexibility

A unique subset of low-risk Wilms tumors is characterized by loss of function of TRIM28 (KAP1), a gene critical in early renal development: A Children's Oncology Group study

A unique subset of low-risk Wilms tumors is characterized by loss of function of TRIM28

A Children's Oncology Group and TARGET initiative exploring the genetic landscape of Wilms tumor

Reprogramming of Sertoli cells to fetal-like Leydig cells by Wt1 ablation

β-Catenin Activation in a Novel Liver Progenitor Cell Type Is Sufficient to Cause Hepatocellular Carcinoma and Hepatoblastoma

Ubiquitin specific protease 18 (Usp18) is a WT1 transcriptional target

Rare variant detection using family-based sequencing analysis

WT1 Mutation and 11P15 Loss of Heterozygosity Predict Relapse in Very Low-Risk Wilms Tumors Treated With Surgery Alone: A Children's Oncology Group Study

Wt1 ablation and Igf2 upregulation in mice result in Wilms tumors with elevated ERK1/2 phosphorylation

The Wilms Tumor Gene, Wt1, Is Required for Sox9 Expression and Maintenance of Tubular Architecture in the Developing Testis

Loss of heterozygosity for chromosomes 1p and 16q is an adverse prognostic factor in favorable-histology wilms tumor : A report from the national wilms tumor study group: Molecular oncology: genomics

Upregulation of c-MYC in WT1-mutant tumors: assessment of WT1 putative transcriptional targets using cDNA microarray expression profiling of genetically defined Wilms' tumors

Absence of PPP2R1A mutations in Wilms tumor

Microsatellite instability in preinvasive and invasive head and neck squamous carcinoma

Allelic loss and replication errors at microsatellite loci on chromosome 11p in head and neck squamous carcinoma: association with aggressive biological features

Sequential loss of heterozygosity at microsatellite motifs in preinvasive and invasive head and neck squamous carcinoma

Aktive Filter

Suchergebnisse filtern