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Regulation of hepatic autophagy by stress‐sensing transcription factor CREBH

The FASEB journal, 2019-07, Vol.33 (7), p.7896-7914

2019

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The mitochondrial NAD kinase functions as a major metabolic regulator upon increased energy demand

Molecular metabolism (Germany), 2022-10, Vol.64, p.101562-101562, Article 101562

2022

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Regulation of hepatic circadian metabolism by the E3 ubiquitin ligase HRD1-controlled CREBH/PPARα transcriptional program

Molecular metabolism (Germany), 2021-07, Vol.49, p.101192-101192, Article 101192

2021

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Lysine Acetylation of CREBH Regulates Fasting-Induced Hepatic Lipid Metabolism

Molecular and cellular biology, 2015-12, Vol.35 (24), p.4121-4134

2015

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Liver-Enriched Transcription Factor CREBH Interacts With Peroxisome Proliferator-Activated Receptor α to Regulate Metabolic Hormone FGF21

Endocrinology (Philadelphia), 2014-03, Vol.155 (3), p.769-782

2014

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Exposure to fine airborne particulate matters induces hepatic fibrosis in murine models

Journal of hepatology, 2015-12, Vol.63 (6), p.1397-1404

2015

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A hepatokine derived from the ER protein CREBH promotes triglyceride metabolism by stimulating lipoprotein lipase activity

Science signaling, 2023-01, Vol.16 (768), p.eadd6702

2023

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CREBH Couples Circadian Clock With Hepatic Lipid Metabolism

Diabetes (New York, N.Y.), 2016-11, Vol.65 (11), p.3369-3383

2016

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IRE1α prevents hepatic steatosis by processing and promoting the degradation of select microRNAs

Science signaling, 2018-05, Vol.11 (530)

2018

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The mitochondrial NAD+ transporter SLC25A51 is a fasting-induced gene affecting SIRT3 functions

Metabolism, clinical and experimental, 2022-10, Vol.135, p.155275-155275, Article 155275

2022

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Deficiency of the Mitochondrial NAD Kinase Causes Stress-Induced Hepatic Steatosis in Mice

Gastroenterology (New York, N.Y. 1943), 2018-01, Vol.154 (1), p.224-237

2018

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MCP-1-induced protein attenuates post-infarct cardiac remodeling and dysfunction through mitigating NF-κB activation and suppressing inflammation-associated microRNA expression

Basic research in cardiology, 2015-05, Vol.110 (3), p.26-26, Article 26

2015

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Toll-like Receptor (TLR) Signaling Interacts with CREBH to Modulate High-density Lipoprotein (HDL) in Response to Bacterial Endotoxin

The Journal of biological chemistry, 2016-10, Vol.291 (44), p.23149-23158

2016

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Inhalation Exposure to PM 2.5 Counteracts Hepatic Steatosis in Mice Fed High-fat Diet by Stimulating Hepatic Autophagy

Scientific reports, 2017-11, Vol.7 (1), p.16286

2017

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Regulation of hepatic autophagy by stress‐sensing transcription factor CREBH

The mitochondrial NAD kinase functions as a major metabolic regulator upon increased energy demand

Regulation of hepatic circadian metabolism by the E3 ubiquitin ligase HRD1-controlled CREBH/PPARα transcriptional program

Lysine Acetylation of CREBH Regulates Fasting-Induced Hepatic Lipid Metabolism

Liver-Enriched Transcription Factor CREBH Interacts With Peroxisome Proliferator-Activated Receptor α to Regulate Metabolic Hormone FGF21

Exposure to fine airborne particulate matters induces hepatic fibrosis in murine models

A hepatokine derived from the ER protein CREBH promotes triglyceride metabolism by stimulating lipoprotein lipase activity

CREBH Couples Circadian Clock With Hepatic Lipid Metabolism

IRE1α prevents hepatic steatosis by processing and promoting the degradation of select microRNAs

The mitochondrial NAD+ transporter SLC25A51 is a fasting-induced gene affecting SIRT3 functions

Deficiency of the Mitochondrial NAD Kinase Causes Stress-Induced Hepatic Steatosis in Mice

MCP-1-induced protein attenuates post-infarct cardiac remodeling and dysfunction through mitigating NF-κB activation and suppressing inflammation-associated microRNA expression

Toll-like Receptor (TLR) Signaling Interacts with CREBH to Modulate High-density Lipoprotein (HDL) in Response to Bacterial Endotoxin

Inhalation Exposure to PM 2.5 Counteracts Hepatic Steatosis in Mice Fed High-fat Diet by Stimulating Hepatic Autophagy

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