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History and genomic sequence analysis of the herpes simplex virus 1 KOS and KOS1.1 sub-strains

Virology (New York, N.Y.), 2016-01, Vol.487, p.215-221

2016

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Palmitoylation of the cysteine-rich endodomain of the SARS–coronavirus spike glycoprotein is important for spike-mediated cell fusion

Virology (New York, N.Y.), 2007-04, Vol.360 (2), p.264-274

2007

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Replication-defective viruses as vaccines and vaccine vectors

Virology (New York, N.Y.), 2006-01, Vol.344 (1), p.230-239

2006

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Genetic analysis of the SARS-coronavirus spike glycoprotein functional domains involved in cell-surface expression and cell-to-cell fusion

Virology (New York, N.Y.), 2005-10, Vol.341 (2), p.215-230

2005

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Intramuscular delivery of replication-defective herpes simplex virus gives antigen expression in muscle syncytia and improved protection against pathogenic HSV-2 strains

Virology (New York, N.Y.), 2018-01, Vol.513, p.129-135

2018

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Protection from genital herpes disease, seroconversion and latent infection in a non-lethal murine genital infection model by immunization with an HSV-2 replication-defective mutant virus

Virology (New York, N.Y.), 2016-01, Vol.488, p.61-67

2016

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Role for herpes simplex virus 1 ICP27 in the inhibition of type I interferon signaling

Virology (New York, N.Y.), 2008-05, Vol.374 (2), p.487-494

2008

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Herpes simplex virus 1 microRNAs expressed abundantly during latent infection are not essential for latency in mouse trigeminal ganglia

Virology (New York, N.Y.), 2011-09, Vol.417 (2), p.239-247

2011

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Expression of SARS coronavirus 1 spike protein from a herpesviral vector induces innate immune signaling and neutralizing antibody responses

Virology (New York, N.Y.), 2021-07, Vol.559, p.165-172

2021

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Herpes simplex virus-1 infection causes the secretion of a type I interferon-antagonizing protein and inhibits signaling at or before Jak-1 activation

Virology (New York, N.Y.), 2010-01, Vol.396 (1), p.21-29

2010

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Production of immunogenic West Nile virus-like particles using a herpes simplex virus 1 recombinant vector

Virology (New York, N.Y.), 2016-09, Vol.496, p.186-193

2016

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Inhibitors of the sodium potassium ATPase that impair herpes simplex virus replication identified via a chemical screening approach

Virology (New York, N.Y.), 2007-09, Vol.366 (2), p.340-348

2007

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Disruption of the U(L)41 gene in the herpes simplex virus 2 dl5-29 mutant increases its immunogenicity and protective capacity in a murine model of genital herpes

Virology (New York, N.Y.), 2008-03, Vol.372 (1), p.165

2008

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Herpes simplex virus replication compartments can form by coalescence of smaller compartments

Virology (New York, N.Y.), 2003-05, Vol.309 (2), p.232-247

2003

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Immunization with a replication-defective herpes simplex virus 2 mutant reduces herpes simplex virus 1 infection and prevents ocular disease

Virology (New York, N.Y.), 2007-11, Vol.368 (2), p.227-231

2007

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Ability of herpes simplex virus vectors to boost immune responses to DNA vectors and to protect against challenge by simian immunodeficiency virus

Virology (New York, N.Y.), 2007-01, Vol.357 (2), p.199-214

2007

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Properties of a herpes simplex virus multiple immediate-early gene-deleted recombinant as a vaccine vector

Virology (New York, N.Y.), 2007-01, Vol.357 (2), p.186-198

2007

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Evidence for a direct interaction between HSV-1 ICP27 and ICP8 proteins

Virology (New York, N.Y.), 2005-01, Vol.331 (1), p.94-105

2005

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Biological Properties of Herpes Simplex Virus 2 Replication-Defective Mutant Strains in a Murine Nasal Infection Model

Virology (New York, N.Y.), 2000-12, Vol.278 (1), p.137-150

2000

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C-terminal region of herpes simplex virus ICP8 protein needed for intranuclear localization

Virology (New York, N.Y.), 2003-05, Vol.309 (2), p.219-231

2003

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Type 3 Reovirus Neuroinvasion after Intramuscular Inoculation: Direct Invasion of Nerve Terminals and Age-Dependent Pathogenesis

Virology (New York, N.Y.), 2002-11, Vol.303 (2), p.222-231

2002

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Type 3 Reovirus Neuroinvasion after Intramuscular Inoculation: Viral Genetic Determinants of Lethality and Spinal Cord Infection

Virology (New York, N.Y.), 2002-11, Vol.303 (2), p.213-221

2002

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Herpes Simplex Virus 1 ICP27 Is Required for Transcription of Two Viral Late (γ2) Genes in Infected Cells

Virology (New York, N.Y.), 2001-05, Vol.283 (2), p.273-284

2001

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Influence of Mucosal and Parenteral Immunization with a Replication-Defective Mutant of HSV-2 on Immune Responses and Protection from Genital Challenge

Virology (New York, N.Y.), 1998-03, Vol.243 (1), p.178-187

1998

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Herpes Simplex Virus Infection Blocks Events in the G1 Phase of the Cell Cycle

Virology (New York, N.Y.), 2000-02, Vol.267 (2), p.326-334

2000

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History and genomic sequence analysis of the herpes simplex virus 1 KOS and KOS1.1 sub-strains

Palmitoylation of the cysteine-rich endodomain of the SARS–coronavirus spike glycoprotein is important for spike-mediated cell fusion

Replication-defective viruses as vaccines and vaccine vectors

Genetic analysis of the SARS-coronavirus spike glycoprotein functional domains involved in cell-surface expression and cell-to-cell fusion

Intramuscular delivery of replication-defective herpes simplex virus gives antigen expression in muscle syncytia and improved protection against pathogenic HSV-2 strains

Protection from genital herpes disease, seroconversion and latent infection in a non-lethal murine genital infection model by immunization with an HSV-2 replication-defective mutant virus

Role for herpes simplex virus 1 ICP27 in the inhibition of type I interferon signaling

Herpes simplex virus 1 microRNAs expressed abundantly during latent infection are not essential for latency in mouse trigeminal ganglia

Expression of SARS coronavirus 1 spike protein from a herpesviral vector induces innate immune signaling and neutralizing antibody responses

Herpes simplex virus-1 infection causes the secretion of a type I interferon-antagonizing protein and inhibits signaling at or before Jak-1 activation

Production of immunogenic West Nile virus-like particles using a herpes simplex virus 1 recombinant vector

Inhibitors of the sodium potassium ATPase that impair herpes simplex virus replication identified via a chemical screening approach

Disruption of the U(L)41 gene in the herpes simplex virus 2 dl5-29 mutant increases its immunogenicity and protective capacity in a murine model of genital herpes

Herpes simplex virus replication compartments can form by coalescence of smaller compartments

Immunization with a replication-defective herpes simplex virus 2 mutant reduces herpes simplex virus 1 infection and prevents ocular disease

Ability of herpes simplex virus vectors to boost immune responses to DNA vectors and to protect against challenge by simian immunodeficiency virus

Properties of a herpes simplex virus multiple immediate-early gene-deleted recombinant as a vaccine vector

Evidence for a direct interaction between HSV-1 ICP27 and ICP8 proteins

Biological Properties of Herpes Simplex Virus 2 Replication-Defective Mutant Strains in a Murine Nasal Infection Model

C-terminal region of herpes simplex virus ICP8 protein needed for intranuclear localization

Type 3 Reovirus Neuroinvasion after Intramuscular Inoculation: Direct Invasion of Nerve Terminals and Age-Dependent Pathogenesis

Type 3 Reovirus Neuroinvasion after Intramuscular Inoculation: Viral Genetic Determinants of Lethality and Spinal Cord Infection

Herpes Simplex Virus 1 ICP27 Is Required for Transcription of Two Viral Late (γ2) Genes in Infected Cells

Influence of Mucosal and Parenteral Immunization with a Replication-Defective Mutant of HSV-2 on Immune Responses and Protection from Genital Challenge

Herpes Simplex Virus Infection Blocks Events in the G1 Phase of the Cell Cycle

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