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IL-17 Promotes Tumor Development through the Induction of Tumor Promoting Microenvironments at Tumor Sites and Myeloid-Derived Suppressor Cells
The Journal of immunology (1950), 2010-03, Vol.184 (5), p.2281-2288
2010
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Immunoprevention of chemical carcinogenesis through early recognition of oncogene mutations
The Journal of immunology (1950), 2015-03, Vol.194 (6), p.2683-2695
2015
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Heat Shock Proteins HSP27 and HSP70 Are Present in the Skin and Are Important Mediators of Allergic Contact Hypersensitivity
The Journal of immunology (1950), 2009-01, Vol.182 (1), p.675-683
2009
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CD8+ IL-17-Producing T Cells Are Important in Effector Functions for the Elicitation of Contact Hypersensitivity Responses
The Journal of immunology (1950), 2006-11, Vol.177 (10), p.6852-6858
2006
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IL-17 and IFN-gamma mediate the elicitation of contact hypersensitivity responses by different mechanisms and both are required for optimal responses
The Journal of immunology (1950), 2009-07, Vol.183 (2), p.1463-1470
2009
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A Critical Role for the Proapoptotic Protein Bid in Ultraviolet-Induced Immune Suppression and Cutaneous Apoptosis
The Journal of immunology (1950), 2008-09, Vol.181 (5), p.3077-3088
2008
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IL-23 Inhibits Melanoma Development by Augmenting DNA Repair and Modulating T Cell Subpopulations
The Journal of immunology (1950), 2017-01, Vol.198 (2), p.950-961
2017
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In Vivo Suppression of Heat Shock Protein (HSP)27 and HSP70 Accelerates DMBA-Induced Skin Carcinogenesis by Inducing Antigenic Unresponsiveness to the Initiating Carcinogenic Chemical
The Journal of immunology (1950), 2015-05, Vol.194 (10), p.4796-4803
2015
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CD4 T Cell-Induced, Bid-Dependent Apoptosis of Cutaneous Dendritic Cells Regulates T Cell Expansion and Immune Responses
Journal of Immunology, 2006-11, Vol.177 (9), p.5956-5967
2006
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In vivo suppression of HSP27 and HSP70 accelerates DMBA-induced skin carcinogenesis by inducing antigenic unresponsiveness to the initiating carcinogenic chemical
The Journal of immunology (1950), 2015-04, Vol.194 (10), p.4796-4803
2015
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IL-17 promotes tumor development through the induction of tumor promoting microenvironments at tumor sites and myeloid derived suppressor cells1
The Journal of immunology (1950), 2010-01, Vol.184 (5), p.2281-2288
2010
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IL-17 and IFN-γ mediate the elicitation of contact hypersensitivity responses by different mechanisms and both are required for optimal responses1
The Journal of immunology (1950), 2009-06, Vol.183 (2), p.1463-1470
2009
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CD8+ Tc1 and TIL-17 cells mediate allergic contact hypersensitivity by different mechanisms (37.18)
The Journal of immunology (1950), 2007-04, Vol.178 (1_Supplement), p.S22-S22
2007
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Induction of biologically active IL-1 beta-converting enzyme and mature IL-1 beta in human keratinocytes by inflammatory and immunologic stimuli
The Journal of immunology (1950), 1997-12, Vol.159 (12), p.6203
1997
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CD8+ IL-17 producing T cells are important in effector functions for the elicitation of contact hypersensitivity responses1
The Journal of immunology (1950), 2006-11, Vol.177 (10), p.6852-6858
2006
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A critical role for the proapoptotic protein Bid in UV-induced immune suppression and cutaneous apoptosis1
The Journal of immunology (1950), 2008-09, Vol.181 (5), p.3077-3088
2008
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Metabolic requirements for induction of contact hypersensitivity to immunotoxic polyaromatic hydrocarbons
The Journal of immunology (1950), 1995-10, Vol.155 (7), p.3530
1995
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IL-17 and IFN-{gamma} Mediate the Elicitation of Contact Hypersensitivity Responses by Different Mechanisms and Both Are Required for Optimal Responses
The Journal of immunology (1950), 2009-07, Vol.183 (2), p.1463
2009
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Regulation of ultraviolet radiation-induced cutaneous DNA damage by type I interferons
The Journal of immunology (1950), 2020-05, Vol.204 (1_Supplement), p.162-162.9
2020
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Neuronal Repellent Slit2 Inhibits Dendritic Cell Migration and the Development of Immune Responses
The Journal of immunology (1950), 2003-12, Vol.171 (12), p.6519-6526
2003
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IL-17 promotes inflammation associated tumor development (100.14)
The Journal of immunology (1950), 2010-04, Vol.184 (1_Supplement), p.100-100.14
2010
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