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Mesenteric lymph from rats subjected to trauma-hemorrhagic shock are injurious to rat pulmonary microvascular endothelial cells as well as human umbilical vein endothelial cells

Shock (Augusta, Ga.), 2001-10, Vol.16 (4), p.290-293

2001

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Endotoxin-induced mesenteric microvascular changes involve inos-derived nitric oxide : Results from a study using iNOS knock out mice

Shock (Augusta, Ga.), 2000-05, Vol.13 (5), p.397-403

2000

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Mesenteric lymph duct ligation provides long term protection against hemorrhagic shock-induced lung injury

Shock (Augusta, Ga.), 2000-09, Vol.14 (3), p.416-9; discussion 419-20

2000

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Hypoxia combined with Escherichia coli produces irreversible gut mucosal injury characterized by increased intestinal cytokine production and DNA degradation

Shock (Augusta, Ga.), 2001-09, Vol.16 (3), p.189-195

2001

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Injury-enhanced calcium mobilization in circulating rat neutrophils models human PMN responses

Shock (Augusta, Ga.), 2001-07, Vol.16 (1), p.15-20

2001

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The role of lymph factors in lung injury, bone marrow suppression, and endothelial cell dysfunction in a primate model of trauma-hemorrhagic shock

Shock (Augusta, Ga.), 2004-09, Vol.22 (3), p.221-228

2004

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Hypertonic saline resuscitation limits neutrophil activation after trauma-hemorrhagic shock

Shock (Augusta, Ga.), 2003-04, Vol.19 (4), p.328-333

2003

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THE RELATIVE ROLE OF CASPASE-DEPENDENT AND INDEPENDENT APOPTOSIS OF ENDOTHELIAL AND PULMONARY EPITHELIAL CELLS, INDUCED BY T/HS LYMPH, VARIES BY CELLTYPE

Shock (Augusta, Ga.), 2006-06, Vol.25 (Supplement 1), p.55

2006

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Persistent HIF-1α activation in gut ischemia/reperfusion injury: Potential role of bacteria and lipopolysaccharide

Shock (Augusta, Ga.), 2004-09, Vol.22 (3), p.270-277

2004

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Serine proteases are involved in the pathogenesis of trauma-hemorrhagic shock-induced gut and lung injury

Shock (Augusta, Ga.), 2003-05, Vol.19 (5), p.452-456

2003

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Trauma-hemorrhagic shock mesenteric lymph from rat contains a modified form of albumin that is implicated in endothelial cell toxicity

Shock (Augusta, Ga.), 2005-05, Vol.23 (5), p.417-425

2005

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Hypertonic saline improves intestinal mucosa barrier function and lung injury after trauma-hemorrhagic shock

Shock (Augusta, Ga.), 2002-06, Vol.17 (6), p.496-501

2002

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Nitric oxide directly impairs intestinal barrier function

Shock (Augusta, Ga.), 2002-02, Vol.17 (2), p.139-145

2002

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ROLE OF AIF IN TRAUMA HEMORRHAGIC SHOCK-INDUCED ENDOTHELIAL APOPTOSIS

Shock (Augusta, Ga.), 2006-06, Vol.25 (Supplement 1), p.10

2006

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MESENTERIC LYMPH FROM MALE RATS INDUCES ENDOTHELIAL CELL APOPTOSIS IN A NEW IN VIVO MODEL: 278

Shock (Augusta, Ga.), 2003-06, Vol.19 (Supplement), p.95

2003

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Lipopolysaccharide-induced enterocyte-derived nitric oxide induces intestinal monolayer permeability in an autocrine fashion

Shock (Augusta, Ga.), 2002-03, Vol.17 (3), p.180-184

2002

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Lymph from a primate baboon trauma hemorrhagic shock model activates human neutrophils

Shock (Augusta, Ga.), 2006-05, Vol.25 (5), p.460-463

2006

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INTESTINAL BACTERIAL OVERGROWTH-INDUCED BACTERIAL TRANSCATION RESULTS IN THE GENERATION OF TOXIC MESENTERIC LYMPH THAT INJURES ENDOTHELIAL CELLS: 137

Shock (Augusta, Ga.), 2002-06, Vol.17 (Supplement), p.46

2002

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Trauma-hemorrhage-induced neutrophil priming is prevented by mesenteric lymph duct ligation

Shock (Augusta, Ga.), 2002-12, Vol.18 (6), p.513-517

2002

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Trauma/hemorrhagic shock mesenteric lymph upregulates adhesion molecule expression and IL-6 production in human umbilical vein endothelial cells

Shock (Augusta, Ga.), 2002-06, Vol.17 (6), p.491-495

2002

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Post-hemorrhagic shock mesenteric lymph is cytotoxic to endothelial cells and activates neutrophils

Shock (Augusta, Ga.), 1998-12, Vol.10 (6), p.407-414

1998

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ALBUMIN PROTECTS AGAINST GUT-INDUCED LUNG INJURY IN VITRO AND IN VIVO: 159

Shock (Augusta, Ga.), 2003-06, Vol.19 (Supplement), p.54

2003

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T/HS MESENTERIC LYMPH DIRECTLY LEADS TO LUNG INJURY AND IMPAIRED RED BLOOD CELL DEFORMABILITY IN A RAT MODEL: 174

Shock (Augusta, Ga.), 2003-06, Vol.19 (Supplement), p.59

2003

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Effect of trauma-hemorrhagic shock on red blood cell deformability and shape

Shock (Augusta, Ga.), 2003-03, Vol.19 (3), p.268-273

2003

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Title: PANCREATIC DUCT LIGATION REDUCES LUNG INJURY FOLLOWING TRAUMA/HEMORRHAGIC SHOCK: 138

Shock (Augusta, Ga.), 2003-06, Vol.19 (Supplement), p.47

2003

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Mesenteric lymph from rats subjected to trauma-hemorrhagic shock are injurious to rat pulmonary microvascular endothelial cells as well as human umbilical vein endothelial cells

Endotoxin-induced mesenteric microvascular changes involve inos-derived nitric oxide : Results from a study using iNOS knock out mice

Mesenteric lymph duct ligation provides long term protection against hemorrhagic shock-induced lung injury

Hypoxia combined with Escherichia coli produces irreversible gut mucosal injury characterized by increased intestinal cytokine production and DNA degradation

Injury-enhanced calcium mobilization in circulating rat neutrophils models human PMN responses

The role of lymph factors in lung injury, bone marrow suppression, and endothelial cell dysfunction in a primate model of trauma-hemorrhagic shock

Hypertonic saline resuscitation limits neutrophil activation after trauma-hemorrhagic shock

THE RELATIVE ROLE OF CASPASE-DEPENDENT AND INDEPENDENT APOPTOSIS OF ENDOTHELIAL AND PULMONARY EPITHELIAL CELLS, INDUCED BY T/HS LYMPH, VARIES BY CELLTYPE

Persistent HIF-1α activation in gut ischemia/reperfusion injury: Potential role of bacteria and lipopolysaccharide

Serine proteases are involved in the pathogenesis of trauma-hemorrhagic shock-induced gut and lung injury

Trauma-hemorrhagic shock mesenteric lymph from rat contains a modified form of albumin that is implicated in endothelial cell toxicity

Hypertonic saline improves intestinal mucosa barrier function and lung injury after trauma-hemorrhagic shock

Nitric oxide directly impairs intestinal barrier function

ROLE OF AIF IN TRAUMA HEMORRHAGIC SHOCK-INDUCED ENDOTHELIAL APOPTOSIS

MESENTERIC LYMPH FROM MALE RATS INDUCES ENDOTHELIAL CELL APOPTOSIS IN A NEW IN VIVO MODEL: 278

Lipopolysaccharide-induced enterocyte-derived nitric oxide induces intestinal monolayer permeability in an autocrine fashion

Lymph from a primate baboon trauma hemorrhagic shock model activates human neutrophils

INTESTINAL BACTERIAL OVERGROWTH-INDUCED BACTERIAL TRANSCATION RESULTS IN THE GENERATION OF TOXIC MESENTERIC LYMPH THAT INJURES ENDOTHELIAL CELLS: 137

Trauma-hemorrhage-induced neutrophil priming is prevented by mesenteric lymph duct ligation

Trauma/hemorrhagic shock mesenteric lymph upregulates adhesion molecule expression and IL-6 production in human umbilical vein endothelial cells

Post-hemorrhagic shock mesenteric lymph is cytotoxic to endothelial cells and activates neutrophils

ALBUMIN PROTECTS AGAINST GUT-INDUCED LUNG INJURY IN VITRO AND IN VIVO: 159

T/HS MESENTERIC LYMPH DIRECTLY LEADS TO LUNG INJURY AND IMPAIRED RED BLOOD CELL DEFORMABILITY IN A RAT MODEL: 174

Effect of trauma-hemorrhagic shock on red blood cell deformability and shape

Title: PANCREATIC DUCT LIGATION REDUCES LUNG INJURY FOLLOWING TRAUMA/HEMORRHAGIC SHOCK: 138

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