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Part II: Minimum Quality Threshold in Preclinical Sepsis Studies (MQTiPSS) for Types of Infections and Organ Dysfunction Endpoints

Shock (Augusta, Ga.), 2019-01, Vol.51 (1), p.23-32

2019

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Premise for Standardized Sepsis Models

Shock (Augusta, Ga.), 2019-01, Vol.51 (1), p.4-9

2019

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Role of activated neutrophils in chest trauma-induced septic acute lung injury

Shock (Augusta, Ga.), 2012-07, Vol.38 (1), p.98-106

2012

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Effect of gender and sex hormones on immune responses following shock

Shock (Augusta, Ga.), 2000-08, Vol.14 (2), p.81-90

2000

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Programmed death 1 expression as a marker for immune and physiological dysfunction in the critically ill surgical patient

Shock (Augusta, Ga.), 2012-08, Vol.38 (2), p.117-122

2012

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Effect of PD-1: PD-L1 in Invariant Natural Killer T-Cell Emigration and Chemotaxis Following Sepsis

Shock (Augusta, Ga.), 2016-05, Vol.45 (5), p.534-539

2016

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Part II: Minimum Quality Threshold in Pre-Clinical Sepsis Studies (MQTiPSS) for Types of Infections and Organ Dysfunction Endpoints

Shock (Augusta, Ga.), 2019-01, Vol.51 (1), p.23-32

2019

Volltextzugriff (PDF)

Inflammatory Mechanisms in Sepsis: Elevated Invariant Natural Killer T-Cell Numbers in Mouse and Their Modulatory Effect on Macrophage Function

Shock (Augusta, Ga.), 2013-08, Vol.40 (2), p.122-128

2013

Volltextzugriff (PDF)

Local tissue expression of the cell death ligand, fas ligand, plays a central role in the development of extrapulmonary acute lung injury

Shock (Augusta, Ga.), 2011-08, Vol.36 (2), p.138-143

2011

Volltextzugriff (PDF)

Differential effects of macrophage inflammatory chemokine-2 and keratinocyte-derived chemokine on hemorrhage-induced neutrophil priming for lung inflammation: Assessment by adoptive cells transfer in mice

Shock (Augusta, Ga.), 2003-04, Vol.19 (4), p.358-365

2003

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The acute immunological response to blood transfusion is influenced by polymicrobial sepsis

Shock (Augusta, Ga.), 2012-12, Vol.38 (6), p.598-606

2012

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Divergent effects of activated neutrophils on inflammation, Kupffer cell/splenocyte activation, and lung injury following blunt chest trauma

Shock (Augusta, Ga.), 2012-02, Vol.37 (2), p.210-218

2012

Volltextzugriff (PDF)

Flutamide: a novel agent for restoring the depressed cell-mediated immunity following soft-tissue trauma and hemorrhagic shock

Shock (Augusta, Ga.), 1997-10, Vol.8 (4), p.242-248

1997

Volltextzugriff (PDF)

Evolution of an immune suppressive macrophage phenotype as a product of P38 MAPK activation in polymicrobial sepsis

Shock (Augusta, Ga.), 2001-01, Vol.15 (1), p.42-48

2001

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Local Tissue Expression of the Cell Death Ligand, FasL, Plays a Central Role in the Development of Extra-Pulmonary Acute Lung Injury

Shock (Augusta, Ga.), 2011-08, Vol.36 (2), p.138-143

2011

Volltextzugriff (PDF)

Gender dimorphism in trauma-hemorrhage-induced thymocyte apoptosis

Shock (Augusta, Ga.), 1999-10, Vol.12 (4), p.316-322

1999

Volltextzugriff (PDF)

EFFECT OF GENDER AND SEX HORMONES ON IMMUNE RESPONSES FOLLOWING SHOCK

Shock (Augusta, Ga.), 2000-08, Vol.14 (2), p.81-90

2000

Volltextzugriff (PDF)

NF-κB activation has tissue-specific effects on immune cell apoptosis during polymicrobial sepsis

Shock (Augusta, Ga.), 2002-10, Vol.18 (4), p.380-386

2002

Volltextzugriff (PDF)

NEUTROPHIL SEQUESTRATION IN LUNG IS A TRANSIENT EVENT DURING POLYMICROBIAL SEPSIS: 88

Shock (Augusta, Ga.), 2002-06, Vol.17 (Supplement), p.30

2002

Volltextzugriff (PDF)

INSIGHTS INTO THE CONTRIBUTION OF STAT4 AND STAT6 SIGNALING TO THE MORBIDITY AND MORTALITY SEEN IN SEPSIS: 182

Shock (Augusta, Ga.), 2001-06, Vol.15 (Supplement), p.61

2001

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BLOCKADE OF FAS/FASL SIGNALING PREVENTS INCREASED INTESTINAL PERMIABILITY DURING SEPSIS: 209

Shock (Augusta, Ga.), 2001-06, Vol.15 (Supplement), p.70

2001

Volltextzugriff (PDF)

INHIBITION OF NF-KB REDUCES APOPTOSIS IN PEYERʼS PATCH B-CELLS DURING POLY-MICROBIAL SEPSIS

Shock (Augusta, Ga.), 1999-06, Vol.11 (Supplement), p.29

1999

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ROLE OF MIP-2 IN SUPPRESSION OF NEUTROPHIL APOPTOSIS: 220

Shock (Augusta, Ga.), 2001-06, Vol.15 (Supplement), p.74

2001

Volltextzugriff (PDF)

THE ROLE OF CASPASES IN MACROPHAGE CELL DYSFUNCTION AND APOPTOSIS DURING SEPSIS

Shock (Augusta, Ga.), 1999-06, Vol.11 (Supplement), p.63

1999

Volltextzugriff (PDF)

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Part II: Minimum Quality Threshold in Preclinical Sepsis Studies (MQTiPSS) for Types of Infections and Organ Dysfunction Endpoints

Minimum Quality Threshold in Pre-Clinical Sepsis Studies (MQTiPSS): An International Expert Consensus Initiative for Improvement of Animal Modeling in Sepsis

Premise for Standardized Sepsis Models

Role of activated neutrophils in chest trauma-induced septic acute lung injury

Effect of gender and sex hormones on immune responses following shock

Programmed death 1 expression as a marker for immune and physiological dysfunction in the critically ill surgical patient

Effect of PD-1: PD-L1 in Invariant Natural Killer T-Cell Emigration and Chemotaxis Following Sepsis

Part II: Minimum Quality Threshold in Pre-Clinical Sepsis Studies (MQTiPSS) for Types of Infections and Organ Dysfunction Endpoints

Inflammatory Mechanisms in Sepsis: Elevated Invariant Natural Killer T-Cell Numbers in Mouse and Their Modulatory Effect on Macrophage Function

Local tissue expression of the cell death ligand, fas ligand, plays a central role in the development of extrapulmonary acute lung injury

Differential effects of macrophage inflammatory chemokine-2 and keratinocyte-derived chemokine on hemorrhage-induced neutrophil priming for lung inflammation: Assessment by adoptive cells transfer in mice

The acute immunological response to blood transfusion is influenced by polymicrobial sepsis

Divergent effects of activated neutrophils on inflammation, Kupffer cell/splenocyte activation, and lung injury following blunt chest trauma

Flutamide: a novel agent for restoring the depressed cell-mediated immunity following soft-tissue trauma and hemorrhagic shock

Evolution of an immune suppressive macrophage phenotype as a product of P38 MAPK activation in polymicrobial sepsis

Local Tissue Expression of the Cell Death Ligand, FasL, Plays a Central Role in the Development of Extra-Pulmonary Acute Lung Injury

Gender dimorphism in trauma-hemorrhage-induced thymocyte apoptosis

EFFECT OF GENDER AND SEX HORMONES ON IMMUNE RESPONSES FOLLOWING SHOCK

NF-κB activation has tissue-specific effects on immune cell apoptosis during polymicrobial sepsis

NEUTROPHIL SEQUESTRATION IN LUNG IS A TRANSIENT EVENT DURING POLYMICROBIAL SEPSIS: 88

INSIGHTS INTO THE CONTRIBUTION OF STAT4 AND STAT6 SIGNALING TO THE MORBIDITY AND MORTALITY SEEN IN SEPSIS: 182

BLOCKADE OF FAS/FASL SIGNALING PREVENTS INCREASED INTESTINAL PERMIABILITY DURING SEPSIS: 209

INHIBITION OF NF-KB REDUCES APOPTOSIS IN PEYERʼS PATCH B-CELLS DURING POLY-MICROBIAL SEPSIS

ROLE OF MIP-2 IN SUPPRESSION OF NEUTROPHIL APOPTOSIS: 220

THE ROLE OF CASPASES IN MACROPHAGE CELL DYSFUNCTION AND APOPTOSIS DURING SEPSIS

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