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Insights into Chronic Wasting Disease and Bovine Spongiform Encephalopathy Species Barriers by Use of Real-Time Conversion

Journal of virology, 2015-09, Vol.89 (18), p.9524-9531

2015

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PrPC Governs Susceptibility to Prion Strains in Bank Vole, While Other Host Factors Modulate Strain Features

Journal of virology, 2016-12, Vol.90 (23), p.10660-10669

2016

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Deer Prion Proteins Modulate the Emergence and Adaptation of Chronic Wasting Disease Strains

Journal of virology, 2015-12, Vol.89 (24), p.12362-12373

2015

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Generating Bona Fide Mammalian Prions with Internal Deletions

Journal of virology, 2016-08, Vol.90 (15), p.6963-6975

2016

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Assessment of the PrPc Amino-Terminal Domain in Prion Species Barriers

Journal of virology, 2016-12, Vol.90 (23), p.10752-10761

2016

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Absence of Evidence for a Causal Link between Bovine Spongiform Encephalopathy Strain Variant L-BSE and Known Forms of Sporadic Creutzfeldt-Jakob Disease in Human PrP Transgenic Mice

Journal of virology, 2016-12, Vol.90 (23), p.10867-10874

2016

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Role of the goat K222-PrP(C) polymorphic variant in prion infection resistance

Journal of virology, 2014-03, Vol.88 (5), p.2670-2676

2014

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Pathogenic Mutations within the Hydrophobic Domain of the Prion Protein Lead to the Formation of Protease-Sensitive Prion Species with Increased Lethality

Journal of Virology, 2014-03, Vol.88 (5), p.2690-2703

2014

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Oral Prion Neuroinvasion Occurs Independently of PrP C Expression in the Gut Epithelium

Journal of virology, 2018-10, Vol.92 (19)

2018

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Mouse prion protein (PrP) segment 100 to 104 regulates conversion of PrP(C) to PrP(Sc) in prion-infected neuroblastoma cells

Journal of virology, 2012-05, Vol.86 (10), p.5626-5636

2012

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Prion Strain- and Species-Dependent Effects of Antiprion Molecules in Primary Neuronal Cultures

Journal of Virology, 2007-12, Vol.81 (24), p.13794-13800

2007

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The glycosylation status of PrPC is a key factor in determining transmissible spongiform encephalopathy transmission between species

Journal of virology, 2015-05, Vol.89 (9), p.4738-4747

2015

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Endogenous Brain Lipids Inhibit Prion Amyloid Formation In Vitro

Journal of virology, 2017-05, Vol.91 (9)

2017

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Evaluation of Quinacrine Treatment for Prion Diseases

Journal of Virology, 2003-08, Vol.77 (15), p.8462-8469

2003

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Bile Acids Reduce Prion Conversion, Reduce Neuronal Loss, and Prolong Male Survival in Models of Prion Disease

Journal of virology, 2015-08, Vol.89 (15), p.7660-7672

2015

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Mapping the prion protein using recombinant antibodies

Journal of Virology, 1998-11, Vol.72 (11), p.9413-9418

1998

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PrP Knockout Cells Expressing Transmembrane PrP Resist Prion Infection

Journal of virology, 2017-01, Vol.91 (2)

2017

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Prion Propagation and Toxicity Occur In Vitro with Two-Phase Kinetics Specific to Strain and Neuronal Type

Journal of Virology, 2013-03, Vol.87 (5), p.2535-2548

2013

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PrP Expression and Replication by Schwann Cells: Implications in Prion Spreading

Journal of Virology, 2002-03, Vol.76 (5), p.2434-2439

2002

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The Charge Structure of Helix 1 in the Prion Protein Regulates Conversion to Pathogenic PrPSc

Journal of Virology, 2006-09, Vol.80 (17), p.8521-8529

2006

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Glycan-Controlled Epitopes of Prion Protein Include a Major Determinant of Susceptibility to Sheep Scrapie

Journal of Virology, 2004-09, Vol.78 (17), p.9270-9276

2004

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Infection of Cell Lines with Experimental and Natural Ovine Scrapie Agents

Journal of Virology, 2010-03, Vol.84 (5), p.2444-2452

2010

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PrPc Does Not Mediate Internalization of PrPSc but Is Required at an Early Stage for De Novo Prion Infection of Rov Cells

Journal of Virology, 2007-10, Vol.81 (19), p.10786-10791

2007

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Mutated but Not Deleted Ovine PrP(C) N-Terminal Polybasic Region Strongly Interferes with Prion Propagation in Transgenic Mice

Journal of virology, 2016-02, Vol.90 (3), p.1638-1646

2016

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Susceptibility of Sheep for Scrapie as Assessed by In Vitro Conversion of Nine Naturally Occurring Variants of PrP

Journal of Virology, 2000-02, Vol.74 (3), p.1407-1414

2000

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Insights into Chronic Wasting Disease and Bovine Spongiform Encephalopathy Species Barriers by Use of Real-Time Conversion

PrPC Governs Susceptibility to Prion Strains in Bank Vole, While Other Host Factors Modulate Strain Features

Deer Prion Proteins Modulate the Emergence and Adaptation of Chronic Wasting Disease Strains

Generating Bona Fide Mammalian Prions with Internal Deletions

Assessment of the PrPc Amino-Terminal Domain in Prion Species Barriers

Absence of Evidence for a Causal Link between Bovine Spongiform Encephalopathy Strain Variant L-BSE and Known Forms of Sporadic Creutzfeldt-Jakob Disease in Human PrP Transgenic Mice

Role of the goat K222-PrP(C) polymorphic variant in prion infection resistance

Pathogenic Mutations within the Hydrophobic Domain of the Prion Protein Lead to the Formation of Protease-Sensitive Prion Species with Increased Lethality

Oral Prion Neuroinvasion Occurs Independently of PrP C Expression in the Gut Epithelium

Mouse prion protein (PrP) segment 100 to 104 regulates conversion of PrP(C) to PrP(Sc) in prion-infected neuroblastoma cells

Prion Strain- and Species-Dependent Effects of Antiprion Molecules in Primary Neuronal Cultures

The glycosylation status of PrPC is a key factor in determining transmissible spongiform encephalopathy transmission between species

Endogenous Brain Lipids Inhibit Prion Amyloid Formation In Vitro

Evaluation of Quinacrine Treatment for Prion Diseases

Bile Acids Reduce Prion Conversion, Reduce Neuronal Loss, and Prolong Male Survival in Models of Prion Disease

Mapping the prion protein using recombinant antibodies

PrP Knockout Cells Expressing Transmembrane PrP Resist Prion Infection

Prion Propagation and Toxicity Occur In Vitro with Two-Phase Kinetics Specific to Strain and Neuronal Type

PrP Expression and Replication by Schwann Cells: Implications in Prion Spreading

The Charge Structure of Helix 1 in the Prion Protein Regulates Conversion to Pathogenic PrPSc

Glycan-Controlled Epitopes of Prion Protein Include a Major Determinant of Susceptibility to Sheep Scrapie

Infection of Cell Lines with Experimental and Natural Ovine Scrapie Agents

PrPc Does Not Mediate Internalization of PrPSc but Is Required at an Early Stage for De Novo Prion Infection of Rov Cells

Mutated but Not Deleted Ovine PrP(C) N-Terminal Polybasic Region Strongly Interferes with Prion Propagation in Transgenic Mice

Susceptibility of Sheep for Scrapie as Assessed by In Vitro Conversion of Nine Naturally Occurring Variants of PrP

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