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Gestational Diabetes Mellitus Impairs Nrf2-Mediated Adaptive Antioxidant Defenses and Redox Signaling in Fetal Endothelial Cells In Utero

Diabetes (New York, N.Y.), 2013-12, Vol.62 (12), p.4088-4097

2013

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Targeted upregulation of pyruvate dehydrogenase kinase (PDK)-4 in slow-twitch skeletal muscle underlies the stable modification of the regulatory characteristics of PDK induced by high-fat feeding

Diabetes (New York, N.Y.), 2000-05, Vol.49 (5), p.775-781

2000

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Altered Phenotype of β-Cells and Other Pancreatic Cell Lineages in Patients With Diffuse Congenital Hyperinsulinism in Infancy Caused by Mutations in the ATP-Sensitive K-Channel

Diabetes (New York, N.Y.), 2015-09, Vol.64 (9), p.3182-3188

2015

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Potential role of peroxisome proliferator-activated receptor-α in the modulation of glucose-stimulated insulin secretion

Diabetes (New York, N.Y.), 2004-02, Vol.53 (suppl_1), p.S71-S81

2004

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Damaging loss of self-control by stressed β-cells

Diabetes (New York, N.Y.), 2012-02, Vol.61 (2), p.267-269

2012

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Damaging loss of self-control by stressed [beta]-cells

Diabetes (New York, N.Y.), 2012-02, Vol.61 (2), p.267

2012

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Acute ω-3 Fatty Acid Enrichment Selectively Reverses High-Saturated Fat Feeding-Induced Insulin Hypersecretion But Does Not Improve Peripheral Insulin Resistance

Diabetes (New York, N.Y.), 2004-02, Vol.53 (suppl 1), p.S166-S171

2004

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Selective modification of pyruvate dehydrogenase kinase isoform expression in rat pancreatic islets elicited by starvation and activation of peroxisome proliferator-activated receptor-α implications for glucose-stimulated insulin secretion

Diabetes (New York, N.Y.), 2001-12, Vol.50 (12), p.2729-2736

2001

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Peroxisome Proliferator–Activated Receptor-α and Glucocorticoids Interactively Regulate Insulin Secretion During Pregnancy

Diabetes (New York, N.Y.), 2006-12, Vol.55 (12), p.3501-3508

2006

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Altered Phenotype of [Beta]-Cells and Other Pancreatic Cell Lineages in Patients With Diffuse Congenital Hyperinsulinism in Infancy Caused by Mutations in the ATP-Sensitive K-Channel

Diabetes (New York, N.Y.), 2015-09, Vol.64 (9), p.3182

2015

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A key role for beta-cell cytosolic phospholipase A(2) in the maintenance of insulin stores but not in the initiation of insulin secretion

Diabetes (New York, N.Y.), 2002-01, Vol.51 (1), p.98

2002

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A Key Role for β-Cell Cytosolic Phospholipase A2 in the Maintenance of Insulin Stores But Not in the Initiation of Insulin Secretion

Diabetes (New York, N.Y.), 2002-01, Vol.51 (1), p.98-104

2002

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Potential role of peroxisome proliferator-activated receptor-[alpha] in the modulation of glucose-stimulated insulin secretion

Diabetes (New York, N.Y.), 2004-02, Vol.53 (2), p.S71

2004

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Potential role of peroxisome proliferator-activated receptor-α in the modulation of glucose-stimulated insulin secretion

Diabetes (New York, N.Y.), 2004-02, Vol.53 (2), p.S71

2004

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Peroxisome Proliferator-Activated Receptor-[alpha] and Glucocorticoids Interactively Regulate Insulin Secretion During Pregnancy

Diabetes (New York, N.Y.), 2006-12, Vol.55 (12), p.3501

2006

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Selective Modification of Pyruvate Dehydrogenase Kinase Isoform Expression in Rat Pancreatic Islets Elicited by Starvation and Activation of Peroxisome Proliferator–Activated Receptor-α

Diabetes (New York, N.Y.), 2001-12, Vol.50 (12), p.2729-2736

2001

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Acute [omega]-3 fatty acid enrichment selectively reverses high-saturated fat feeding-induced insulin hypersecretion but does not improve peripheral insulin resistance.(Section IV: lipid modulators of islet function)

Diabetes (New York, N.Y.), 2004-02, Vol.53 (2), p.S166

2004

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Acute ω-3 fatty acid enrichment selectively reverses high-saturated fat feeding-induced insulin hypersecretion but does not improve peripheral insulin resistance

Diabetes (New York, N.Y.), 2004-02, Vol.53 (2), p.S166

2004

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Influence of Caloric Restriction on the Control of Skeletal-Muscle Glucose Oxidation by Insulin and Leptin

Diabetes (New York, N.Y.), 2000-05, Vol.49 (5), p.A431

2000

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Hypoleptinemia Is Associated with Increased Insulin Sensitivity in Conjunction with Enhanced Glycogen Turnover in Slow-Twitch Skeletal Muscle

Diabetes (New York, N.Y.), 2000-05, Vol.49 (5), p.A428

2000

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Selective modification of pyruvate dehydrogenase kinase isoform expression in rat pancreatic islets elicited by starvation and activation of peroxisome proliferator-activated

Diabetes (New York, N.Y.), 2001-12, Vol.50 (12), p.2729

2001

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A key role for [beta]-cell cytosolic phospholipase [A.sub.2] in the maintenance of insulin stores but not in the initiation of insulin secretion

Diabetes (New York, N.Y.), 2002-01, Vol.51 (1), p.98

2002

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Targeted upregulation of pyruvate dehydrogenase kinase (PDK)-4 in slow-twitch skeletal muscle underlies the stable modification of the regulatory characteristics of PDK induced

Diabetes (New York, N.Y.), 2000-05, Vol.49 (5), p.775

2000

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Gestational Diabetes Mellitus Impairs Nrf2-Mediated Adaptive Antioxidant Defenses and Redox Signaling in Fetal Endothelial Cells In Utero

Targeted upregulation of pyruvate dehydrogenase kinase (PDK)-4 in slow-twitch skeletal muscle underlies the stable modification of the regulatory characteristics of PDK induced by high-fat feeding

Altered Phenotype of β-Cells and Other Pancreatic Cell Lineages in Patients With Diffuse Congenital Hyperinsulinism in Infancy Caused by Mutations in the ATP-Sensitive K-Channel

Potential role of peroxisome proliferator-activated receptor-α in the modulation of glucose-stimulated insulin secretion

Damaging loss of self-control by stressed β-cells

Damaging loss of self-control by stressed [beta]-cells

Acute ω-3 Fatty Acid Enrichment Selectively Reverses High-Saturated Fat Feeding-Induced Insulin Hypersecretion But Does Not Improve Peripheral Insulin Resistance

Selective modification of pyruvate dehydrogenase kinase isoform expression in rat pancreatic islets elicited by starvation and activation of peroxisome proliferator-activated receptor-α implications for glucose-stimulated insulin secretion

Peroxisome Proliferator–Activated Receptor-α and Glucocorticoids Interactively Regulate Insulin Secretion During Pregnancy

Altered Phenotype of [Beta]-Cells and Other Pancreatic Cell Lineages in Patients With Diffuse Congenital Hyperinsulinism in Infancy Caused by Mutations in the ATP-Sensitive K-Channel

A key role for beta-cell cytosolic phospholipase A(2) in the maintenance of insulin stores but not in the initiation of insulin secretion

A Key Role for β-Cell Cytosolic Phospholipase A2 in the Maintenance of Insulin Stores But Not in the Initiation of Insulin Secretion

Potential role of peroxisome proliferator-activated receptor-[alpha] in the modulation of glucose-stimulated insulin secretion

Potential role of peroxisome proliferator-activated receptor-α in the modulation of glucose-stimulated insulin secretion

Peroxisome Proliferator-Activated Receptor-[alpha] and Glucocorticoids Interactively Regulate Insulin Secretion During Pregnancy

Selective Modification of Pyruvate Dehydrogenase Kinase Isoform Expression in Rat Pancreatic Islets Elicited by Starvation and Activation of Peroxisome Proliferator–Activated Receptor-α

Acute [omega]-3 fatty acid enrichment selectively reverses high-saturated fat feeding-induced insulin hypersecretion but does not improve peripheral insulin resistance.(Section IV: lipid modulators of islet function)

Acute ω-3 fatty acid enrichment selectively reverses high-saturated fat feeding-induced insulin hypersecretion but does not improve peripheral insulin resistance

Influence of Caloric Restriction on the Control of Skeletal-Muscle Glucose Oxidation by Insulin and Leptin

Hypoleptinemia Is Associated with Increased Insulin Sensitivity in Conjunction with Enhanced Glycogen Turnover in Slow-Twitch Skeletal Muscle

Selective modification of pyruvate dehydrogenase kinase isoform expression in rat pancreatic islets elicited by starvation and activation of peroxisome proliferator-activated

A key role for [beta]-cell cytosolic phospholipase [A.sub.2] in the maintenance of insulin stores but not in the initiation of insulin secretion

Targeted upregulation of pyruvate dehydrogenase kinase (PDK)-4 in slow-twitch skeletal muscle underlies the stable modification of the regulatory characteristics of PDK induced

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