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Epigenetics and approaches to targeted epigenetic therapy in acute myeloid leukemia

Blood, 2016-01, Vol.127 (1), p.42-52

2016

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Hitting the target in IDH2 mutant AML

Blood, 2017-08, Vol.130 (6), p.693-694

Wouters, Bas J.

2017

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Double CEBPA mutations, but not single CEBPA mutations, define a subgroup of acute myeloid leukemia with a distinctive gene expression profile that is uniquely associated with a favorable outcome

Blood, 2009-03, Vol.113 (13), p.3088-3091

2009

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Allele-specific expression of GATA2 due to epigenetic dysregulation in CEBPA double-mutant AML

Blood, 2021-07, Vol.138 (2), p.160-177

2021

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BCL6 maintains survival and self-renewal of primary human acute myeloid leukemia cells

Blood, 2021-02, Vol.137 (6), p.812-825

2021

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A decade of genome-wide gene expression profiling in acute myeloid leukemia: flashback and prospects

Blood, 2009-01, Vol.113 (2), p.291-298

2009

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Risk stratification of intermediate-risk acute myeloid leukemia: integrative analysis of a multitude of gene mutation and gene expression markers

Blood, 2011-07, Vol.118 (4), p.1069-1076

2011

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Distinct gene expression profiles of acute myeloid/T-lymphoid leukemia with silenced CEBPA and mutations in NOTCH1

Blood, 2007-11, Vol.110 (10), p.3706-3714

2007

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Genome-wide epigenetic analysis delineates a biologically distinct immature acute leukemia with myeloid/T-lymphoid features

Blood, 2009-03, Vol.113 (12), p.2795-2804

2009

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Gene expression profiling of minimally differentiated acute myeloid leukemia: M0 is a distinct entity subdivided by RUNX1 mutation status

Blood, 2009-10, Vol.114 (14), p.3001-3007

2009

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A recurrent in-frame insertion in a CEBPA transactivation domain is a polymorphism rather than a mutation that does not affect gene expression profiling–based clustering of AML

Blood, 2007-01, Vol.109 (1), p.389-390

2007

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Oncogene EVI1 Drives Acute Myeloid Leukemia Via a Targetable EVI1-CtBP1/2 Interaction and By Repressing Myeloid Regulator CEBPA

Blood, 2022-11, Vol.140 (Supplement 1), p.83-84

2022

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Response: CEBPA promoter hypermethylation in a subset of myeloid/T-lymphoid leukemias with a distinct gene expression profile

Blood, 2009-02, Vol.113 (8), p.1866-1866

2009

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A decade of genorne-wide gene expression profiling in acute myeloid leukemia : flashback and prospects

Blood, 2009, Vol.113 (2), p.291-298

2009

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CEBPα Is a Transcriptional Repressor of T-Cell Related Genes Explaining the Myeloid/T-Lymphoid Features of CEBPα-Silenced AML

Blood, 2011-11, Vol.118 (21), p.554-554

2011

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Double, but Not Single, CEBPA mutations Define a Subgroup of Acute Myeloid Leukemia with Favorable Outcome and a Distinct Gene Expression Profile

Blood, 2008-11, Vol.112 (11), p.141-141

2008

Volltextzugriff (PDF)

Genetic vs. Epigenetic Disruption of the CEBPA Locus Yields Epigenomically and Biologically Distinct Leukemia Phenotypes

Blood, 2007-11, Vol.110 (11), p.2117-2117

2007

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Identification of a Subgroup of Biphenotypic Myeloid/T-Cell Acute Leukemias with a CEBPA-Mutant Gene Expression Profile, Frequent CEBPA Promoter Hypermethylation and NOTCH1 Mutations

Blood, 2006-11, Vol.108 (11), p.2237-2237

2006

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Unique Gene Expression Profiles of AML Patients with CEBPA Mutations

Blood, 2005-11, Vol.106 (11), p.745-745

2005

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Tribbles Homolog 2 (Trib2) Inactivates C/EBPalpha and Causes Acute Myelogenous Leukemia

Blood, 2006-11, Vol.108 (11), p.776-776

2006

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Epigenetics and approaches to targeted epigenetic therapy in acute myeloid leukemia

Hitting the target in IDH2 mutant AML

Double CEBPA mutations, but not single CEBPA mutations, define a subgroup of acute myeloid leukemia with a distinctive gene expression profile that is uniquely associated with a favorable outcome

Prognostic impact, concurrent genetic mutations, and gene expression features of AML with CEBPA mutations in a cohort of 1182 cytogenetically normal AML patients: further evidence for CEBPA double mutant AML as a distinctive disease entity

Allele-specific expression of GATA2 due to epigenetic dysregulation in CEBPA double-mutant AML

BCL6 maintains survival and self-renewal of primary human acute myeloid leukemia cells

A decade of genome-wide gene expression profiling in acute myeloid leukemia: flashback and prospects

Risk stratification of intermediate-risk acute myeloid leukemia: integrative analysis of a multitude of gene mutation and gene expression markers

Distinct gene expression profiles of acute myeloid/T-lymphoid leukemia with silenced CEBPA and mutations in NOTCH1

Genome-wide epigenetic analysis delineates a biologically distinct immature acute leukemia with myeloid/T-lymphoid features

Gene expression profiling of minimally differentiated acute myeloid leukemia: M0 is a distinct entity subdivided by RUNX1 mutation status

A recurrent in-frame insertion in a CEBPA transactivation domain is a polymorphism rather than a mutation that does not affect gene expression profiling–based clustering of AML

Oncogene EVI1 Drives Acute Myeloid Leukemia Via a Targetable EVI1-CtBP1/2 Interaction and By Repressing Myeloid Regulator CEBPA

Response: CEBPA promoter hypermethylation in a subset of myeloid/T-lymphoid leukemias with a distinct gene expression profile

A decade of genorne-wide gene expression profiling in acute myeloid leukemia : flashback and prospects

CEBPα Is a Transcriptional Repressor of T-Cell Related Genes Explaining the Myeloid/T-Lymphoid Features of CEBPα-Silenced AML

Double, but Not Single, CEBPA mutations Define a Subgroup of Acute Myeloid Leukemia with Favorable Outcome and a Distinct Gene Expression Profile

Genetic vs. Epigenetic Disruption of the CEBPA Locus Yields Epigenomically and Biologically Distinct Leukemia Phenotypes

Identification of a Subgroup of Biphenotypic Myeloid/T-Cell Acute Leukemias with a CEBPA-Mutant Gene Expression Profile, Frequent CEBPA Promoter Hypermethylation and NOTCH1 Mutations

Unique Gene Expression Profiles of AML Patients with CEBPA Mutations

Tribbles Homolog 2 (Trib2) Inactivates C/EBPalpha and Causes Acute Myelogenous Leukemia

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