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Association of plasminogen activator inhibitor (PAI)-1 (SERPINE1) SNPs with myocardial infarction, plasma PAI-1, and metabolic parameters: the HIFMECH study

Arteriosclerosis, thrombosis, and vascular biology, 2007-10, Vol.27 (10), p.2250-2257

2007

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Plasma thrombin-activatable fibrinolysis inhibitor antigen concentration and genotype in relation to myocardial infarction in the north and south of Europe

Arteriosclerosis, thrombosis, and vascular biology, 2002-05, Vol.22 (5), p.867-873

2002

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Influence of PAI-1 on adipose tissue growth and metabolic parameters in a murine model of diet-induced obesity

Arteriosclerosis, thrombosis, and vascular biology, 2000-04, Vol.20 (4), p.1150-1154

2000

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Thrombin-activatable fibrinolysis inhibitor antigen levels and cardiovascular risk factors

Arteriosclerosis, thrombosis, and vascular biology, 2000-09, Vol.20 (9), p.2156-2161

2000

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PAI-1 produced ex vivo by human adipose tissue is relevant to PAI-1 blood level

Arteriosclerosis, thrombosis, and vascular biology, 1999-05, Vol.19 (5), p.1361-1365

1999

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Metabolic determinants are much more important than genetic polymorphisms in determining the PAI-1 activity and antigen plasma concentrations: a family study with part of the Stanislas Cohort

Arteriosclerosis, thrombosis, and vascular biology, 1998-01, Vol.18 (1), p.84-91

1998

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Plasma PAI-1 levels are more strongly related to liver steatosis than to adipose tissue accumulation

Arteriosclerosis, thrombosis, and vascular biology, 2003-07, Vol.23 (7), p.1262-1268

2003

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The A -844G polymorphism in the PAI-1 gene is associated with a higher risk of venous thrombosis in factor V Leiden carriers

Arteriosclerosis, thrombosis, and vascular biology, 2000-05, Vol.20 (5), p.1387-1391

2000

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Nutritionally induced obesity is attenuated in transgenic mice overexpressing plasminogen activator inhibitor-1

Arteriosclerosis, thrombosis, and vascular biology, 2003-01, Vol.23 (1), p.78-84

2003

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Development of a genotype 325-specific proCPU/TAFI ELISA

Arteriosclerosis, thrombosis, and vascular biology, 2003-06, Vol.23 (6), p.1122-1127

2003

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Stimulating effect of oxidized low density lipoproteins on plasminogen activator inhibitor-1 synthesis by endothelial cells

Arteriosclerosis and thrombosis, 1991-11, Vol.11 (6), p.1821-1829

1991

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Intracellular calcium mobilization suppresses the TNF-alpha-stimulated synthesis of PAI-1 in human endothelial cells. Indications that calcium acts at a translational level

Arteriosclerosis, thrombosis, and vascular biology, 1997-08, Vol.17 (8), p.1550-1560

1997

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Five frequent polymorphisms of the PAI-1 gene: lack of association between genotypes, PAI activity, and triglyceride levels in a healthy population

Arteriosclerosis, thrombosis, and vascular biology, 1997-05, Vol.17 (5), p.851-858

1997

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Stromal cells are the main plasminogen activator inhibitor-1-producing cells in human fat: evidence of differences between visceral and subcutaneous deposits

Arteriosclerosis, thrombosis, and vascular biology, 2002-01, Vol.22 (1), p.173-178

2002

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Plasma plasminogen activator inhibitor-1 in angina pectoris. Influence of plasma insulin and acute-phase response

Arteriosclerosis (Dallas, Tex.), 1989-05, Vol.9 (3), p.362-367

1989

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Association of plasminogen activator inhibitor (PAI)-1 (SERPINE1) SNPs with myocardial infarction, plasma PAI-1, and metabolic parameters: the HIFMECH study

Plasma thrombin-activatable fibrinolysis inhibitor antigen concentration and genotype in relation to myocardial infarction in the north and south of Europe

Influence of PAI-1 on adipose tissue growth and metabolic parameters in a murine model of diet-induced obesity

Thrombin-activatable fibrinolysis inhibitor antigen levels and cardiovascular risk factors

PAI-1 produced ex vivo by human adipose tissue is relevant to PAI-1 blood level

Metabolic determinants are much more important than genetic polymorphisms in determining the PAI-1 activity and antigen plasma concentrations: a family study with part of the Stanislas Cohort

Plasma PAI-1 levels are more strongly related to liver steatosis than to adipose tissue accumulation

The A -844G polymorphism in the PAI-1 gene is associated with a higher risk of venous thrombosis in factor V Leiden carriers

Nutritionally induced obesity is attenuated in transgenic mice overexpressing plasminogen activator inhibitor-1

Development of a genotype 325-specific proCPU/TAFI ELISA

Stimulating effect of oxidized low density lipoproteins on plasminogen activator inhibitor-1 synthesis by endothelial cells

Intracellular calcium mobilization suppresses the TNF-alpha-stimulated synthesis of PAI-1 in human endothelial cells. Indications that calcium acts at a translational level

Five frequent polymorphisms of the PAI-1 gene: lack of association between genotypes, PAI activity, and triglyceride levels in a healthy population

Stromal cells are the main plasminogen activator inhibitor-1-producing cells in human fat: evidence of differences between visceral and subcutaneous deposits

Plasma plasminogen activator inhibitor-1 in angina pectoris. Influence of plasma insulin and acute-phase response

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