UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Conjugation of Transferrin to Azide-Modified CdSe/ZnS Core-Shell Quantum Dots using Cyclooctyne Click Chemistry

Angewandte Chemie (International ed.), 2012-10, Vol.51 (42), p.10523-10527

International ed. in English, 2012

Volltextzugriff (PDF)

Manganese chelation therapy extends survival in a mouse model of M1000 prion disease

Journal of neurochemistry, 2010-07, Vol.114 (2), p.440-451

2010

Volltextzugriff (PDF)

Neurotoxicity from glutathione depletion is dependent on extracellular trace copper

Journal of neuroscience research, 2003-03, Vol.71 (6), p.889-897

2003

Volltextzugriff (PDF)

The Alzheimer’s therapeutic PBT2 promotes amyloid‐β degradation and GSK3 phosphorylation via a metal chaperone activity

Journal of neurochemistry, 2011-10, Vol.119 (1), p.220-230

2011

Volltextzugriff (PDF)

Type-I interferons in Parkinson's disease: innate inflammatory response drives fate of neurons in model of degenerative brain disorder: An editorial comment on 'Type-I interferons mediate the neuroinflammatory response and neurotoxicity induced by rotenone'

Journal of neurochemistry, 2017-04, Vol.141 (1), p.9-11

2017

Volltextzugriff (PDF)

Type‐I interferons in Parkinson's disease: innate inflammatory response drives fate of neurons in model of degenerative brain disorder

Journal of neurochemistry, 2017-04, Vol.141 (1), p.9-11

2017

Volltextzugriff (PDF)

Protein Labelling with Versatile Phosphorescent Metal Complexes for Live Cell Luminescence Imaging

Chemistry : a European journal, 2015-09, Vol.21 (40), p.14146-14155

2015

Volltextzugriff (PDF)

Immunotherapy as a therapeutic treatment for neurodegenerative disorders

Journal of neurochemistry, 2003-11, Vol.87 (4), p.801-808

2003

Volltextzugriff (PDF)

The modulation of metal bio‐availability as a therapeutic strategy for the treatment of Alzheimer's disease

The FEBS journal, 2007-08, Vol.274 (15), p.3775-3783

2007

Volltextzugriff (PDF)

Restored degradation of the Alzheimer's amyloid-β peptide by targeting amyloid formation

Journal of neurochemistry, 2009-03, Vol.108 (5), p.1198-1207

2009

Volltextzugriff (PDF)

Homocysteine potentiates copper‐ and amyloid beta peptide‐mediated toxicity in primary neuronal cultures: possible risk factors in the Alzheimer's‐type neurodegenerative pathways

Journal of neurochemistry, 2001-03, Vol.76 (5), p.1509-1520

2001

Volltextzugriff (PDF)

Gene knockout of amyloid precursor protein and amyloid precursor‐like protein‐2 increases cellular copper levels in primary mouse cortical neurons and embryonic fibroblasts

Journal of neurochemistry, 2004-10, Vol.91 (2), p.423-428

2004

Volltextzugriff (PDF)

Conjugation of Transferrin to Azide‐Modified CdSe/ZnS Core–Shell Quantum Dots using Cyclooctyne Click Chemistry

Angewandte Chemie, 2012-10, Vol.124 (42), p.10675-10679

2012

Volltextzugriff (PDF)

In vitro gamma‐secretase cleavage of the Alzheimer's amyloid precursor protein correlates to a subset of presenilin complexes and is inhibited by zinc

The FEBS journal, 2005-11, Vol.272 (21), p.5544-5557

2005

Volltextzugriff (PDF)

Membrane‐targeted strategies for modulating APP and Aβ‐mediated toxicity

Journal of cellular and molecular medicine, 2009-02, Vol.13 (2), p.249-261

2009

Volltextzugriff (PDF)

Exacerbation of Copper Toxicity in Primary Neuronal Cultures Depleted of Cellular Glutathione

Journal of neurochemistry, 1999-05, Vol.72 (5), p.2092-2098

1999

Volltextzugriff (PDF)

Zinc and oxidative stress induces depletion and aggregation of endogenous TDP-43

Alzheimer's & dementia, 2010-07, Vol.6 (4), p.S77-S78

2010

Volltextzugriff (PDF)

Investigating copper-regulated protein expression in Menkes fibroblasts using antibody microarrays

Proteomics (Weinheim), 2008-05, Vol.8 (9), p.1819-1831

2008

Volltextzugriff (PDF)

Inhibition of α-synuclein Aggregation Leads to Therapeutic Benefits in Multiple Animal Models of Parkinson'S Disease

Alzheimer's & dementia, 2010-07, Vol.6 (4), p.e62-e63

2010

Volltextzugriff (PDF)

The role of autophagy in potentially therapeutic copper complexes

Alzheimer's & dementia, 2009-07, Vol.5 (4), p.P414-P414

2009

Volltextzugriff (PDF)

P2-314: Neuroprotective metallo-complexes activate EGFR and inhibit amyloid-beta peptide accumulation

Alzheimer's & dementia, 2008-07, Vol.4 (4), p.T464-T465

2008

Volltextzugriff (PDF)

P2-292: Second generation MPAC PBT2 inhibits tau phosphorylation and promotes Aβ degradation

Alzheimer's & dementia, 2008-07, Vol.4 (4), p.T458-T458

2008

Volltextzugriff (PDF)

P2-293: Platinum-based inhibitors of amyloid-β as therapeutic agents for Alzheimer's disease

Alzheimer's & dementia, 2008-07, Vol.4 (4), p.T458-T458

2008

Volltextzugriff (PDF)

The Hydrophobic Core Sequence Modulates the Neurotoxic and Secondary Structure Properties of the Prion Peptide 106‐126

Journal of neurochemistry, 1999-10, Vol.73 (4), p.1557-1565

1999

Volltextzugriff (PDF)

MINIREVIEW: The modulation of metal bio-availability as a therapeutic strategy for the treatment of Alzheimer's disease

The FEBS journal, 2007-08, Vol.274 (15), p.3775

2007

Volltextzugriff (PDF)

1 – 25 von 211

Suche als RSS-Feed

Aktive Filter

KollektionWiley-Blackwell Full Collection

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(197)
Mit Volltextzugriff
(196)

Publikationsform

Artikel
(207)
Reviews
(4)

Sprache

Englisch
(208)
Japanisch
(25)
Deutsch
(3)

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Neu hinzugefügt

... seit 90 Tagen
(1)

Menü

Weitere Dienste

Einstellungen

Conjugation of Transferrin to Azide-Modified CdSe/ZnS Core-Shell Quantum Dots using Cyclooctyne Click Chemistry

Manganese chelation therapy extends survival in a mouse model of M1000 prion disease

Neurotoxicity from glutathione depletion is dependent on extracellular trace copper

The Alzheimer’s therapeutic PBT2 promotes amyloid‐β degradation and GSK3 phosphorylation via a metal chaperone activity

Type-I interferons in Parkinson's disease: innate inflammatory response drives fate of neurons in model of degenerative brain disorder: An editorial comment on 'Type-I interferons mediate the neuroinflammatory response and neurotoxicity induced by rotenone'

Type‐I interferons in Parkinson's disease: innate inflammatory response drives fate of neurons in model of degenerative brain disorder

Protein Labelling with Versatile Phosphorescent Metal Complexes for Live Cell Luminescence Imaging

Immunotherapy as a therapeutic treatment for neurodegenerative disorders

The modulation of metal bio‐availability as a therapeutic strategy for the treatment of Alzheimer's disease

Restored degradation of the Alzheimer's amyloid-β peptide by targeting amyloid formation

Homocysteine potentiates copper‐ and amyloid beta peptide‐mediated toxicity in primary neuronal cultures: possible risk factors in the Alzheimer's‐type neurodegenerative pathways

Gene knockout of amyloid precursor protein and amyloid precursor‐like protein‐2 increases cellular copper levels in primary mouse cortical neurons and embryonic fibroblasts

Conjugation of Transferrin to Azide‐Modified CdSe/ZnS Core–Shell Quantum Dots using Cyclooctyne Click Chemistry

In vitro gamma‐secretase cleavage of the Alzheimer's amyloid precursor protein correlates to a subset of presenilin complexes and is inhibited by zinc

Membrane‐targeted strategies for modulating APP and Aβ‐mediated toxicity

Exacerbation of Copper Toxicity in Primary Neuronal Cultures Depleted of Cellular Glutathione

Zinc and oxidative stress induces depletion and aggregation of endogenous TDP-43

Investigating copper-regulated protein expression in Menkes fibroblasts using antibody microarrays

Inhibition of α-synuclein Aggregation Leads to Therapeutic Benefits in Multiple Animal Models of Parkinson'S Disease

The role of autophagy in potentially therapeutic copper complexes

P2-314: Neuroprotective metallo-complexes activate EGFR and inhibit amyloid-beta peptide accumulation

P2-292: Second generation MPAC PBT2 inhibits tau phosphorylation and promotes Aβ degradation

P2-293: Platinum-based inhibitors of amyloid-β as therapeutic agents for Alzheimer's disease

The Hydrophobic Core Sequence Modulates the Neurotoxic and Secondary Structure Properties of the Prion Peptide 106‐126

MINIREVIEW: The modulation of metal bio-availability as a therapeutic strategy for the treatment of Alzheimer's disease

Aktive Filter

Suchergebnisse filtern