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Inherited interstitial deletion of chromosomes 5p and 16q without apparent phenotypic effect: further confirmation

Prenatal diagnosis, 2000-02, Vol.20 (2), p.144-148

2000

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Haemophilia B Brandenberg‐type promoter mutation

Haemophilia : the official journal of the World Federation of Hemophilia, 1999-01, Vol.5 (1), p.73-75

1999

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Reported in vivo splice-site mutations in the factor IX gene: Severity of splicing defects and a hypothesis for predicting deleterious splice donor mutations

Human mutation, 1999, Vol.13 (3), p.221-231

1999

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DUSP22 AND TP63 REARRANGEMENTS PREDICT OUTCOME OF ALK‐NEGATIVE ANAPLASTIC LARGE CELL LYMPHOMA: A DANISH COHORT STUDY

Hematological oncology, 2017-06, Vol.35 (S2), p.391-392

2017

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Germline mutations in Peruvian patients with hemophilia B: Pattern of mutation in AmerIndians is similar to the putative endogenous germline pattern

Human mutation, 1998, Vol.11 (5), p.372-376

1998

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Deletions with inversions: Report of a mutation and review of the literature

Human mutation, 1993, Vol.2 (1), p.53-57

1993

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Interphase FISH to detect PBX1/E2A fusion resulting from the der(19)t(1;19)(q23;p13.3) or t(1;19)(q23;p13.3) in paediatric patients with acute lymphoblastic leukaemia

British journal of haematology, 2005-04, Vol.129 (1), p.45-52

2005

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A past mutation at isoleucine 397 is now a common cause of moderate/mild haemophilia B

British journal of haematology, 1990-06, Vol.75 (2), p.212-216

1990

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Concomitant myelodysplastic syndrome and chronic myeloid leukaemia: treatment outcomes with imatinib mesylate

British journal of haematology, 2003-10, Vol.123 (2), p.366-367

2003

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Reported In Vivo Splice-Site Mutations in the Factor IX Gene: Severity of Splicing Defects and a Hypothesis for Predicting Deleterious Splice Donor Mutations

Human mutation, 1999-01, Vol.13 (3), p.221-231

1999

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A past mutation at isoleucine 397 is now a common cause of moderat/mild haemokphilia B

British journal of haematology, 1990, Vol.75 (2), p.212-216

1990

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A past mutation at isoleucine super(397) is now a common cause of moderate/mild haemophilia B

British journal of haematology, 1990-01, Vol.75 (2), p.212-216

1990

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Myeloproliferative neoplasms in the young: Mayo Clinic experience with 361 patients age 40 years or younger

American journal of hematology, 2018-12, Vol.93 (12), p.1474-1484

2018

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Azacitidine with or without Entinostat for the treatment of therapy‐related myeloid neoplasm: further results of the E1905 North American Leukemia Intergroup study

British journal of haematology, 2016-02, Vol.172 (3), p.384-391

2016

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Momelotinib for myelofibrosis: 12‐year survival data and retrospective comparison to ruxolitinib

American journal of hematology, 2022-12, Vol.97 (12), p.E433-E435

2022

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Obesity in adult acute myeloid leukemia is not associated with inferior response or survival even when dose capping anthracyclines: An ECOG‐ACRIN analysis

Cancer, 2023-08, Vol.129 (16), p.2479-2490

2023

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De novo isolated myeloid sarcoma: comparative analysis of survival in 19 consecutive cases

British journal of haematology, 2021-11, Vol.195 (3), p.413-416

2021

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Typical, atypical and cryptic t(15;17)(q24;q21) (PML::RARA) observed in acute promyelocytic leukemia: A retrospective review of 831 patients with concurrent chromosome and PML::RARA dual‐color dual‐fusion FISH studies

Genes chromosomes & cancer, 2022-10, Vol.61 (10), p.629-634

2022

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Acute myeloid leukemia after age 70 years: A retrospective comparison of survival following treatment with intensive versus HMA ± venetoclax chemotherapy

American journal of hematology, 2021-04, Vol.96 (4), p.E108-E111

2021

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Comparative study of therapy‐related and de novo adult b‐cell acute lymphoblastic leukaemia

British journal of haematology, 2022-02, Vol.196 (4), p.963-968

2022

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A dynamic 3‐factor survival model for acute myeloid leukemia that accounts for response to induction chemotherapy

American journal of hematology, 2022-09, Vol.97 (9), p.1127-1134

2022

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Acute leukemias harboring KMT2A/MLLT10 fusion: a 10‐year experience from a single genomics laboratory

Genes chromosomes & cancer, 2019-08, Vol.58 (8), p.567-577

2019

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Cryptic and atypical KMT2A‐USP2 and KMT2A‐USP8 rearrangements identified by mate pair sequencing in infant and childhood leukemia

Genes chromosomes & cancer, 2020-07, Vol.59 (7), p.422-427

2020

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20+ Years and alive with primary myelofibrosis: Phenotypic signature of very long‐lived patients

American journal of hematology, 2019-03, Vol.94 (3), p.286-290

2019

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A randomized trial of three novel regimens for recurrent acute myeloid leukemia demonstrates the continuing challenge of treating this difficult disease

American journal of hematology, 2019-01, Vol.94 (1), p.111-117

2019

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Inherited interstitial deletion of chromosomes 5p and 16q without apparent phenotypic effect: further confirmation

Haemophilia B Brandenberg‐type promoter mutation

Reported in vivo splice-site mutations in the factor IX gene: Severity of splicing defects and a hypothesis for predicting deleterious splice donor mutations

DUSP22 AND TP63 REARRANGEMENTS PREDICT OUTCOME OF ALK‐NEGATIVE ANAPLASTIC LARGE CELL LYMPHOMA: A DANISH COHORT STUDY

Germline mutations in Peruvian patients with hemophilia B: Pattern of mutation in AmerIndians is similar to the putative endogenous germline pattern

Deletions with inversions: Report of a mutation and review of the literature

Interphase FISH to detect PBX1/E2A fusion resulting from the der(19)t(1;19)(q23;p13.3) or t(1;19)(q23;p13.3) in paediatric patients with acute lymphoblastic leukaemia

A past mutation at isoleucine 397 is now a common cause of moderate/mild haemophilia B

Concomitant myelodysplastic syndrome and chronic myeloid leukaemia: treatment outcomes with imatinib mesylate

Reported In Vivo Splice-Site Mutations in the Factor IX Gene: Severity of Splicing Defects and a Hypothesis for Predicting Deleterious Splice Donor Mutations

A past mutation at isoleucine 397 is now a common cause of moderat/mild haemokphilia B

A past mutation at isoleucine super(397) is now a common cause of moderate/mild haemophilia B

Myeloproliferative neoplasms in the young: Mayo Clinic experience with 361 patients age 40 years or younger

Azacitidine with or without Entinostat for the treatment of therapy‐related myeloid neoplasm: further results of the E1905 North American Leukemia Intergroup study

Momelotinib for myelofibrosis: 12‐year survival data and retrospective comparison to ruxolitinib

Obesity in adult acute myeloid leukemia is not associated with inferior response or survival even when dose capping anthracyclines: An ECOG‐ACRIN analysis

De novo isolated myeloid sarcoma: comparative analysis of survival in 19 consecutive cases

Typical, atypical and cryptic t(15;17)(q24;q21) (PML::RARA) observed in acute promyelocytic leukemia: A retrospective review of 831 patients with concurrent chromosome and PML::RARA dual‐color dual‐fusion FISH studies

Acute myeloid leukemia after age 70 years: A retrospective comparison of survival following treatment with intensive versus HMA ± venetoclax chemotherapy

Comparative study of therapy‐related and de novo adult b‐cell acute lymphoblastic leukaemia

A dynamic 3‐factor survival model for acute myeloid leukemia that accounts for response to induction chemotherapy

Acute leukemias harboring KMT2A/MLLT10 fusion: a 10‐year experience from a single genomics laboratory

Cryptic and atypical KMT2A‐USP2 and KMT2A‐USP8 rearrangements identified by mate pair sequencing in infant and childhood leukemia

20+ Years and alive with primary myelofibrosis: Phenotypic signature of very long‐lived patients

A randomized trial of three novel regimens for recurrent acute myeloid leukemia demonstrates the continuing challenge of treating this difficult disease

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