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Circulating miR-103a-3p contributes to angiotensin II-induced renal inflammation and fibrosis via a SNRK/NF-κB/p65 regulatory axis

Nature communications, 2019-05, Vol.10 (1), p.2145-2145, Article 2145

2019

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Intrarenal production of B-cell survival factors in human lupus nephritis

Modern pathology, 2011-01, Vol.24 (1), p.98-107

2011

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Genetic and pharmacological inhibition of microRNA-92a maintains podocyte cell cycle quiescence and limits crescentic glomerulonephritis

Nature communications, 2017-11, Vol.8 (1), p.1829-15, Article 1829

2017

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Epithelial cell cycle arrest in G2/M mediates kidney fibrosis after injury

Nature medicine, 2010-05, Vol.16 (5), p.535-543

2010

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A genome-wide association study in Han Chinese identifies multiple susceptibility loci for IgA nephropathy

Nature genetics, 2012-02, Vol.44 (2), p.178-182

2012

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The Regulatory T-cell Transcription Factor Foxp3 Protects against Crescentic Glomerulonephritis

Scientific reports, 2017-05, Vol.7 (1), p.1481-13, Article 1481

2017

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The genetic architecture of membranous nephropathy and its potential to improve non-invasive diagnosis

Nature communications, 2020-03, Vol.11 (1), p.1600-18, Article 1600

2020

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Genome-wide association study identifies susceptibility loci for IgA nephropathy

Nature genetics, 2011-04, Vol.43 (4), p.321-327

2011

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Genome-wide association analyses define pathogenic signaling pathways and prioritize drug targets for IgA nephropathy

Nature genetics, 2023-07, Vol.55 (7), p.1091-1105

2023

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Discovery of new risk loci for IgA nephropathy implicates genes involved in immunity against intestinal pathogens

Nature genetics, 2014-11, Vol.46 (11), p.1187-1196

2014

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MicroRNA-146a-deficient mice develop immune complex glomerulonephritis

Scientific reports, 2019-10, Vol.9 (1), p.15597-13, Article 15597

2019

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Uncontrolled C3 activation causes membranoproliferative glomerulonephritis in mice deficient in complement factor H

Nature genetics, 2002-08, Vol.31 (4), p.424-428

2002

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Global transcriptomic changes in glomerular endothelial cells in mice with podocyte depletion and glomerulosclerosis

Cell death & disease, 2021-07, Vol.12 (7), p.687-687, Article 687

2021

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Mir-17–92 regulates bone marrow homing of plasma cells and production of immunoglobulin G2c

Nature communications, 2015-04, Vol.6 (1), p.6764-6764, Article 6764

2015

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Homozygous C1q deficiency causes glomerulonephritis associated with multiple apoptotic bodies

Nature genetics, 1998-05, Vol.19 (1), p.56-59

1998

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Jund is a determinant of macrophage activation and is associated with glomerulonephritis susceptibility

Nature genetics, 2008-05, Vol.40 (5), p.553-559

2008

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C9orf72 ablation causes immune dysregulation characterized by leukocyte expansion, autoantibody production and glomerulonephropathy in mice

Scientific reports, 2016-03, Vol.6 (1), p.23204-23204, Article 23204

2016

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p53 induces miR199a-3p to suppress SOCS7 for STAT3 activation and renal fibrosis in UUO

Scientific reports, 2017-02, Vol.7 (1), p.43409-43409, Article 43409

2017

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Overexpression of Toll-like receptor 8 correlates with the progression of podocyte injury in murine autoimmune glomerulonephritis

Scientific reports, 2014-12, Vol.4 (1), p.7290-7290, Article 7290

2014

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Estrogen receptor-α deficiency attenuates autoimmune disease in (NZB × NZW)F1 mice

Genes and immunity, 2008-03, Vol.9 (2), p.137-152

2008

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BXSB-type genome causes murine autoimmune glomerulonephritis: pathological correlation between telomeric region of chromosome 1 and Yaa

Genes and immunity, 2014-04, Vol.15 (3), p.182-189

2014

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MicroRNAs in kidney physiology and disease

Nature reviews. Nephrology, 2015-01, Vol.11 (1), p.23-33

2015

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Genetic Polymorphism of NPHS1 Modifies the Clinical Manifestations of Ig A Nephropathy

Laboratory investigation, 2003-08, Vol.83 (8), p.1193-1200

2003

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The kinase TBK1 controls IgA class switching by negatively regulating noncanonical NF-κB signaling

Nature immunology, 2012-11, Vol.13 (11), p.1101-1109

2012

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Circulating miR-103a-3p contributes to angiotensin II-induced renal inflammation and fibrosis via a SNRK/NF-κB/p65 regulatory axis

The tetraspanin CD9 controls migration and proliferation of parietal epithelial cells and glomerular disease progression

Intrarenal production of B-cell survival factors in human lupus nephritis

Genetic and pharmacological inhibition of microRNA-92a maintains podocyte cell cycle quiescence and limits crescentic glomerulonephritis

Epithelial cell cycle arrest in G2/M mediates kidney fibrosis after injury

A genome-wide association study in Han Chinese identifies multiple susceptibility loci for IgA nephropathy

The Regulatory T-cell Transcription Factor Foxp3 Protects against Crescentic Glomerulonephritis

The genetic architecture of membranous nephropathy and its potential to improve non-invasive diagnosis

Genome-wide association study identifies susceptibility loci for IgA nephropathy

Genome-wide association analyses define pathogenic signaling pathways and prioritize drug targets for IgA nephropathy

Discovery of new risk loci for IgA nephropathy implicates genes involved in immunity against intestinal pathogens

MicroRNA-146a-deficient mice develop immune complex glomerulonephritis

Uncontrolled C3 activation causes membranoproliferative glomerulonephritis in mice deficient in complement factor H

Global transcriptomic changes in glomerular endothelial cells in mice with podocyte depletion and glomerulosclerosis

Mir-17–92 regulates bone marrow homing of plasma cells and production of immunoglobulin G2c

Homozygous C1q deficiency causes glomerulonephritis associated with multiple apoptotic bodies

Jund is a determinant of macrophage activation and is associated with glomerulonephritis susceptibility

C9orf72 ablation causes immune dysregulation characterized by leukocyte expansion, autoantibody production and glomerulonephropathy in mice

p53 induces miR199a-3p to suppress SOCS7 for STAT3 activation and renal fibrosis in UUO

Overexpression of Toll-like receptor 8 correlates with the progression of podocyte injury in murine autoimmune glomerulonephritis

Estrogen receptor-α deficiency attenuates autoimmune disease in (NZB × NZW)F1 mice

BXSB-type genome causes murine autoimmune glomerulonephritis: pathological correlation between telomeric region of chromosome 1 and Yaa

MicroRNAs in kidney physiology and disease

Genetic Polymorphism of NPHS1 Modifies the Clinical Manifestations of Ig A Nephropathy

The kinase TBK1 controls IgA class switching by negatively regulating noncanonical NF-κB signaling

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