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A CASE OF PULMONARY HYPERTENSION ASSOCIATED WITH MYELOFIBROSIS: THERAPEUTICAL CHALLENGES FROM ACUTE TO CHRONIC SETTING

European heart journal supplements, 2024-05, Vol.26 (Supplement_2), p.ii96-ii97

2024

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Mismatch repair, G(2)/M cell cycle arrest and lethality after DNA damage

Carcinogenesis (New York), 1999-12, Vol.20 (12), p.2317

1999

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Mismatch repair, G2/M cell cycle arrest and lethality after DNA damage

Carcinogenesis (New York), 1999-12, Vol.20 (12), p.2317-2326

1999

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Mismatch repair and differential sensitivity of mouse and human cells to methylating agents

Carcinogenesis (New York), 1999-02, Vol.20 (2), p.205-214

1999

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Effect of hMSH6 cDNA expression on the phenotype of mismatch repair-deficient colon cancer cell line HCT15

Carcinogenesis (New York), 1999-03, Vol.20 (3), p.373-382

1999

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Mismatch repair, G sub(2)/M cell cycle arrest and lethality after DNA damage

Carcinogenesis (New York), 1999-12, Vol.20 (12), p.2317-2326

1999

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Genetic consequences of tolerance to methylation DNA damage in mammalian cells

Carcinogenesis (New York), 1993-10, Vol.14 (10), p.2097-2103

1993

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Tolerance to methylnitrosourea-induced DNA damage is associated with 6-thioguanine resistance in CHO cells

Carcinogenesis (New York), 1989-07, Vol.10 (7), p.1219-1223

1989

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Adjuvant CMF for node-negative and estrogen receptor-negative breast cancer patients

Journal of the National Cancer Institute. Monographs, 1992 (11), p.77-83

1992

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Cytotoxicity, mutations and SCEs induced by methylating agents are reduced in CHO cells expressing an active mammalian O 6 -methylguanine-DNA methyltransferase gene

Carcinogenesis (New York), 1987-10, Vol.8 (10), p.1417-1421

1987

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Isolation of clones displaying enhanced resistance to methylating agents in O 6 -methylguanine-DNA methyltransferase-proficient CHO cells

Carcinogenesis (New York), 1988-07, Vol.9 (7), p.1217-1222

1988

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Behaviorally augmented versus other components in organophosphate tolerance: The role of reinforcement and response factors1

Fundamental and applied toxicology, 1985-12, Vol.5 (6), p.S213-S224

BIGNAMI, G

1985

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Glial Hyaluronate-Binding Protein in Polar Spongioblastoma

Journal of neuropathology and experimental neurology, 1989-03, Vol.48 (2), p.187-196

1989

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Isolation of clones displaying enhanced resistance to methylating agents in O6-methylguanine-DNA methyltransferase-proficient CHO cells

Carcinogenesis (New York), 1988-07, Vol.9 (7), p.1217-1222

1988

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Cytotoxicity, mutations and SCEs induced by methylating agents are reduced in CHO cells expressing an active mammalian O6-methylguanine-DNA methyltransferase gene

Carcinogenesis (New York), 1987-10, Vol.8 (10), p.1417-1421

1987

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SHORT COMMUNICATION: O6-methyltransferase-deficient and -proficient CHO cells differ in their responses to ethyl-and methyl-nitrosourea-induced DNA alkylation

Carcinogenesis (New York), 1989-07, Vol.10 (7), p.1329-1332

1989

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O6-methyltransferase-deficient and -proficient CHO cells differ in their responses to ethyl- and methyl-nitrosourea-induced DNA alkylation

Carcinogenesis (New York), 1989-07, Vol.10 (7), p.1329-1332

1989

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PLASMINOGEN ACTIVATORS IN RAT NEURAL TISSUES DURING DEVELOPMENT AND REGENERATION: 99

Journal of neuropathology and experimental neurology, 1982-05, Vol.41 (3), p.374

1982

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SHORT COMMUNICATION: O 6 -methyltransferase-deficient and -proficient CHO cells differ in their responses to ethyl-and methyl-nitrosourea-induced DNA alkylation

Carcinogenesis (New York), 1989, Vol.10 (7), p.1329-1332

1989

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97 GLIAL FIBRILLARY ACIDIC PROTEIN (GFAP) IN PINEAL NEOPLASMS

Journal of neuropathology and experimental neurology, 1980-05, Vol.39 (3), p.369-369

1980

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Behaviorally augmented versus other components in organophosphate tolerance: The role of reinforcement and response factors

Fundamental and applied toxicology, 1985-12, Vol.5 (6), p.S213-S224

1985

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A CASE OF PULMONARY HYPERTENSION ASSOCIATED WITH MYELOFIBROSIS: THERAPEUTICAL CHALLENGES FROM ACUTE TO CHRONIC SETTING

Mismatch repair, G(2)/M cell cycle arrest and lethality after DNA damage

Mismatch repair, G2/M cell cycle arrest and lethality after DNA damage

Mismatch repair and differential sensitivity of mouse and human cells to methylating agents

Effect of hMSH6 cDNA expression on the phenotype of mismatch repair-deficient colon cancer cell line HCT15

Mismatch repair, G sub(2)/M cell cycle arrest and lethality after DNA damage

Genetic consequences of tolerance to methylation DNA damage in mammalian cells

Tolerance to methylnitrosourea-induced DNA damage is associated with 6-thioguanine resistance in CHO cells

Adjuvant CMF for node-negative and estrogen receptor-negative breast cancer patients

Cytotoxicity, mutations and SCEs induced by methylating agents are reduced in CHO cells expressing an active mammalian O 6 -methylguanine-DNA methyltransferase gene

Isolation of clones displaying enhanced resistance to methylating agents in O 6 -methylguanine-DNA methyltransferase-proficient CHO cells

Behaviorally augmented versus other components in organophosphate tolerance: The role of reinforcement and response factors1

Glial Hyaluronate-Binding Protein in Polar Spongioblastoma

Isolation of clones displaying enhanced resistance to methylating agents in O6-methylguanine-DNA methyltransferase-proficient CHO cells

Cytotoxicity, mutations and SCEs induced by methylating agents are reduced in CHO cells expressing an active mammalian O6-methylguanine-DNA methyltransferase gene

SHORT COMMUNICATION: O6-methyltransferase-deficient and -proficient CHO cells differ in their responses to ethyl-and methyl-nitrosourea-induced DNA alkylation

O6-methyltransferase-deficient and -proficient CHO cells differ in their responses to ethyl- and methyl-nitrosourea-induced DNA alkylation

PLASMINOGEN ACTIVATORS IN RAT NEURAL TISSUES DURING DEVELOPMENT AND REGENERATION: 99

SHORT COMMUNICATION: O 6 -methyltransferase-deficient and -proficient CHO cells differ in their responses to ethyl-and methyl-nitrosourea-induced DNA alkylation

97 GLIAL FIBRILLARY ACIDIC PROTEIN (GFAP) IN PINEAL NEOPLASMS

Behaviorally augmented versus other components in organophosphate tolerance: The role of reinforcement and response factors

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