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RORγt drives production of the cytokine GM-CSF in helper T cells, which is essential for the effector phase of autoimmune neuroinflammation

Nature immunology, 2011-06, Vol.12 (6), p.560

2011

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Act1 mediates IL-17-induced EAE pathogenesis selectively in NG2+ glial cells

Nature neuroscience, 2013-10, Vol.16 (10), p.1401-1408

2013

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The encephalitogenicity of T(H)17 cells is dependent on IL-1- and IL-23-induced production of the cytokine GM-CSF

Nature immunology, 2011-06, Vol.12 (6), p.568

2011

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RRx-001 ameliorates inflammatory diseases by acting as a potent covalent NLRP3 inhibitor

Cellular & molecular immunology, 2021-06, Vol.18 (6), p.1425-1436

2021

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The origin and application of experimental autoimmune encephalomyelitis

Nature reviews. Immunology, 2007-11, Vol.7 (11), p.904-912

Baxter, Alan G

2007

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A regenerative approach to the treatment of multiple sclerosis

Nature (London), 2013-10, Vol.502 (7471), p.327-332

2013

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A new type of microglia gene targeting shows TAK1 to be pivotal in CNS autoimmune inflammation

Nature neuroscience, 2013-11, Vol.16 (11), p.1618-1626

2013

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The aryl hydrocarbon receptor links TH17-cell-mediated autoimmunity to environmental toxins

Nature (London), 2008-05, Vol.453 (7191), p.106-109

2008

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Sequential transcriptional changes dictate safe and effective antigen-specific immunotherapy

Nature communications, 2014-09, Vol.5 (1), p.4741, Article 4741

2014

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Cuprizone and EAE mouse frontal cortex proteomics revealed proteins altered in multiple sclerosis

Scientific reports, 2021-03, Vol.11 (1), p.7174-16, Article 7174

2021

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The interleukin 23 receptor is essential for the terminal differentiation of interleukin 17-producing effector T helper cells in vivo

Nature immunology, 2009-03, Vol.10 (3), p.314-324

2009

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B cell-derived transforming growth factor-β1 expression limits the induction phase of autoimmune neuroinflammation

Scientific reports, 2016-10, Vol.6 (1), p.34594-34594, Article 34594

2016

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LINGO-1 antagonist promotes spinal cord remyelination and axonal integrity in MOG-induced experimental autoimmune encephalomyelitis

Nature medicine, 2007-10, Vol.13 (10), p.1228-1233

2007

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CKD-506: A novel HDAC6-selective inhibitor that exerts therapeutic effects in a rodent model of multiple sclerosis

Scientific reports, 2021-07, Vol.11 (1), p.14466-16, Article 14466

2021

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RORγt+ cells selectively express redundant cation channels linked to the Golgi apparatus

Scientific reports, 2016-03, Vol.6 (1), p.23682-23682, Article 23682

2016

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Intravenous C16 and angiopoietin-1 improve the efficacy of placenta-derived mesenchymal stem cell therapy for EAE

Scientific reports, 2018-03, Vol.8 (1), p.4649, Article 4649

2018

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Autoimmunity: Rubbing salt in the wound

Nature (London), 2013-04, Vol.496 (7446), p.437

2013

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Fasudil regulates T cell responses through polarization of BV-2 cells in mice experimental autoimmune encephalomyelitis

Acta pharmacologica Sinica, 2014-11, Vol.35 (11), p.1428-1438

2014

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In vitro and in vivo pharmacological models to assess demyelination and remyelination

Neuropsychopharmacology (New York, N.Y.), 2009-01, Vol.34 (1), p.55-73

Merrill, Jean E

2009

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Cannabinoids control spasticity and tremor in a multiple sclerosis model

Nature (London), 2000-03, Vol.404 (6773), p.84-87

2000

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Induction of pathogenic TH17 cells by inducible salt-sensing kinase SGK1

Nature (London), 2013-03, Vol.496 (7446), p.513-517

2013

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High doses of α-galactosylceramide potentiate experimental autoimmune encephalomyelitis by directly enhancing Th17 response

Cell research, 2010-04, Vol.20 (4), p.480-491

2010

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TGFβ1 signaling sustains aryl hydrocarbon receptor (AHR) expression and restrains the pathogenic potential of T H 17 cells by an AHR-independent mechanism

Cell death & disease, 2018-11, Vol.9 (11), p.1130

2018

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Sodium chloride drives autoimmune disease by the induction of pathogenic TH17 cells

Nature (London), 2013-04, Vol.496 (7446), p.518-522

2013

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RORγt drives production of the cytokine GM-CSF in helper T cells, which is essential for the effector phase of autoimmune neuroinflammation

Act1 mediates IL-17-induced EAE pathogenesis selectively in NG2+ glial cells

The encephalitogenicity of T(H)17 cells is dependent on IL-1- and IL-23-induced production of the cytokine GM-CSF

RRx-001 ameliorates inflammatory diseases by acting as a potent covalent NLRP3 inhibitor

The origin and application of experimental autoimmune encephalomyelitis

A regenerative approach to the treatment of multiple sclerosis

A new type of microglia gene targeting shows TAK1 to be pivotal in CNS autoimmune inflammation

The aryl hydrocarbon receptor links TH17-cell-mediated autoimmunity to environmental toxins

Sequential transcriptional changes dictate safe and effective antigen-specific immunotherapy

Cuprizone and EAE mouse frontal cortex proteomics revealed proteins altered in multiple sclerosis

The interleukin 23 receptor is essential for the terminal differentiation of interleukin 17-producing effector T helper cells in vivo

B cell-derived transforming growth factor-β1 expression limits the induction phase of autoimmune neuroinflammation

LINGO-1 antagonist promotes spinal cord remyelination and axonal integrity in MOG-induced experimental autoimmune encephalomyelitis

CKD-506: A novel HDAC6-selective inhibitor that exerts therapeutic effects in a rodent model of multiple sclerosis

RORγt+ cells selectively express redundant cation channels linked to the Golgi apparatus

Intravenous C16 and angiopoietin-1 improve the efficacy of placenta-derived mesenchymal stem cell therapy for EAE

Autoimmunity: Rubbing salt in the wound

Fasudil regulates T cell responses through polarization of BV-2 cells in mice experimental autoimmune encephalomyelitis

In vitro and in vivo pharmacological models to assess demyelination and remyelination

Cannabinoids control spasticity and tremor in a multiple sclerosis model

Induction of pathogenic TH17 cells by inducible salt-sensing kinase SGK1

High doses of α-galactosylceramide potentiate experimental autoimmune encephalomyelitis by directly enhancing Th17 response

TGFβ1 signaling sustains aryl hydrocarbon receptor (AHR) expression and restrains the pathogenic potential of T H 17 cells by an AHR-independent mechanism

Sodium chloride drives autoimmune disease by the induction of pathogenic TH17 cells

Salt-responsive gut commensal modulates T H 17 axis and disease

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