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Killer immunoglobulin receptor genes in spondyloarthritis

Current opinion in rheumatology, 2016-07, Vol.28 (4), p.368-375

2016

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New treatment paradigms in spondyloarthritis

Current opinion in rheumatology, 2018-01, Vol.30 (1), p.79-86

2018

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The inflammatory function of human IgA

Cellular and molecular life sciences : CMLS, 2019-03, Vol.76 (6), p.1041-1055

2019

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The novel cytokine interleukin-36α is expressed in psoriatic and rheumatoid arthritis synovium

Annals of the rheumatic diseases, 2013-09, Vol.72 (9), p.1569-1574

2013

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The IL-17A-Producing CD8+ T-Cell Population in Psoriatic Lesional Skin Comprises Mucosa-Associated Invariant T Cells and Conventional T Cells

Journal of investigative dermatology, 2014-12, Vol.134 (12), p.2898-2907

2014

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Fc gamma receptor-TLR cross-talk elicits pro-inflammatory cytokine production by human M2 macrophages

Nature communications, 2014-11, Vol.5 (1), p.5444-5444, Article 5444

2014

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Intimal lining layer macrophages but not synovial sublining macrophages display an IL-10 polarized-like phenotype in chronic synovitis

Arthritis research & therapy, 2012-04, Vol.14 (2), p.R74-R74

2012

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C-Reactive Protein Promotes Inflammation through FcγR-Induced Glycolytic Reprogramming of Human Macrophages

The Journal of immunology (1950), 2019-07, Vol.203 (1), p.225-235

2019

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IgG Subclasses Shape Cytokine Responses by Human Myeloid Immune Cells through Differential Metabolic Reprogramming

The Journal of immunology (1950), 2020-12, Vol.205 (12), p.3400-3407

2020

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Identification and characterisation of citrullinated antigen-specific B cells in peripheral blood of patients with rheumatoid arthritis

Annals of the rheumatic diseases, 2016-06, Vol.75 (6), p.1170-1176

2016

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Interleukin‐17A Is Produced by CD4+ but Not CD8+ T Cells in Synovial Fluid Following T Cell Receptor Activation and Regulates Different Inflammatory Mediators Compared to Tumor Necrosis Factor in a Model of Psoriatic Arthritis Synovitis

Arthritis & rheumatology (Hoboken, N.J.), 2020-08, Vol.72 (8), p.1303-1313

2020

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Expansion of Interleukin‐22– and Granulocyte–Macrophage Colony‐Stimulating Factor–Expressing, but Not Interleukin‐17A–Expressing, Group 3 Innate Lymphoid Cells in the Inflamed Joints of Patients With Spondyloarthritis

Arthritis & rheumatology (Hoboken, N.J.), 2019-03, Vol.71 (3), p.392-402

2019

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Transmembrane TNF drives osteoproliferative joint inflammation reminiscent of human spondyloarthritis

The Journal of experimental medicine, 2020-10, Vol.217 (10)

2020

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C-Reactive Protein Controls IL-23 Production by Human Monocytes

International journal of molecular sciences, 2021-10, Vol.22 (21), p.11638

2021

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Anti-IL-17A treatment reduces serum inflammatory, angiogenic and tissue remodeling biomarkers accompanied by less synovial high endothelial venules in peripheral spondyloarthritis

Scientific reports, 2020-12, Vol.10 (1), p.21094-21094, Article 21094

2020

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FcαRI co-stimulation converts human intestinal CD103 + dendritic cells into pro-inflammatory cells through glycolytic reprogramming

Nature communications, 2018-02, Vol.9 (1), p.863-13, Article 863

2018

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Residual disease activity in psoriatic arthritis: discordance between the rheumatologist's opinion and minimal disease activity measurement

Rheumatology (Oxford, England), 2018-02, Vol.57 (2), p.283-290

2018

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Progression From Subclinical Inflammation to Overt Spondyloarthritis in First‐Degree Relatives of Patients in Association With HLA–B27: The Pre‐Spondyloarthritis Cohort

Arthritis care & research (2010), 2022-12, Vol.74 (12), p.2076-2084

2022

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Soluble immune complexes shift the TLR-induced cytokine production of distinct polarized human macrophage subsets towards IL-10

PloS one, 2012-04, Vol.7 (4), p.e35994

2012

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ER stress abrogates the immunosuppressive effect of IL-10 on human macrophages through inhibition of STAT3 activation

Inflammation research, 2019-09, Vol.68 (9), p.775-785

2019

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The Transcriptional Coactivator Bob1 Is Associated With Pathologic B Cell Responses in Autoimmune Tissue Inflammation

Arthritis & rheumatology (Hoboken, N.J.), 2017-04, Vol.69 (4), p.750-762

2017

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Recent Advances in the Treatment of Immune-Mediated Inflammatory Diseases

Methods in molecular biology (Clifton, N.J.), 2016, Vol.1371, p.143

2016

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Genetic studies on components of the Wnt signalling pathway and the severity of joint destruction in rheumatoid arthritis

Annals of the rheumatic diseases, 2013-05, Vol.72 (5), p.769-775

2013

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Response to: 'To DAPSA or not to DAPSA? That is not the question' by Schoels et al

Annals of the rheumatic diseases, 2019-07, Vol.78 (7), p.e62

2019

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Comment on: Residual disease activity in psoriatic arthritis: discordance between the rheumatologist's opinion and minimal disease activity measurement: reply

Rheumatology (Oxford, England), 2018-02, Vol.57 (2), p.402-402

2018

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Killer immunoglobulin receptor genes in spondyloarthritis

New treatment paradigms in spondyloarthritis

The inflammatory function of human IgA

The novel cytokine interleukin-36α is expressed in psoriatic and rheumatoid arthritis synovium

The IL-17A-Producing CD8+ T-Cell Population in Psoriatic Lesional Skin Comprises Mucosa-Associated Invariant T Cells and Conventional T Cells

Fc gamma receptor-TLR cross-talk elicits pro-inflammatory cytokine production by human M2 macrophages

Intimal lining layer macrophages but not synovial sublining macrophages display an IL-10 polarized-like phenotype in chronic synovitis

C-Reactive Protein Promotes Inflammation through FcγR-Induced Glycolytic Reprogramming of Human Macrophages

IgG Subclasses Shape Cytokine Responses by Human Myeloid Immune Cells through Differential Metabolic Reprogramming

Identification and characterisation of citrullinated antigen-specific B cells in peripheral blood of patients with rheumatoid arthritis

Interleukin‐17A Is Produced by CD4+ but Not CD8+ T Cells in Synovial Fluid Following T Cell Receptor Activation and Regulates Different Inflammatory Mediators Compared to Tumor Necrosis Factor in a Model of Psoriatic Arthritis Synovitis

Expansion of Interleukin‐22– and Granulocyte–Macrophage Colony‐Stimulating Factor–Expressing, but Not Interleukin‐17A–Expressing, Group 3 Innate Lymphoid Cells in the Inflamed Joints of Patients With Spondyloarthritis

Transmembrane TNF drives osteoproliferative joint inflammation reminiscent of human spondyloarthritis

C-Reactive Protein Controls IL-23 Production by Human Monocytes

Anti-IL-17A treatment reduces serum inflammatory, angiogenic and tissue remodeling biomarkers accompanied by less synovial high endothelial venules in peripheral spondyloarthritis

FcαRI co-stimulation converts human intestinal CD103 + dendritic cells into pro-inflammatory cells through glycolytic reprogramming

Residual disease activity in psoriatic arthritis: discordance between the rheumatologist's opinion and minimal disease activity measurement

Progression From Subclinical Inflammation to Overt Spondyloarthritis in First‐Degree Relatives of Patients in Association With HLA–B27: The Pre‐Spondyloarthritis Cohort

Soluble immune complexes shift the TLR-induced cytokine production of distinct polarized human macrophage subsets towards IL-10

ER stress abrogates the immunosuppressive effect of IL-10 on human macrophages through inhibition of STAT3 activation

The Transcriptional Coactivator Bob1 Is Associated With Pathologic B Cell Responses in Autoimmune Tissue Inflammation

Recent Advances in the Treatment of Immune-Mediated Inflammatory Diseases

Genetic studies on components of the Wnt signalling pathway and the severity of joint destruction in rheumatoid arthritis

Response to: 'To DAPSA or not to DAPSA? That is not the question' by Schoels et al

Comment on: Residual disease activity in psoriatic arthritis: discordance between the rheumatologist's opinion and minimal disease activity measurement: reply

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