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Akt1 is required for physiological cardiac growth

Circulation (New York, N.Y.), 2006-05, Vol.113 (17), p.2097-2104

2006

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Reversion of cardiac dysfunction by a novel orally available calcium/calmodulin-dependent protein kinase II inhibitor, RA306, in a genetic model of dilated cardiomyopathy

Cardiovascular research, 2020-02, Vol.116 (2), p.329-338

2020

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First‐in‐human study of the safety, tolerability, pharmacokinetics and pharmacodynamics of single and multiple oral doses of SAR247799, a selective G‐protein‐biased sphingosine‐1 phosphate receptor‐1 agonist for endothelial protection

British journal of clinical pharmacology, 2021-02, Vol.87 (2), p.598-611

2021

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Raf-1 kinase is required for cardiac hypertrophy and cardiomyocyte survival in response to pressure overload

Circulation (New York, N.Y.), 2004-08, Vol.110 (6), p.718-723

2004

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Ras-Dependent Induction of Cellular Responses by Constitutively Active Phosphatidylinositol-3 Kinase

Science (American Association for the Advancement of Science), 1995-04, Vol.268 (5207), p.100-102

1995

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The role of the Grb2-p38 MAPK signaling pathway in cardiac hypertrophy and fibrosis

The Journal of clinical investigation, 2003-03, Vol.111 (6), p.833-841

2003

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RGS4 Controls Renal Blood Flow and Inhibits Cyclosporine‐Mediated Nephrotoxicity

American journal of transplantation, 2010-02, Vol.10 (2), p.231-241

2010

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HOX11 interacts with protein phosphatases PP2A and PP1 and disrupts a G2/M cell-cycle checkpoint

Nature (London), 1997-01, Vol.385 (6615), p.454-458

1997

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RGS4 causes increased mortality and reduced cardiac hypertrophy in response to pressure overload

The Journal of clinical investigation, 1999-09, Vol.104 (5), p.567-576

1999

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Activation of Raf-1 by 14-3-3 proteins

Nature (London), 1994-10, Vol.371 (6498), p.612-614

1994

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Role of 14-3-3-Mediated p38 Mitogen-Activated Protein Kinase Inhibition in Cardiac Myocyte Survival

Circulation research, 2003-11, Vol.93 (11), p.1026-1028

2003

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Akt2: A Critical Regulator of Cardiomyocyte Survival and Metabolism

Pediatric cardiology, 2011-03, Vol.32 (3), p.317-322

Muslin, Anthony J.

2011

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Endothelial‐protective effects of a G‐protein‐biased sphingosine‐1 phosphate receptor‐1 agonist, SAR247799, in type‐2 diabetes rats and a randomized placebo‐controlled patient trial

British journal of clinical pharmacology, 2021-05, Vol.87 (5), p.2303-2320

2021

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RGS4 inhibits G-protein signaling in cardiomyocytes

Circulation (New York, N.Y.), 1999-01, Vol.99 (3), p.441-447

1999

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G-Protein Signaling Participates in the Development of Diabetic Cardiomyopathy

Diabetes (New York, N.Y.), 2004-12, Vol.53 (12), p.3082-3090

2004

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TRB3 Function in Cardiac Endoplasmic Reticulum Stress

Circulation research, 2010-05, Vol.106 (9), p.1516-1523

2010

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Hypertension and prolonged vasoconstrictor signaling in RGS2-deficient mice

The Journal of clinical investigation, 2003-02, Vol.111 (4), p.445-452

2003

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Grb2 is required for atherosclerotic lesion formation

Arteriosclerosis, thrombosis, and vascular biology, 2007-06, Vol.27 (6), p.1361-1367

2007

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New insights into cardiac aging

Circulation (New York, N.Y.), 2009-10, Vol.120 (17), p.1654-1656

Muslin, Anthony J

2009

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Modulation of doxorubicin-induced cardiac dysfunction in dominant-negative p38α mitogen-activated protein kinase mice

Free radical biology & medicine, 2010-11, Vol.49 (9), p.1422-1431

2010

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14-3-3 protein regulates Ask1 signaling and protects against diabetic cardiomyopathy

Biochemical pharmacology, 2008-05, Vol.75 (9), p.1797-1806

2008

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Grb2 is required for the development of neointima in response to vascular injury

Arteriosclerosis, thrombosis, and vascular biology, 2003-10, Vol.23 (10), p.1788-1793

2003

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The physician scientist training program in internal medicine at Washington University School of Medicine

Academic medicine, 2009-04, Vol.84 (4), p.468-471

2009

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Road Rage: Cardiac Rab1 and ER-to-Golgi Traffic

Circulation research, 2001-12, Vol.89 (12), p.1087-1088

Muslin, Anthony J

2001

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Requirement for p38 Mitogen-Activated Protein Kinase Activity in Neointima Formation After Vascular Injury

Circulation (New York, N.Y.), 2008-08, Vol.118 (6), p.658-666

2008

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Akt1 is required for physiological cardiac growth

Reversion of cardiac dysfunction by a novel orally available calcium/calmodulin-dependent protein kinase II inhibitor, RA306, in a genetic model of dilated cardiomyopathy

First‐in‐human study of the safety, tolerability, pharmacokinetics and pharmacodynamics of single and multiple oral doses of SAR247799, a selective G‐protein‐biased sphingosine‐1 phosphate receptor‐1 agonist for endothelial protection

Raf-1 kinase is required for cardiac hypertrophy and cardiomyocyte survival in response to pressure overload

Ras-Dependent Induction of Cellular Responses by Constitutively Active Phosphatidylinositol-3 Kinase

The role of the Grb2-p38 MAPK signaling pathway in cardiac hypertrophy and fibrosis

RGS4 Controls Renal Blood Flow and Inhibits Cyclosporine‐Mediated Nephrotoxicity

HOX11 interacts with protein phosphatases PP2A and PP1 and disrupts a G2/M cell-cycle checkpoint

RGS4 causes increased mortality and reduced cardiac hypertrophy in response to pressure overload

Activation of Raf-1 by 14-3-3 proteins

Role of 14-3-3-Mediated p38 Mitogen-Activated Protein Kinase Inhibition in Cardiac Myocyte Survival

Akt2: A Critical Regulator of Cardiomyocyte Survival and Metabolism

Endothelial‐protective effects of a G‐protein‐biased sphingosine‐1 phosphate receptor‐1 agonist, SAR247799, in type‐2 diabetes rats and a randomized placebo‐controlled patient trial

RGS4 inhibits G-protein signaling in cardiomyocytes

G-Protein Signaling Participates in the Development of Diabetic Cardiomyopathy

TRB3 Function in Cardiac Endoplasmic Reticulum Stress

Hypertension and prolonged vasoconstrictor signaling in RGS2-deficient mice

Grb2 is required for atherosclerotic lesion formation

New insights into cardiac aging

Modulation of doxorubicin-induced cardiac dysfunction in dominant-negative p38α mitogen-activated protein kinase mice

14-3-3 protein regulates Ask1 signaling and protects against diabetic cardiomyopathy

Grb2 is required for the development of neointima in response to vascular injury

The physician scientist training program in internal medicine at Washington University School of Medicine

Road Rage: Cardiac Rab1 and ER-to-Golgi Traffic

Requirement for p38 Mitogen-Activated Protein Kinase Activity in Neointima Formation After Vascular Injury

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