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Low BCL-xL expression in triple-negative breast cancer cells favors chemotherapy efficacy, and this effect is limited by cancer-associated fibroblasts

Scientific reports, 2024-06, Vol.14 (1), p.14177-10, Article 14177

2024

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Stress in the metastatic journey - the role of cell communication and clustering in breast cancer progression and treatment resistance

Disease models & mechanisms, 2024-03, Vol.17 (3)

2024

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Targeting of MCL-1 in breast cancer-associated fibroblasts reverses their myofibroblastic phenotype and pro-invasive properties

Cell death & disease, 2022-09, Vol.13 (9), p.787-787, Article 787

2022

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miR-491-5p-induced apoptosis in ovarian carcinoma depends on the direct inhibition of both BCL-XL and EGFR leading to BIM activation

Cell death & disease, 2014-10, Vol.5 (10), p.e1445-e1445

2014

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Targeting of BCL-2 Family Members during Anticancer Treatment: A Necessary Compromise between Individual Cell and Ecosystemic Responses?

Biomolecules (Basel, Switzerland), 2020-07, Vol.10 (8), p.1109

2020

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Tight Sequestration of BH3 Proteins by BCL-xL at Subcellular Membranes Contributes to Apoptotic Resistance

Cell reports (Cambridge), 2016-12, Vol.17 (12), p.3347-3358

2016

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DNA methylation signal has a major role in the response of human breast cancer cells to the microenvironment

Oncogenesis (New York, NY), 2017-10, Vol.6 (10), p.e390-e390

2017

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Constitutive p53 heightens mitochondrial apoptotic priming and favors cell death induction by BH3 mimetic inhibitors of BCL-xL

Cell death & disease, 2016-02, Vol.7 (2), p.e2083-e2083

2016

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TOM22, a core component of the mitochondria outer membrane protein translocation pore, is a mitochondrial receptor for the proapoptotic protein Bax

Cell death and differentiation, 2007-04, Vol.14 (4), p.785-794

2007

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Bcl-xL controls a switch between cell death modes during mitotic arrest

Cell death & disease, 2014-06, Vol.5 (6), p.e1291-e1291

2014

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Mitochondria at Center of Exchanges between Cancer Cells and Cancer-Associated Fibroblasts during Tumor Progression

Cancers, 2020-10, Vol.12 (10), p.3017

2020

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pRb/E2F-1-mediated caspase-dependent induction of Noxa amplifies the apoptotic effects of the Bcl-2/Bcl-xL inhibitor ABT-737

Cell death and differentiation, 2013-05, Vol.20 (5), p.755-764

2013

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Sensitization of osteosarcoma cells to apoptosis by oncostatin M depends on STAT5 and p53

Oncogene, 2007-10, Vol.26 (46), p.6653-6664

2007

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Mitochondrial targeting by use of lipid nanocapsules loaded with SV30, an analogue of the small-molecule Bcl-2 inhibitor HA14-1

Journal of controlled release, 2011-04, Vol.151 (1), p.74-82

2011

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Development of an absolute assignment predictor for triple-negative breast cancer subtyping using machine learning approaches

Computers in biology and medicine, 2021-02, Vol.129, p.104171-104171, Article 104171

2021

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Concomitant intensive chemoradiotherapy induction in non-metastatic inflammatory breast cancer: long-term follow-up

British journal of cancer, 2007-10, Vol.97 (7), p.883-887

2007

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E2F1 interacts with BCL‐xL and regulates its subcellular localization dynamics to trigger cell death

EMBO reports, 2018-02, Vol.19 (2), p.234-243

2018

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472 Identification of a Role for Anti-apoptic Bcl-xL in Breast Cancer Initiating Cell Maintenance

European journal of cancer (1990), 2012-07, Vol.48, p.S113-S114

2012

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Impact of the cGAS/STING pathway on the tumour ecosystem under chemotherapeutic pressure in triple negative breast cancer

Journal of comparative pathology, 2024-04, Vol.210, p.60-60

2024

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P53 and PTEN expression contribute to the inhibition of EGFR downstream signaling pathway by cetuximab

Cancer gene therapy, 2009-06, Vol.16 (6), p.498-507

2009

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Cell cycle and cell death associates in western France

Cell death and differentiation, 2007-05, Vol.14 (5), p.1066-1067

2007

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Caspase 8: The killer you can't live without

Nature medicine, 2000-05, Vol.6 (5), p.498-500

2000

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Mitochondria as the target of the pro-apoptotic protein Bax

Biochimica et biophysica acta, 2006-09, Vol.1757 (9), p.1301-1311

2006

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The First α Helix of Bax Plays a Necessary Role in Its Ligand-Induced Activation by the BH3-Only Proteins Bid and PUMA

Molecular cell, 2004-12, Vol.16 (5), p.807-818

2004

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Low BCL-xL expression in triple-negative breast cancer cells favors chemotherapy efficacy, and this effect is limited by cancer-associated fibroblasts

Stress in the metastatic journey - the role of cell communication and clustering in breast cancer progression and treatment resistance

Targeting of MCL-1 in breast cancer-associated fibroblasts reverses their myofibroblastic phenotype and pro-invasive properties

miR-491-5p-induced apoptosis in ovarian carcinoma depends on the direct inhibition of both BCL-XL and EGFR leading to BIM activation

Targeting of BCL-2 Family Members during Anticancer Treatment: A Necessary Compromise between Individual Cell and Ecosystemic Responses?

Tight Sequestration of BH3 Proteins by BCL-xL at Subcellular Membranes Contributes to Apoptotic Resistance

DNA methylation signal has a major role in the response of human breast cancer cells to the microenvironment

Constitutive p53 heightens mitochondrial apoptotic priming and favors cell death induction by BH3 mimetic inhibitors of BCL-xL

TOM22, a core component of the mitochondria outer membrane protein translocation pore, is a mitochondrial receptor for the proapoptotic protein Bax

Bcl-xL controls a switch between cell death modes during mitotic arrest

Keeping Cell Death Alive: An Introduction into the French Cell Death Research Network

Mitochondria at Center of Exchanges between Cancer Cells and Cancer-Associated Fibroblasts during Tumor Progression

pRb/E2F-1-mediated caspase-dependent induction of Noxa amplifies the apoptotic effects of the Bcl-2/Bcl-xL inhibitor ABT-737

Sensitization of osteosarcoma cells to apoptosis by oncostatin M depends on STAT5 and p53

Mitochondrial targeting by use of lipid nanocapsules loaded with SV30, an analogue of the small-molecule Bcl-2 inhibitor HA14-1

Development of an absolute assignment predictor for triple-negative breast cancer subtyping using machine learning approaches

Concomitant intensive chemoradiotherapy induction in non-metastatic inflammatory breast cancer: long-term follow-up

E2F1 interacts with BCL‐xL and regulates its subcellular localization dynamics to trigger cell death

472 Identification of a Role for Anti-apoptic Bcl-xL in Breast Cancer Initiating Cell Maintenance

Impact of the cGAS/STING pathway on the tumour ecosystem under chemotherapeutic pressure in triple negative breast cancer

P53 and PTEN expression contribute to the inhibition of EGFR downstream signaling pathway by cetuximab

Cell cycle and cell death associates in western France

Caspase 8: The killer you can't live without

Mitochondria as the target of the pro-apoptotic protein Bax

The First α Helix of Bax Plays a Necessary Role in Its Ligand-Induced Activation by the BH3-Only Proteins Bid and PUMA

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