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KATP channels and NO dilate redundantly intramuscular arterioles during electrical stimulation of the skeletal muscle in mice

Pflügers Archiv, 2021-11, Vol.473 (11), p.1795-1806

2021

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Endothelium-Derived Hyperpolarizing Factor and Myoendothelial Coupling: The in vivo Perspective

Frontiers in physiology, 2020-12, Vol.11, p.602930-602930

2020

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ESC Working Group on Coronary Pathophysiology and Microcirculation position paper on ‘coronary microvascular dysfunction in cardiovascular disease’

Cardiovascular research, 2020-03, Vol.116 (4), p.741-755

2020

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Magnetofection—A highly efficient tool for antisense oligonucleotide delivery in vitro and in vivo

Molecular therapy, 2003-05, Vol.7 (5), p.700-710

2003

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Connexins and gap junctions in the EDHF phenomenon and conducted vasomotor responses

Pflügers Archiv, 2010-05, Vol.459 (6), p.897-914

2010

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ADAMTS-7 inhibits re-endothelialization of injured arteries and promotes vascular remodeling through cleavage of thrombospondin-1

Circulation (New York, N.Y.), 2015-03, Vol.131 (13), p.1191-1201

2015

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Expression of Connexin43 Stimulates Endothelial Angiogenesis Independently of Gap Junctional Communication In Vitro

International journal of molecular sciences, 2021-07, Vol.22 (14), p.7400

2021

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Connexin40 Is Essential for the Pressure Control of Renin Synthesis and Secretion

Circulation research, 2007-03, Vol.100 (4), p.556-563

2007

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Genetic Deficit of SK3 and IK1 Channels Disrupts the Endothelium-Derived Hyperpolarizing Factor Vasodilator Pathway and Causes Hypertension

Circulation (New York, N.Y.), 2009-05, Vol.119 (17), p.2323-2332

2009

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Transgenic Mice for cGMP Imaging

Circulation research, 2013-08, Vol.113 (4), p.365-371

2013

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Flow increase is decisive to initiate angiogenesis in veins exposed to altered hemodynamics

PloS one, 2015-01, Vol.10 (1), p.e0117407-e0117407

2015

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Impaired Endothelium-Derived Hyperpolarizing Factor–Mediated Dilations and Increased Blood Pressure in Mice Deficient of the Intermediate-Conductance Ca2+-Activated K+ Channel

Circulation research, 2006-09, Vol.99 (5), p.537-544

2006

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Connexin45 is expressed in vascular smooth muscle but its function remains elusive

PloS one, 2012-07, Vol.7 (7), p.e42287-e42287

2012

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Cardiovascular Functions of Ena/VASP Proteins: Past, Present and Beyond

Cells (Basel, Switzerland), 2023-06, Vol.12 (13), p.1740

2023

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High flow conditions increase connexin43 expression in a rat arteriovenous and angioinductive loop model

PloS one, 2013-11, Vol.8 (11), p.e78782-e78782

2013

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Impaired Conduction of Vasodilation Along Arterioles in Connexin40-Deficient Mice

Circulation research, 2000-03, Vol.86 (6), p.649-655

2000

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Nitric oxide-induced decrease in calcium sensitivity of resistance arteries is attributable to activation of the myosin light chain phosphatase and antagonized by the RhoA/Rho kinase pathway

Circulation (New York, N.Y.), 2003-06, Vol.107 (24), p.3081-3087

2003

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Different pathways with distinct properties conduct dilations in the microcirculation in vivo

Cardiovascular research, 2010-02, Vol.85 (3), p.604-613

de Wit, Cor

2010

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EDHF and gap junctions: important regulators of vascular tone within the microcirculation

Current pharmaceutical biotechnology, 2007-02, Vol.8 (1), p.11-25

2007

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Retinoic acid treatment protects MRL/lpr lupus mice from the development of glomerular disease

Kidney international, 2004-09, Vol.66 (3), p.1018-1028

2004

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Semiautomatic Quantification of Angiogenesis

The Journal of surgical research, 2010-07, Vol.162 (1), p.132-139

2010

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Depression and coronary heart disease: 2018 position paper of the ESC working group on coronary pathophysiology and microcirculation

European heart journal, 2020-05, Vol.41 (17), p.1687-1696

2020

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Distinct Endothelium-Derived Hyperpolarizing Factors Emerge In Vitro and In Vivo and Are Mediated in Part via Connexin 40–Dependent Myoendothelial Coupling

Hypertension (Dallas, Tex. 1979), 2011-04, Vol.57 (4), p.802-808

2011

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Restoring a critical element in renin-producing cells: connexin40 hits the brakes on renin release

Hypertension (Dallas, Tex. 1979), 2014-06, Vol.63 (6), p.1161-1162

de Wit, Cor

2014

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Coronary vascular regulation, remodelling, and collateralization: mechanisms and clinical implications on behalf of the working group on coronary pathophysiology and microcirculation

European heart journal, 2015-12, Vol.36 (45), p.3134-3146

2015

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KATP channels and NO dilate redundantly intramuscular arterioles during electrical stimulation of the skeletal muscle in mice

Endothelium-Derived Hyperpolarizing Factor and Myoendothelial Coupling: The in vivo Perspective

ESC Working Group on Coronary Pathophysiology and Microcirculation position paper on ‘coronary microvascular dysfunction in cardiovascular disease’

Magnetofection—A highly efficient tool for antisense oligonucleotide delivery in vitro and in vivo

Connexins and gap junctions in the EDHF phenomenon and conducted vasomotor responses

ADAMTS-7 inhibits re-endothelialization of injured arteries and promotes vascular remodeling through cleavage of thrombospondin-1

Expression of Connexin43 Stimulates Endothelial Angiogenesis Independently of Gap Junctional Communication In Vitro

Connexin40 Is Essential for the Pressure Control of Renin Synthesis and Secretion

Genetic Deficit of SK3 and IK1 Channels Disrupts the Endothelium-Derived Hyperpolarizing Factor Vasodilator Pathway and Causes Hypertension

Transgenic Mice for cGMP Imaging

Flow increase is decisive to initiate angiogenesis in veins exposed to altered hemodynamics

Impaired Endothelium-Derived Hyperpolarizing Factor–Mediated Dilations and Increased Blood Pressure in Mice Deficient of the Intermediate-Conductance Ca2+-Activated K+ Channel

Connexin45 is expressed in vascular smooth muscle but its function remains elusive

Cardiovascular Functions of Ena/VASP Proteins: Past, Present and Beyond

High flow conditions increase connexin43 expression in a rat arteriovenous and angioinductive loop model

Impaired Conduction of Vasodilation Along Arterioles in Connexin40-Deficient Mice

Nitric oxide-induced decrease in calcium sensitivity of resistance arteries is attributable to activation of the myosin light chain phosphatase and antagonized by the RhoA/Rho kinase pathway

Different pathways with distinct properties conduct dilations in the microcirculation in vivo

EDHF and gap junctions: important regulators of vascular tone within the microcirculation

Retinoic acid treatment protects MRL/lpr lupus mice from the development of glomerular disease

Semiautomatic Quantification of Angiogenesis

Depression and coronary heart disease: 2018 position paper of the ESC working group on coronary pathophysiology and microcirculation

Distinct Endothelium-Derived Hyperpolarizing Factors Emerge In Vitro and In Vivo and Are Mediated in Part via Connexin 40–Dependent Myoendothelial Coupling

Restoring a critical element in renin-producing cells: connexin40 hits the brakes on renin release

Coronary vascular regulation, remodelling, and collateralization: mechanisms and clinical implications on behalf of the working group on coronary pathophysiology and microcirculation

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