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Congenital myasthenic syndromes: pathogenesis, diagnosis, and treatment

Lancet neurology, 2015-04, Vol.14 (4), p.420-434

2015

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Principal pathway coupling agonist binding to channel gating in nicotinic receptors

Nature, 2005-11, Vol.438 (7065), p.243-247

2005

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Recent advances in Cys-loop receptor structure and function

Nature, 2006-03, Vol.440 (7083), p.448-455

2006

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Genetic Variant in Nicotinic Receptor α4-Subunit Causes Sleep-Related Hyperkinetic Epilepsy via Increased Channel Opening

International journal of molecular sciences, 2022-10, Vol.23 (20), p.12124

2022

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Galantamine-memantine hybrids for Alzheimer's disease: The influence of linker rigidity in biological activity and pharmacokinetic properties

European journal of medicinal chemistry, 2023-12, Vol.261, p.115803-115803, Article 115803

2023

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Structure and gating mechanism of the α7 nicotinic acetylcholine receptor

Cell, 2021-04, Vol.184 (8), p.2121-2134.e13

2021

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Binding to Gating Transduction in Nicotinic Receptors: Cys-Loop Energetically Couples to Pre-M1 and M2-M3 Regions

The Journal of neuroscience, 2009-03, Vol.29 (10), p.3189-3199

2009

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Sleuthing molecular targets for neurological diseases at the neuromuscular junction

Nature reviews. Neuroscience, 2003-05, Vol.4 (5), p.339-352

2003

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Current understanding of congenital myasthenic syndromes

Current opinion in pharmacology, 2005-06, Vol.5 (3), p.308-321

2005

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Nicotinic acetylcholine receptors at the single‐channel level

British journal of pharmacology, 2018-06, Vol.175 (11), p.1789-1804

2018

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Greenland ice sheet mass balance assessed by PROMICE (1995–2015)

Geological Survey of Denmark and Greenland bulletin, 2019-07, Vol.43

2019

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Targeted molecular dynamics study of C-loop closure and channel gating in nicotinic receptors

PLoS computational biology, 2006-09, Vol.2 (9), p.e134-e134

2006

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Highly fatal fast-channel syndrome caused by AChR ε subunit mutation at the agonist binding site

Neurology, 2012-07, Vol.79 (5), p.449

2012

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Impact on AChR open channel noise by pore-peripheral salt bridge depends on voltage and divalent cations

Biophysical journal, 2023-06, Vol.122 (12), p.2430-2444

2023

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Structural bases for stoichiometry‐selective calcium potentiation of a neuronal nicotinic receptor

British journal of pharmacology, 2024-07, Vol.181 (13), p.1973-1992

2024

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Stoichiometry‐selective modulation of α4β2 nicotinic ACh receptors by divalent cations

British journal of pharmacology, 2022-04, Vol.179 (7), p.1353-1370

2022

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On the hot working of FeSi ferritic steels

Materials science & engineering. A, Structural materials : properties, microstructure and processing, 2014-06, Vol.606, p.127-138

2014

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Coupling of agonist binding to channel gating in an ACh-binding protein linked to an ion channel

Nature, 2004-08, Vol.430 (7002), p.896-900

2004

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Novel Modulation of Neuronal Nicotinic Acetylcholine Receptors by Association with the Endogenous Prototoxin lynx1

Neuron (Cambridge, Mass.), 2002-03, Vol.33 (6), p.893-903

2002

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Structural basis for α-bungarotoxin insensitivity of neuronal nicotinic acetylcholine receptors

Neuropharmacology, 2019-12, Vol.160, p.107660-107660, Article 107660

2019

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Proton beam radiation therapy results in significantly reduced toxicity compared with intensity-modulated radiation therapy for head and neck tumors that require ipsilateral radiation

Radiotherapy and oncology, 2016-02, Vol.118 (2), p.286-292

2016

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Congenital myasthenic syndromes: pathogenesis, diagnosis, and treatment

Lancet neurology, 2015-05, Vol.14 (5), p.461-461

2015

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Control of cation permeation through the nicotinic receptor channel

PLoS computational biology, 2008-02, Vol.4 (2), p.e41-e41

2008

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Structural determinants of peripheral O-arylcarbamate FAAH inhibitors render them dual substrates for Abcb1 and Abcg2 and restrict their access to the brain

Pharmacological research, 2014-09, Vol.87, p.87-93

2014

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Structural mechanisms of α7 nicotinic receptor allosteric modulation and activation

Cell, 2024-02, Vol.187 (5), p.1160-1176.e21

2024

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Congenital myasthenic syndromes: pathogenesis, diagnosis, and treatment

Principal pathway coupling agonist binding to channel gating in nicotinic receptors

Recent advances in Cys-loop receptor structure and function

Genetic Variant in Nicotinic Receptor α4-Subunit Causes Sleep-Related Hyperkinetic Epilepsy via Increased Channel Opening

Galantamine-memantine hybrids for Alzheimer's disease: The influence of linker rigidity in biological activity and pharmacokinetic properties

Structure and gating mechanism of the α7 nicotinic acetylcholine receptor

Binding to Gating Transduction in Nicotinic Receptors: Cys-Loop Energetically Couples to Pre-M1 and M2-M3 Regions

Sleuthing molecular targets for neurological diseases at the neuromuscular junction

Current understanding of congenital myasthenic syndromes

Nicotinic acetylcholine receptors at the single‐channel level

Greenland ice sheet mass balance assessed by PROMICE (1995–2015)

Targeted molecular dynamics study of C-loop closure and channel gating in nicotinic receptors

Highly fatal fast-channel syndrome caused by AChR ε subunit mutation at the agonist binding site

Impact on AChR open channel noise by pore-peripheral salt bridge depends on voltage and divalent cations

Structural bases for stoichiometry‐selective calcium potentiation of a neuronal nicotinic receptor

Stoichiometry‐selective modulation of α4β2 nicotinic ACh receptors by divalent cations

On the hot working of FeSi ferritic steels

Coupling of agonist binding to channel gating in an ACh-binding protein linked to an ion channel

Novel Modulation of Neuronal Nicotinic Acetylcholine Receptors by Association with the Endogenous Prototoxin lynx1

Structural basis for α-bungarotoxin insensitivity of neuronal nicotinic acetylcholine receptors

Proton beam radiation therapy results in significantly reduced toxicity compared with intensity-modulated radiation therapy for head and neck tumors that require ipsilateral radiation

Congenital myasthenic syndromes: pathogenesis, diagnosis, and treatment

Control of cation permeation through the nicotinic receptor channel

Structural determinants of peripheral O-arylcarbamate FAAH inhibitors render them dual substrates for Abcb1 and Abcg2 and restrict their access to the brain

Structural mechanisms of α7 nicotinic receptor allosteric modulation and activation

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