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Maternal emulsifier consumption programs offspring metabolic and neuropsychological health in mice

PLoS biology, 2023-08, Vol.21 (8), p.e3002171-e3002171

2023

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Pro-opiomelanocortin (POMC) neuron translatome signatures underlying obesogenic gestational malprogramming in mice

Molecular metabolism (Germany), 2020-06, Vol.36, p.100963-100963, Article 100963

2020

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Peroxisome Proliferator-Activated Receptor (PPAR)β /δ: A New Potential Therapeutic Target for the Treatment of Metabolic Syndrome

Current molecular pharmacology, 2009-01, Vol.2 (1), p.46-55

2009

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The peroxisome proliferator-activated receptor β/δ (PPARβ/δ) agonist GW501516 prevents TNF-α-induced NF-κB activation in human HaCaT cells by reducing p65 acetylation through AMPK and SIRT1

Biochemical pharmacology, 2011-02, Vol.81 (4), p.534-543

2011

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TLR2 and TLR9 modulate enteric nervous system inflammatory responses to lipopolysaccharide

Journal of neuroinflammation, 2016-08, Vol.13 (1), p.187-187, Article 187

2016

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Rat Embryo Enteric Nervous System Primary Cultures and Jug2 Enteric Glial Cells Respond to Bacterial Cell Components Through Up-Regulation of TLR2

Gastroenterology (New York, N.Y. 1943), 2011, Vol.140 (5), p.S-521-S-521

2011

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Abnormal type I collagen post-translational modification and crosslinking in a cyclophilin B KO mouse model of recessive osteogenesis imperfecta

PLoS genetics, 2014-06, Vol.10 (6), p.e1004465-e1004465

2014

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Absence of the ER Cation Channel TMEM38B/TRIC-B Disrupts Intracellular Calcium Homeostasis and Dysregulates Collagen Synthesis in Recessive Osteogenesis Imperfecta

PLoS genetics, 2016-07, Vol.12 (7), p.e1006156-e1006156

2016

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O01 Genetic risk factors associated with increased risk of uveitis in patients with juvenile idiopathic arthritis

Rheumatology (Oxford, England), 2021-04, Vol.60 (Supplement_1)

2021

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Prolyl 3-hydroxylase 1 deficiency causes a recessive metabolic bone disorder resembling lethal/severe osteogenesis imperfecta

Nature genetics, 2007-03, Vol.39 (3), p.359-365

2007

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No evidence that genetic predictors of susceptibility predict changes in core outcomes in JIA

Rheumatology (Oxford, England), 2022-10, Vol.61 (10), p.4136-4144

2022

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Substitution of murine type I collagen A1 3-hydroxylation site alters matrix structure but does not recapitulate osteogenesis imperfecta bone dysplasia

Matrix biology, 2020-08, Vol.90, p.20-39

2020

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Erratum: Corrigendum: Prolyl 3-hydroxylase 1 deficiency causes a recessive metabolic bone disorder resembling lethal/severe osteogenesis imperfecta

Nature genetics, 2008-07, Vol.40 (7), p.927-927

2008

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Corrigendum: Prolyl 3-hydroxylase 1 deficiency causes a recessive metabolic bone disorder resembling lethal/severe osteogenesis imperfecta

Nature genetics, 2008-07, Vol.40 (7), p.927-927

2008

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Correction: Corrigendum: Prolyl 3-hydroxylase 1 deficiency causes a recessive metabolic bone disorder resembling lethal/severe osteogenesis imperfecta

Nature genetics, 2008, Vol.40 (7), p.927-927

2008

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Sociocultural Beliefs Related to Sex among Mexican American Adolescents

Hispanic journal of behavioral sciences, 1998-02, Vol.20 (1), p.60-82

1998

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Maternal emulsifier consumption programs offspring metabolic and neuropsychological health in mice

Pro-opiomelanocortin (POMC) neuron translatome signatures underlying obesogenic gestational malprogramming in mice

Peroxisome Proliferator-Activated Receptor (PPAR)β /δ: A New Potential Therapeutic Target for the Treatment of Metabolic Syndrome

Mitochondrial cristae-remodeling protein OPA1 in POMC neurons couples Ca2+ homeostasis with adipose tissue lipolysis

The peroxisome proliferator-activated receptor β/δ (PPARβ/δ) agonist GW501516 prevents TNF-α-induced NF-κB activation in human HaCaT cells by reducing p65 acetylation through AMPK and SIRT1

TLR2 and TLR9 modulate enteric nervous system inflammatory responses to lipopolysaccharide

Hypothalamic pregnenolone mediates recognition memory in the context of metabolic disorders

Mitochondrial cristae-remodeling protein OPA1 in POMC neurons couples Ca 2+ homeostasis with adipose tissue lipolysis

Rat Embryo Enteric Nervous System Primary Cultures and Jug2 Enteric Glial Cells Respond to Bacterial Cell Components Through Up-Regulation of TLR2

Abnormal type I collagen post-translational modification and crosslinking in a cyclophilin B KO mouse model of recessive osteogenesis imperfecta

Absence of the ER Cation Channel TMEM38B/TRIC-B Disrupts Intracellular Calcium Homeostasis and Dysregulates Collagen Synthesis in Recessive Osteogenesis Imperfecta

O01 Genetic risk factors associated with increased risk of uveitis in patients with juvenile idiopathic arthritis

Prolyl 3-hydroxylase 1 deficiency causes a recessive metabolic bone disorder resembling lethal/severe osteogenesis imperfecta

No evidence that genetic predictors of susceptibility predict changes in core outcomes in JIA

Substitution of murine type I collagen A1 3-hydroxylation site alters matrix structure but does not recapitulate osteogenesis imperfecta bone dysplasia

Erratum: Corrigendum: Prolyl 3-hydroxylase 1 deficiency causes a recessive metabolic bone disorder resembling lethal/severe osteogenesis imperfecta

Corrigendum: Prolyl 3-hydroxylase 1 deficiency causes a recessive metabolic bone disorder resembling lethal/severe osteogenesis imperfecta

Correction: Corrigendum: Prolyl 3-hydroxylase 1 deficiency causes a recessive metabolic bone disorder resembling lethal/severe osteogenesis imperfecta

Sociocultural Beliefs Related to Sex among Mexican American Adolescents

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