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Validation of next generation sequencing technologies in comparison to current diagnostic gold standards for BRAF, EGFR and KRAS mutational analysis

PloS one, 2013-07, Vol.8 (7), p.e69604-e69604

2013

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TP53 Mutations Identified Using NGS Comprise the Overwhelming Majority of TP53 Disruptions in CLL: Results From a Multicentre Study

Frontiers in oncology, 2022-06, Vol.12, p.909615-909615

2022

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Significance of NPM1 Gene Mutations in AML

International journal of molecular sciences, 2021-09, Vol.22 (18), p.10040

2021

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Absence of CALR Mutations in Idiopathic Erythrocytosis Patients with Low Serum Erythropoietin Levels

Acta haematologica, 2018-01, Vol.139 (4), p.217-219

2018

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Glucose-induced oxidative stress in mesangial cells

Kidney international, 2002-02, Vol.61 (2), p.599-608

2002

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PICan: An integromics framework for dynamic cancer biomarker discovery

Molecular oncology, 2015-06, Vol.9 (6), p.1234-1240

2015

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Multidrug resistance gene expression and ABCB1 SNPs in plasma cell myeloma

Leukemia research, 2011-11, Vol.35 (11), p.1457-1463

2011

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Restoration of glutathione levels in vascular smooth muscle cells exposed to high glucose conditions

Free radical biology & medicine, 2001-11, Vol.31 (10), p.1149-1155

2001

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V(H)3-48 and V(H)3-53, as well as V(H)3-21, gene rearrangements define unique subgroups in CLL and are associated with biased lambda light chain restriction, homologous LCDR3 sequences and poor prognosis

Leukemia research, 2007-02, Vol.31 (2), p.231-234

2007

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The impact of next generation sequencing technologies on haematological research – A review

Pathogenesis (Oxford), 2015, Vol.2 (1-2), p.9-16

2015

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VH3–48 and VH3–53, as well as VH3–21, gene rearrangements define unique subgroups in CLL and are associated with biased lambda light chain restriction, homologous LCDR3 sequences and poor prognosis

Leukemia research, 2007-02, Vol.31 (2), p.231-234

2007

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V sub(H)3-48 and V sub(H)3-53, as well as V sub(H)3-21, gene rearrangements define unique subgroups in CLL and are associated with biased lambda light chain restriction, homologous LCDR3 sequences and poor prognosis

Leukemia research, 2007-02, Vol.31 (2), p.231-234

2007

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V H3–48 and V H3–53, as well as V H3–21, gene rearrangements define unique subgroups in CLL and are associated with biased lambda light chain restriction, homologous LCDR3 sequences and poor prognosis

Leukemia research, 2007, Vol.31 (2), p.231-234

2007

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VH 3–48 and VH 3–53, as well as VH 3–21, gene rearrangements define unique subgroups in CLL and are associated with biased lambda light chain restriction, homologous LCDR3 sequences and poor prognosis

Leukemia research, 2006, Vol.31 (2), p.231-234

2006

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New complementary perspectives for inpatient physical function assessment: matched clinician-report and patient-report short form measures from the PROMIS adult physical function item bank

Quality of life research, 2022-07, Vol.31 (7), p.2201-2212

2022

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Formation of osteoclast-like cells is suppressed by low frequency, low intensity electric fields

Journal of orthopaedic research, 1996-01, Vol.14 (1), p.7-15

1996

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Cost effectiveness of therapies for atrial fibrillation. A review

PharmacoEconomics, 2000-10, Vol.18 (4), p.317-333

2000

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Tumor necrosis factor α decreases 1,25‐dihydroxyvitamin D3 receptors in osteoblastic ROS 17/2.8 cells

Journal of bone and mineral research, 1993-08, Vol.8 (8), p.997-1003

1993

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1,25‐Dihydroxyvitamin D reduces parathyroid hormone receptor number in ROS 17/2.8 cells and prevents the glucocorticoid‐induced increase in these receptors: Relationship to adenylate cyclase activation

Journal of bone and mineral research, 1991-06, Vol.6 (6), p.631-637

1991

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Rat osteoblasts and ROS 17/2.8 cells contain a similar protein tyrosine phosphatase

Bone and mineral, 1993, Vol.23 (3), p.267-284

1993

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Validation of next generation sequencing technologies in comparison to current diagnostic gold standards for BRAF, EGFR and KRAS mutational analysis

TP53 Mutations Identified Using NGS Comprise the Overwhelming Majority of TP53 Disruptions in CLL: Results From a Multicentre Study

Significance of NPM1 Gene Mutations in AML

Absence of CALR Mutations in Idiopathic Erythrocytosis Patients with Low Serum Erythropoietin Levels

Glucose-induced oxidative stress in mesangial cells

PICan: An integromics framework for dynamic cancer biomarker discovery

Multidrug resistance gene expression and ABCB1 SNPs in plasma cell myeloma

Restoration of glutathione levels in vascular smooth muscle cells exposed to high glucose conditions

V(H)3-48 and V(H)3-53, as well as V(H)3-21, gene rearrangements define unique subgroups in CLL and are associated with biased lambda light chain restriction, homologous LCDR3 sequences and poor prognosis

Impact of the Types and Relative Quantities of IGHV Gene Mutations in Predicting Prognosis of Patients With Chronic Lymphocytic Leukemia

The impact of next generation sequencing technologies on haematological research – A review

VH3–48 and VH3–53, as well as VH3–21, gene rearrangements define unique subgroups in CLL and are associated with biased lambda light chain restriction, homologous LCDR3 sequences and poor prognosis

V sub(H)3-48 and V sub(H)3-53, as well as V sub(H)3-21, gene rearrangements define unique subgroups in CLL and are associated with biased lambda light chain restriction, homologous LCDR3 sequences and poor prognosis

V H3–48 and V H3–53, as well as V H3–21, gene rearrangements define unique subgroups in CLL and are associated with biased lambda light chain restriction, homologous LCDR3 sequences and poor prognosis

VH 3–48 and VH 3–53, as well as VH 3–21, gene rearrangements define unique subgroups in CLL and are associated with biased lambda light chain restriction, homologous LCDR3 sequences and poor prognosis

New complementary perspectives for inpatient physical function assessment: matched clinician-report and patient-report short form measures from the PROMIS adult physical function item bank

Formation of osteoclast-like cells is suppressed by low frequency, low intensity electric fields

Cost effectiveness of therapies for atrial fibrillation. A review

Tumor necrosis factor α decreases 1,25‐dihydroxyvitamin D3 receptors in osteoblastic ROS 17/2.8 cells

1,25‐Dihydroxyvitamin D reduces parathyroid hormone receptor number in ROS 17/2.8 cells and prevents the glucocorticoid‐induced increase in these receptors: Relationship to adenylate cyclase activation

Rat osteoblasts and ROS 17/2.8 cells contain a similar protein tyrosine phosphatase

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