UB Paderborn / Katalog / Suche / in CDI

Genetic and epigenetic features of bilateral Wilms tumor predisposition in patients from the Children's Oncology Group AREN18B5-Q

Nature communications, 2023-12, Vol.14 (1), p.8006-15, Article 8006

2023

Volltextzugriff (PDF)

Genetic changes associated with relapse in favorable histology Wilms tumor: A Children’s Oncology Group AREN03B2 study

Cell reports. Medicine, 2022-06, Vol.3 (6), p.100644, Article 100644

2022

Volltextzugriff (PDF)

Wilm's tumor 1 promotes memory flexibility

Nature communications, 2019-08, Vol.10 (1), p.3756-18, Article 3756

2019

Volltextzugriff (PDF)

A unique subset of low-risk Wilms tumors is characterized by loss of function of TRIM28 (KAP1), a gene critical in early renal development: A Children's Oncology Group study

PloS one, 2018-12, Vol.13 (12), p.e0208936-e0208936

2018

Volltextzugriff (PDF)

A unique subset of low-risk Wilms tumors is characterized by loss of function of TRIM28

PloS one, 2018-12, Vol.13 (12), p.e0208936

2018

Volltextzugriff (PDF)

Knock-in of the Wt1 R394W mutation causes MDS and cooperates with Flt3/ITD to drive aggressive myeloid neoplasms in mice

Oncotarget, 2018-10, Vol.9 (82), p.35313-35326

2018

Volltextzugriff (PDF)

Wt1 directs the lineage specification of sertoli and granulosa cells by repressing Sf1 expression

Development (Cambridge), 2017-01, Vol.144 (1), p.44-53

2017

Volltextzugriff (PDF)

Significance of TP53 Mutation in Wilms Tumors with Diffuse Anaplasia: A Report from the Children's Oncology Group

Clinical cancer research, 2016-11, Vol.22 (22), p.5582-5591

2016

Volltextzugriff (PDF)

Abstract LB-180: The genetic landscape of Wilms tumor

Cancer research (Chicago, Ill.), 2016-07, Vol.76 (14_Supplement), p.LB-180-LB-180

2016

Volltextzugriff (PDF)

Identification of germline DICER1 mutations and loss of heterozygosity in familial Wilms tumour

Journal of medical genetics, 2016-06, Vol.53 (6), p.385-388

2016

Volltextzugriff (PDF)

Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with β-Catenin Activation or Wt1 Ablation and Igf2 Upregulation1

Neoplasia (New York, N.Y.), 2016-02, Vol.18 (2), p.71-81

2016

Volltextzugriff (PDF)

Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with [beta]-Catenin Activation or Wt1 Ablation and Igf2 Upregulation

Neoplasia (New York, N.Y.), 2016-02, Vol.18 (2), p.71-81

2016

Volltextzugriff (PDF)

Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with β-Catenin Activation or Wt1 Ablation and Igf2 Upregulation

Neoplasia (New York, N.Y.), 2016-02, Vol.18 (2), p.71-81

2016

Volltextzugriff (PDF)

MLLT1 YEATS domain mutations in clinically distinctive Favourable Histology Wilms tumours

Nature communications, 2015-12, Vol.6 (1), p.10013, Article 10013

2015

Volltextzugriff (PDF)

A Wilms Tumor 1 (WT1) Mutation Causes Myelodysplastic Syndrome in a Knock-in Mouse Model, and a Mixed Myelodysplastic/Myeloproliferative Neoplam in Double Knock-in Mice with WT1 and FLT3/ITD Mutations

Blood, 2015-12, Vol.126 (23), p.312-312

2015

Volltextzugriff (PDF)

Clinical outcome and biological predictors of relapse following nephrectomy only for very low risk Wilms tumor (VLR WT): A report from Children’s Oncology Group AREN0532

Journal of clinical oncology, 2015-05, Vol.33 (15_suppl), p.10023-10023

2015

Volltextzugriff (PDF)

Reprogramming of Sertoli cells to fetal-like Leydig cells by Wt1 ablation

Proceedings of the National Academy of Sciences - PNAS, 2015-03, Vol.112 (13), p.4003-4008

2015

Volltextzugriff (PDF)

Reprogramming of Sertoli cells to fetal-like Leydig cells byWt1ablation

Proceedings of the National Academy of Sciences - PNAS, 2015-03, Vol.112 (13), p.4003-4008

2015

Volltextzugriff (PDF)

β-Catenin Activation in a Novel Liver Progenitor Cell Type Is Sufficient to Cause Hepatocellular Carcinoma and Hepatoblastoma

Cancer research (Chicago, Ill.), 2014-08, Vol.74 (16), p.4515-4525

2014

Volltextzugriff (PDF)

Characterization of the Inflammatory Microenvironment and Identification of Potential Therapeutic Targets in Wilms Tumors

Translational oncology, 2014-08, Vol.7 (4), p.484-492

2014

Volltextzugriff (PDF)

Wilms’ Tumor Blastemal Stem Cells Dedifferentiate to Propagate the Tumor Bulk

Stem cell reports, 2014-07, Vol.3 (1), p.24-33

2014

Volltextzugriff (PDF)

Characterization of the Inflammatory Microenvironment and Identification of Potential Therapeutic Targets in Wilms Tumors12

Translational oncology, 2014-06, Vol.7 (4), p.484-492

2014

Volltextzugriff (PDF)

Wt1 functions in ovarian follicle development by regulating granulosa cell differentiation

Human molecular genetics, 2014-01, Vol.23 (2), p.333-341

2014

Volltextzugriff (PDF)

Menü

Weitere Dienste

Einstellungen

Genetic and epigenetic features of bilateral Wilms tumor predisposition in patients from the Children's Oncology Group AREN18B5-Q

Genetic changes associated with relapse in favorable histology Wilms tumor: A Children’s Oncology Group AREN03B2 study

Wilm's tumor 1 promotes memory flexibility

A unique subset of low-risk Wilms tumors is characterized by loss of function of TRIM28 (KAP1), a gene critical in early renal development: A Children's Oncology Group study

A unique subset of low-risk Wilms tumors is characterized by loss of function of TRIM28

Knock-in of the Wt1 R394W mutation causes MDS and cooperates with Flt3/ITD to drive aggressive myeloid neoplasms in mice

Wt1 directs the lineage specification of sertoli and granulosa cells by repressing Sf1 expression

Significance of TP53 Mutation in Wilms Tumors with Diffuse Anaplasia: A Report from the Children's Oncology Group

Abstract LB-180: The genetic landscape of Wilms tumor

Identification of germline DICER1 mutations and loss of heterozygosity in familial Wilms tumour

Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with β-Catenin Activation or Wt1 Ablation and Igf2 Upregulation1

Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with [beta]-Catenin Activation or Wt1 Ablation and Igf2 Upregulation

Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with β-Catenin Activation or Wt1 Ablation and Igf2 Upregulation

MLLT1 YEATS domain mutations in clinically distinctive Favourable Histology Wilms tumours

A Wilms Tumor 1 (WT1) Mutation Causes Myelodysplastic Syndrome in a Knock-in Mouse Model, and a Mixed Myelodysplastic/Myeloproliferative Neoplam in Double Knock-in Mice with WT1 and FLT3/ITD Mutations

Clinical outcome and biological predictors of relapse following nephrectomy only for very low risk Wilms tumor (VLR WT): A report from Children’s Oncology Group AREN0532

Reprogramming of Sertoli cells to fetal-like Leydig cells by Wt1 ablation

Reprogramming of Sertoli cells to fetal-like Leydig cells byWt1ablation

Recurrent DGCR8, DROSHA, and SIX Homeodomain Mutations in Favorable Histology Wilms Tumors

Recurrent DGCR8, DROSHA, and SIX Homeodomain Mutations in Favorable Histology Wilms Tumors

β-Catenin Activation in a Novel Liver Progenitor Cell Type Is Sufficient to Cause Hepatocellular Carcinoma and Hepatoblastoma

Characterization of the Inflammatory Microenvironment and Identification of Potential Therapeutic Targets in Wilms Tumors

Wilms’ Tumor Blastemal Stem Cells Dedifferentiate to Propagate the Tumor Bulk

Characterization of the Inflammatory Microenvironment and Identification of Potential Therapeutic Targets in Wilms Tumors12

Wt1 functions in ovarian follicle development by regulating granulosa cell differentiation

Aktive Filter

Suchergebnisse filtern