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WT1 Mutation and 11P15 Loss of Heterozygosity Predict Relapse in Very Low-Risk Wilms Tumors Treated With Surgery Alone: A Children's Oncology Group Study

Journal of clinical oncology, 2011-02, Vol.29 (6), p.698-703

2011

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Loss of Heterozygosity at 2q37 in Sporadic Wilms' Tumor: Putative Role for miR-562

Clinical cancer research, 2009-10, Vol.15 (19), p.5985-5992

2009

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Treatment With Nephrectomy Only for Small, Stage I/Favorable Histology Wilms’ Tumor: A Report From the National Wilms’ Tumor Study Group

Journal of clinical oncology, 2001-09, Vol.19 (17), p.3719-3724

2001

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β-Catenin Activation in a Novel Liver Progenitor Cell Type Is Sufficient to Cause Hepatocellular Carcinoma and Hepatoblastoma

Cancer research (Chicago, Ill.), 2014-08, Vol.74 (16), p.4515-4525

2014

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Frequent association of β-catenin and WT1 mutations in Wilms tumors

Cancer research (Chicago, Ill.), 2000-11, Vol.60 (22), p.6288-6292

2000

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Long-term Outcomes for Infants With Very Low Risk Wilms Tumor Treated With Surgery Alone in National Wilms Tumor Study-5

Annals of surgery, 2010-03, Vol.251 (3), p.555-558

2010

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Upregulation of c-MYC in WT1-mutant tumors: assessment of WT1 putative transcriptional targets using cDNA microarray expression profiling of genetically defined Wilms' tumors

Oncogene, 2003-06, Vol.22 (24), p.3821-3826

2003

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Loss of heterozygosity for chromosomes 16q and 1p in Wilms' tumors predicts an adverse outcome

Cancer research (Chicago, Ill.), 1994-05, Vol.54 (9), p.2331-2333

1994

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Hereditary hyperparathyroidism-jaw tumor syndrome : the endocrine tumor gene HRPT2 maps to chromosome 1q21-q31

American journal of human genetics, 1995-04, Vol.56 (4), p.944-950

1995

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Sequential loss of heterozygosity at microsatellite motifs in preinvasive and invasive head and neck squamous carcinoma

Cancer research (Chicago, Ill.), 1995-06, Vol.55 (12), p.2656-2659

1995

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Linkage of familial Wilms' tumor predisposition to chromosome 19 and a two-locus model for the etiology of familial tumors

Cancer research (Chicago, Ill.), 1998-04, Vol.58 (7), p.1387-1390

1998

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Microsatellite instability in preinvasive and invasive head and neck squamous carcinoma

The American journal of pathology, 1996-06, Vol.148 (6), p.2067-2072

1996

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Loss of heterozygosity for chromosomes 1p and 16q is an adverse prognostic factor in favorable-histology wilms tumor : A report from the national wilms tumor study group: Molecular oncology: genomics

Journal of clinical oncology, 2005, Vol.23 (29), p.7312-7321

2005

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Allelic loss and replication errors at microsatellite loci on chromosome 11p in head and neck squamous carcinoma: association with aggressive biological features

Clinical cancer research, 1996-05, Vol.2 (5), p.903-907

1996

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Nonlinkage of 16q markers to familial predisposition to wilms tumor

Cancer research (Chicago, Ill.), 1992-11, Vol.52 (21), p.6117-6120

1992

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Evidence for genetic heterogeneity in familial wilms' tumor

Cancer research (Chicago, Ill.), 1997-05, Vol.57 (10), p.1859-1862

1997

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RNA Expression of the WT1 Gene in Wilms' Tumors in Relation to Histology

JNCI : Journal of the National Cancer Institute, 1992-02, Vol.84 (3), p.181-187

1992

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Smallest region of overlap in Wilms tumor deletions uniquely implicates an 11p13 zinc finger gene as the disease locus

Genomics (San Diego, Calif.), 1991-05, Vol.10 (1), p.293-297

1991

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Parental origin of De Novo constitutional deletions of chromosomal band 11p13

American journal of human genetics, 1990-07, Vol.47 (1), p.155-160

1990

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WT1 Mutation and 11P15 Loss of Heterozygosity Predict Relapse in Very Low-Risk Wilms Tumors Treated With Surgery Alone: A Children's Oncology Group Study

Loss of Heterozygosity at 2q37 in Sporadic Wilms' Tumor: Putative Role for miR-562

Treatment With Nephrectomy Only for Small, Stage I/Favorable Histology Wilms’ Tumor: A Report From the National Wilms’ Tumor Study Group

β-Catenin Activation in a Novel Liver Progenitor Cell Type Is Sufficient to Cause Hepatocellular Carcinoma and Hepatoblastoma

Frequent association of β-catenin and WT1 mutations in Wilms tumors

Long-term Outcomes for Infants With Very Low Risk Wilms Tumor Treated With Surgery Alone in National Wilms Tumor Study-5

Upregulation of c-MYC in WT1-mutant tumors: assessment of WT1 putative transcriptional targets using cDNA microarray expression profiling of genetically defined Wilms' tumors

Loss of heterozygosity for chromosomes 16q and 1p in Wilms' tumors predicts an adverse outcome

Hereditary hyperparathyroidism-jaw tumor syndrome : the endocrine tumor gene HRPT2 maps to chromosome 1q21-q31

Sequential loss of heterozygosity at microsatellite motifs in preinvasive and invasive head and neck squamous carcinoma

Linkage of familial Wilms' tumor predisposition to chromosome 19 and a two-locus model for the etiology of familial tumors

Microsatellite instability in preinvasive and invasive head and neck squamous carcinoma

Loss of heterozygosity for chromosomes 1p and 16q is an adverse prognostic factor in favorable-histology wilms tumor : A report from the national wilms tumor study group: Molecular oncology: genomics

Allelic loss and replication errors at microsatellite loci on chromosome 11p in head and neck squamous carcinoma: association with aggressive biological features

Nonlinkage of 16q markers to familial predisposition to wilms tumor

Evidence for genetic heterogeneity in familial wilms' tumor

RNA Expression of the WT1 Gene in Wilms' Tumors in Relation to Histology

Smallest region of overlap in Wilms tumor deletions uniquely implicates an 11p13 zinc finger gene as the disease locus

Parental origin of De Novo constitutional deletions of chromosomal band 11p13

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