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Dietary Antioxidants Significantly Attenuate Hyperoxia-Induced Acute Inflammatory Lung Injury by Enhancing Macrophage Function via Reducing the Accumulation of Airway HMGB1

International journal of molecular sciences, 2020-02, Vol.21 (3), p.977

2020

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The α7 nicotinic acetylcholine receptor agonist, GTS-21, attenuates hyperoxia-induced acute inflammatory lung injury by alleviating the accumulation of HMGB1 in the airways and the circulation

Molecular medicine (Cambridge, Mass.), 2020-06, Vol.26 (1), p.63-63, Article 63

2020

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A Standardized Botanical Composition Mitigated Acute Inflammatory Lung Injury and Reduced Mortality through Extracellular HMGB1 Reduction

Molecules (Basel, Switzerland), 2023-09, Vol.28 (18), p.6560

2023

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The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2020-12, Vol.26 (1), p.1-98

2020

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Carbon monoxide provides protection against hyperoxic lung injury

American journal of physiology. Lung cellular and molecular physiology, 1999-04, Vol.276 (4), p.688-L694

1999

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Correction to: GTS-21, an α7nAChR agonist, increases pulmonary bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2021-08, Vol.27 (1), p.93-93, Article 93

2021

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From nicotine to the cholinergic anti-inflammatory reflex - Can nicotine alleviate the dysregulated inflammation in COVID-19?

Journal of immunotoxicology, 2021-01, Vol.18 (1), p.23-29

2021

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HMGB-1 as a therapeutic target for infectious and inflammatory disorders

Shock (Augusta, Ga.), 2006, Vol.25 (1), p.4-11

2006

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Exogenous administration of heme oxygenase-1 by gene transfer provides protection against hyperoxia-induced lung injury

The Journal of clinical investigation, 1999-04, Vol.103 (7), p.1047-1054

1999

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2-O, 3-O desulfated heparin (ODSH) increases bacterial clearance and attenuates lung injury in cystic fibrosis by restoring HMGB1-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2021-07, Vol.27 (1), p.79-79, Article 79

2021

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Retraction Note: The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2020-12, Vol.26 (1), p.132-132, Article 132

2020

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Hyperoxia sensing: From molecular mechanisms to significance in disease

Journal of immunotoxicology, 2010-12, Vol.7 (4), p.239-254

2010

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Inhibition of extracellular HMGB1 attenuates hyperoxia-induced inflammatory acute lung injury

Redox biology, 2014-01, Vol.2 (C), p.314-322

2014

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Correction to: 2‑O, 3‑O desulfated heparin (ODSH) increases bacterial clearance and attenuates lung injury in cystic fibrosis by restoring HMGB1‑compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2021-09, Vol.27 (1), p.98-98, Article 98

2021

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Expression of Concern to: The α7 nicotine acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2020-02, Vol.26 (1), p.16-16, Article 16

2020

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The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2014-06, Vol.20 (1), p.238-247

2014

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Interleukin-6-Induced Protection in Hyperoxic Acute Lung Injury

American journal of respiratory cell and molecular biology, 2000-05, Vol.22 (5), p.535-542

2000

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The Positive Allosteric Modulation of alpha7-Nicotinic Cholinergic Receptors by GAT107 Increases Bacterial Lung Clearance in Hyperoxic Mice by Decreasing Oxidative Stress in Macrophages

Antioxidants, 2021-01, Vol.10 (1), p.135

2021

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Carbon Nanomaterials Stimulate HMGB1 Release From Macrophages and Induce Cell Migration and Invasion

Toxicological sciences, 2019-12, Vol.172 (2), p.398-410

2019

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Mitogen-activated protein kinase pathway mediates hyperoxia-induced apoptosis in cultured macrophage cells

American journal of physiology. Lung cellular and molecular physiology, 1999-09, Vol.277 (3), p.589-L595

1999

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Alpha-chemokine receptor blockade reduces high mobility group box 1 protein-induced lung inflammation and injury and improves survival in sepsis

American journal of physiology. Lung cellular and molecular physiology, 2005-10, Vol.289 (4), p.L583-L590

2005

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In memoriam Steven Yue Qian (1960–2019)

Redox biology, 2020-10, Vol.37, p.101821, Article 101821

2020

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Nitric Oxide Regulates Gene Expression in Cancers by Controlling Histone Posttranslational Modifications

Cancer research (Chicago, Ill.), 2015-12, Vol.75 (24), p.5299-5308

2015

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Hyperoxia Inhibits Oxidant-induced Apoptosis in Lung Epithelial Cells

The Journal of biological chemistry, 2001-01, Vol.276 (1), p.569-575

2001

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A32 MICROBIAL INTERACTIONS AND HOST DEFENSE: Host And Pathogen Factors Modulate Antibacterial Properties Of Histones And Non-Histone Protein Hmgb1

American journal of respiratory and critical care medicine, 2015-01, Vol.191, p.1

2015

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Dietary Antioxidants Significantly Attenuate Hyperoxia-Induced Acute Inflammatory Lung Injury by Enhancing Macrophage Function via Reducing the Accumulation of Airway HMGB1

The α7 nicotinic acetylcholine receptor agonist, GTS-21, attenuates hyperoxia-induced acute inflammatory lung injury by alleviating the accumulation of HMGB1 in the airways and the circulation

A Standardized Botanical Composition Mitigated Acute Inflammatory Lung Injury and Reduced Mortality through Extracellular HMGB1 Reduction

The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Carbon monoxide provides protection against hyperoxic lung injury

Correction to: GTS-21, an α7nAChR agonist, increases pulmonary bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

From nicotine to the cholinergic anti-inflammatory reflex - Can nicotine alleviate the dysregulated inflammation in COVID-19?

HMGB-1 as a therapeutic target for infectious and inflammatory disorders

Exogenous administration of heme oxygenase-1 by gene transfer provides protection against hyperoxia-induced lung injury

2-O, 3-O desulfated heparin (ODSH) increases bacterial clearance and attenuates lung injury in cystic fibrosis by restoring HMGB1-compromised macrophage function

Retraction Note: The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Hyperoxia sensing: From molecular mechanisms to significance in disease

Inhibition of extracellular HMGB1 attenuates hyperoxia-induced inflammatory acute lung injury

Correction to: 2‑O, 3‑O desulfated heparin (ODSH) increases bacterial clearance and attenuates lung injury in cystic fibrosis by restoring HMGB1‑compromised macrophage function

Expression of Concern to: The α7 nicotine acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Interleukin-6-Induced Protection in Hyperoxic Acute Lung Injury

The Positive Allosteric Modulation of alpha7-Nicotinic Cholinergic Receptors by GAT107 Increases Bacterial Lung Clearance in Hyperoxic Mice by Decreasing Oxidative Stress in Macrophages

Carbon Nanomaterials Stimulate HMGB1 Release From Macrophages and Induce Cell Migration and Invasion

Mitogen-activated protein kinase pathway mediates hyperoxia-induced apoptosis in cultured macrophage cells

Alpha-chemokine receptor blockade reduces high mobility group box 1 protein-induced lung inflammation and injury and improves survival in sepsis

In memoriam Steven Yue Qian (1960–2019)

Nitric Oxide Regulates Gene Expression in Cancers by Controlling Histone Posttranslational Modifications

Hyperoxia Inhibits Oxidant-induced Apoptosis in Lung Epithelial Cells

A32 MICROBIAL INTERACTIONS AND HOST DEFENSE: Host And Pathogen Factors Modulate Antibacterial Properties Of Histones And Non-Histone Protein Hmgb1

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