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Sex Differences in Nonalcoholic Fatty Liver Disease: State of the Art and Identification of Research Gaps

Hepatology (Baltimore, Md.), 2019-10, Vol.70 (4), p.1457-1469

2019

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c‐Jun N‐terminal kinase mediates mouse liver injury through a novel Sab (SH3BP5)‐dependent pathway leading to inactivation of intramitochondrial Src

Hepatology (Baltimore, Md.), 2016-06, Vol.63 (6), p.1987-2003

2016

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The Regulation of JNK Signaling Pathways in Cell Death through the Interplay with Mitochondrial SAB and Upstream Post-Translational Effects

International journal of molecular sciences, 2018-11, Vol.19 (11), p.3657

2018

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c-Jun N-terminal Kinase (JNK)-dependent Acute Liver Injury from Acetaminophen or Tumor Necrosis Factor (TNF) Requires Mitochondrial Sab Protein Expression in Mice

The Journal of biological chemistry, 2011-10, Vol.286 (40), p.35071-35078

2011

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Mitochondrial P-JNK target, SAB (SH3BP5), in regulation of cell death

Frontiers in cell and developmental biology, 2024, Vol.12, p.1359152-1359152

2024

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Hepatic FcRn regulates albumin homeostasis and susceptibility to liver injury

Proceedings of the National Academy of Sciences - PNAS, 2017-04, Vol.114 (14), p.E2862-E2871

2017

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The role of MAP2 kinases and p38 kinase in acute murine liver injury models

Cell death & disease, 2017-06, Vol.8 (6), p.e2903-e2903

2017

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Role of cAMP-responsive Element-binding Protein (CREB)-regulated Transcription Coactivator 3 (CRTC3) in the Initiation of Mitochondrial Biogenesis and Stress Response in Liver Cells

The Journal of biological chemistry, 2011-06, Vol.286 (25), p.22047-22054

2011

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Mechanism and Therapeutic Targets of c-Jun-N-Terminal Kinases Activation in Nonalcoholic Fatty Liver Disease

Biomedicines, 2022-08, Vol.10 (8), p.2035

2022

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Receptor interacting protein kinase 1 mediates murine acetaminophen toxicity independent of the necrosome and not through necroptosis

Hepatology (Baltimore, Md.), 2015-12, Vol.62 (6), p.1847-1857

2015

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New insights into the role and mechanism of c‐Jun‐N‐terminal kinase signaling in the pathobiology of liver diseases

Hepatology (Baltimore, Md.), 2018-05, Vol.67 (5), p.2013-2024

2018

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Acetaminophen Hepatotoxicity: Strong Offense and Weakened Defense

Hepatology (Baltimore, Md.), 2020-05, Vol.71 (5), p.1530-1532

2020

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Intestinal Epithelial Chemokine (C-C Motif) Ligand 7 Overexpression Enhances Acetaminophen-Induced Hepatotoxicity in Mice

The American journal of pathology, 2020-01, Vol.190 (1), p.57-67

2020

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Hepatic Mitochondrial SAB Deletion or Knockdown Alleviates Diet‐Induced Metabolic Syndrome, Steatohepatitis, and Hepatic Fibrosis

Hepatology (Baltimore, Md.), 2021-12, Vol.74 (6), p.3127-3145

2021

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Expression of mitochondrial membrane-linked SAB determines severity of sex-dependent acute liver injury

The Journal of clinical investigation, 2019-12, Vol.129 (12), p.5278-5293

2019

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Oral magnesium prevents acetaminophen-induced acute liver injury by modulating microbial metabolism

Cell host & microbe, 2024-01, Vol.32 (1), p.48-62.e9

2024

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Silencing Glycogen Synthase Kinase-3β Inhibits Acetaminophen Hepatotoxicity and Attenuates JNK Activation and Loss of Glutamate Cysteine Ligase and Myeloid Cell Leukemia Sequence 1

The Journal of biological chemistry, 2010-03, Vol.285 (11), p.8244-8255

2010

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Sex Differences in NAFLD: State of the Art and Identification of Research Gaps

Hepatology (Baltimore, Md.), 2019-09, Vol.70 (4), p.1457-1469

2019

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JNK mediates mouse liver injury through a novel Sab (SH3BP5) dependent pathway leading to inactivation of intramitochondrial Src

Hepatology (Baltimore, Md.), 2016-03, Vol.63 (6), p.1987-2003

2016

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Immune Responses against Allogeneic and Syngeneic Tumors in Aged C57BL/6 Mice

Microbiology and immunology, 2002-01, Vol.46 (7), p.513-519

2002

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Silencing Glycogen Synthase Kinase-3Î² Inhibits Acetaminophen Hepatotoxicity and Attenuates JNK Activation and Loss of Glutamate Cysteine Ligase and Myeloid Cell Leukemia Sequence 1

The Journal of biological chemistry, 2010-03, Vol.285 (11), p.8244

2010

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Silencing Glycogen Synthase Kinase-3b Inhibits Acetaminophen Hepatotoxicity and Attenuates JNK Activation and Loss of Glutamate Cysteine Ligase and Myeloid Cell Leukemia Sequence 1

The Journal of biological chemistry, 2010-03, Vol.285 (11), p.8244-8255

2010

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Silencing glycogen synthase kinase-3beta inhibits acetaminophen hepatotoxicity and attenuates JNK activation and loss of glutamate cysteine ligase and myeloid cell leukemia sequence 1

The Journal of biological chemistry, 2010-03, Vol.285 (11), p.8244

2010

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Cryptic CTL Epitope on a Murine Sarcoma Meth A Generated by Exon Extension as a Novel Mechanism

The Journal of immunology (1950), 2003-05, Vol.170 (9), p.4862-4868

2003

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Post-Heated and Fermented Edible Teas and Their Dried Forms: Used for Drinking in Myanmar

Journal of Home Economics of Japan, 1999/06/15, Vol.50(6), pp.639-646

1999

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Sex Differences in Nonalcoholic Fatty Liver Disease: State of the Art and Identification of Research Gaps

c‐Jun N‐terminal kinase mediates mouse liver injury through a novel Sab (SH3BP5)‐dependent pathway leading to inactivation of intramitochondrial Src

The Regulation of JNK Signaling Pathways in Cell Death through the Interplay with Mitochondrial SAB and Upstream Post-Translational Effects

c-Jun N-terminal Kinase (JNK)-dependent Acute Liver Injury from Acetaminophen or Tumor Necrosis Factor (TNF) Requires Mitochondrial Sab Protein Expression in Mice

Mitochondrial P-JNK target, SAB (SH3BP5), in regulation of cell death

Hepatic FcRn regulates albumin homeostasis and susceptibility to liver injury

The role of MAP2 kinases and p38 kinase in acute murine liver injury models

Role of cAMP-responsive Element-binding Protein (CREB)-regulated Transcription Coactivator 3 (CRTC3) in the Initiation of Mitochondrial Biogenesis and Stress Response in Liver Cells

Mechanism and Therapeutic Targets of c-Jun-N-Terminal Kinases Activation in Nonalcoholic Fatty Liver Disease

Receptor interacting protein kinase 1 mediates murine acetaminophen toxicity independent of the necrosome and not through necroptosis

New insights into the role and mechanism of c‐Jun‐N‐terminal kinase signaling in the pathobiology of liver diseases

Acetaminophen Hepatotoxicity: Strong Offense and Weakened Defense

Intestinal Epithelial Chemokine (C-C Motif) Ligand 7 Overexpression Enhances Acetaminophen-Induced Hepatotoxicity in Mice

Hepatic Mitochondrial SAB Deletion or Knockdown Alleviates Diet‐Induced Metabolic Syndrome, Steatohepatitis, and Hepatic Fibrosis

Expression of mitochondrial membrane-linked SAB determines severity of sex-dependent acute liver injury

Oral magnesium prevents acetaminophen-induced acute liver injury by modulating microbial metabolism

Silencing Glycogen Synthase Kinase-3β Inhibits Acetaminophen Hepatotoxicity and Attenuates JNK Activation and Loss of Glutamate Cysteine Ligase and Myeloid Cell Leukemia Sequence 1

Sex Differences in NAFLD: State of the Art and Identification of Research Gaps

JNK mediates mouse liver injury through a novel Sab (SH3BP5) dependent pathway leading to inactivation of intramitochondrial Src

Immune Responses against Allogeneic and Syngeneic Tumors in Aged C57BL/6 Mice

Silencing Glycogen Synthase Kinase-3Î² Inhibits Acetaminophen Hepatotoxicity and Attenuates JNK Activation and Loss of Glutamate Cysteine Ligase and Myeloid Cell Leukemia Sequence 1

Silencing Glycogen Synthase Kinase-3b Inhibits Acetaminophen Hepatotoxicity and Attenuates JNK Activation and Loss of Glutamate Cysteine Ligase and Myeloid Cell Leukemia Sequence 1

Silencing glycogen synthase kinase-3beta inhibits acetaminophen hepatotoxicity and attenuates JNK activation and loss of glutamate cysteine ligase and myeloid cell leukemia sequence 1

Cryptic CTL Epitope on a Murine Sarcoma Meth A Generated by Exon Extension as a Novel Mechanism

Post-Heated and Fermented Edible Teas and Their Dried Forms: Used for Drinking in Myanmar

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