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Strategic role of frontal white matter tracts in vascular cognitive impairment: a voxel-based lesion-symptom mapping study in CADASIL

Brain (London, England : 1878), 2011-08, Vol.134 (8), p.2366-2375

2011

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Cortical Superficial Siderosis in Different Types of Cerebral Small Vessel Disease

Stroke (1970), 2017-05, Vol.48 (5), p.1404-1407

2017

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Segmentation of incident lacunes during the course of ischemic cerebral small vessel diseases

Frontiers in neurology, 2023-03, Vol.14, p.1113644-1113644

2023

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Contrast-based fully automatic segmentation of white matter hyperintensities: method and validation

PloS one, 2012-11, Vol.7 (11), p.e48953-e48953

2012

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Editorial: Cerebral Small Vessel Diseases: From Vessel Alterations to Cortical Parenchymal Injury

Frontiers in neurology, 2020-02, Vol.11, p.92-92

2020

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Updates on Prevention of Hemorrhagic and Lacunar Strokes

대한뇌졸중학회지, 2018, 20(2), 13, pp.167-179

2018

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In vivo high-resolution 7 Tesla MRI shows early and diffuse cortical alterations in CADASIL

PloS one, 2014-08, Vol.9 (8), p.e106311-e106311

2014

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Cerebral Amyloid Angiopathy Related Inflammation With Prominent Meningeal Involvement. A Report of 2 Cases

Frontiers in neurology, 2019-09, Vol.10

2019

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ADC histograms from routine DWI for longitudinal studies in cerebral small vessel disease: a field study in CADASIL

PloS one, 2014-05, Vol.9 (5), p.e97173-e97173

2014

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Different Types of White Matter Hyperintensities in CADASIL

Frontiers in neurology, 2018-07, Vol.9, p.526-526

2018

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Retinal vascular density in CADASIL (Cerebral Autosomal Dominant Arteriopathy with Subcortical Infarcts and Leukoencephalopathy)

BMJ neurology open, 2023-05, Vol.5 (1), p.e000417-e000417

2023

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Alteration of the Cortex Shape as a Proxy of White Matter Swelling in Severe Cerebral Small Vessel Disease

Frontiers in neurology, 2019-07, Vol.10, p.753-753

2019

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Impact of regional cortical and subcortical changes on processing speed in cerebral small vessel disease

NeuroImage clinical, 2013-01, Vol.2, p.854-861

2013

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Cerebral Autosomal Dominant Arteriopathy With Subcortical Infarcts and Leukoencephalopathy: Lessons From Neuroimaging

Stroke (1970), 2020-01, Vol.51 (1), p.21-28

2020

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Three-Dimensional MRI Analysis of Individual Volume of Lacunes in CADASIL

Stroke (1970), 2009-01, Vol.40 (1), p.124-128

2009

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Different types of white matter hyperintensities in CADASIL: Insights from 7-Tesla MRI

Journal of cerebral blood flow and metabolism, 2018-09, Vol.38 (9), p.1654-1663

2018

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Brain atrophy in cerebral small vessel diseases: Extent, consequences, technical limitations and perspectives: The HARNESS initiative

Journal of Cerebral Blood Flow & Metabolism, 2020-02, Vol.40 (2), p.231-245

2020

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Incident lacunes preferentially localize to the edge of white matter hyperintensities: insights into the pathophysiology of cerebral small vessel disease

Brain (London, England : 1878), 2013-09, Vol.136 (Pt 9), p.2717-2726

2013

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Cerebral Autosomal Dominant Arteriopathy with Subcortical Infarcts and Leukoencephalopathy (CADASIL) as a model of small vessel disease: update on clinical, diagnostic, and management aspects

BMC medicine, 2017-02, Vol.15 (1), p.41-41, Article 41

2017

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The Epidermal Growth Factor Domain of the Mutation Does Not Appear to Influence Disease Progression in CADASIL When Brain Volume and Sex Are Taken into Account

American journal of neuroradiology : AJNR, 2022-05, Vol.43 (5), p.715-720

2022

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White Matter Hyperintensities of the Corpus Callosum Are Associated With Clinical Severity in CADASIL

Stroke (1970), 2023-04, Vol.54 (4), p.e138-e141

2023

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Phenotypic variability in 446 CADASIL patients: Impact of NOTCH3 gene mutation location in addition to the effects of age, sex and vascular risk factors

Journal of cerebral blood flow and metabolism, 2023-01, Vol.43 (1), p.153-166

2023

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Cerebral Microbleeds and the Risk of Incident Ischemic Stroke in CADASIL (Cerebral Autosomal Dominant Arteriopathy With Subcortical Infarcts and Leukoencephalopathy)

Stroke (1970), 2017-10, Vol.48 (10), p.2699-2703

2017

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The effect of NOTCH3 pathogenic variant position on CADASIL disease severity: NOTCH3 EGFr 1-6 pathogenic variant are associated with a more severe phenotype and lower survival compared with EGFr 7-34 pathogenic variant

Genetics in medicine, 2019-03, Vol.21 (3), p.676-682

2019

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Predictors of Clinical Worsening in Cerebral Autosomal Dominant Arteriopathy With Subcortical Infarcts and Leukoencephalopathy: Prospective Cohort Study

Stroke (1970), 2016-01, Vol.47 (1), p.4-11

2016

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Strategic role of frontal white matter tracts in vascular cognitive impairment: a voxel-based lesion-symptom mapping study in CADASIL

Cortical Superficial Siderosis in Different Types of Cerebral Small Vessel Disease

Segmentation of incident lacunes during the course of ischemic cerebral small vessel diseases

Contrast-based fully automatic segmentation of white matter hyperintensities: method and validation

Editorial: Cerebral Small Vessel Diseases: From Vessel Alterations to Cortical Parenchymal Injury

Updates on Prevention of Hemorrhagic and Lacunar Strokes

In vivo high-resolution 7 Tesla MRI shows early and diffuse cortical alterations in CADASIL

Cerebral Amyloid Angiopathy Related Inflammation With Prominent Meningeal Involvement. A Report of 2 Cases

ADC histograms from routine DWI for longitudinal studies in cerebral small vessel disease: a field study in CADASIL

Different Types of White Matter Hyperintensities in CADASIL

Retinal vascular density in CADASIL (Cerebral Autosomal Dominant Arteriopathy with Subcortical Infarcts and Leukoencephalopathy)

Alteration of the Cortex Shape as a Proxy of White Matter Swelling in Severe Cerebral Small Vessel Disease

Impact of regional cortical and subcortical changes on processing speed in cerebral small vessel disease

Cerebral Autosomal Dominant Arteriopathy With Subcortical Infarcts and Leukoencephalopathy: Lessons From Neuroimaging

Three-Dimensional MRI Analysis of Individual Volume of Lacunes in CADASIL

Different types of white matter hyperintensities in CADASIL: Insights from 7-Tesla MRI

Brain atrophy in cerebral small vessel diseases: Extent, consequences, technical limitations and perspectives: The HARNESS initiative

Incident lacunes preferentially localize to the edge of white matter hyperintensities: insights into the pathophysiology of cerebral small vessel disease

Cerebral Autosomal Dominant Arteriopathy with Subcortical Infarcts and Leukoencephalopathy (CADASIL) as a model of small vessel disease: update on clinical, diagnostic, and management aspects

The Epidermal Growth Factor Domain of the Mutation Does Not Appear to Influence Disease Progression in CADASIL When Brain Volume and Sex Are Taken into Account

White Matter Hyperintensities of the Corpus Callosum Are Associated With Clinical Severity in CADASIL

Phenotypic variability in 446 CADASIL patients: Impact of NOTCH3 gene mutation location in addition to the effects of age, sex and vascular risk factors

Cerebral Microbleeds and the Risk of Incident Ischemic Stroke in CADASIL (Cerebral Autosomal Dominant Arteriopathy With Subcortical Infarcts and Leukoencephalopathy)

The effect of NOTCH3 pathogenic variant position on CADASIL disease severity: NOTCH3 EGFr 1-6 pathogenic variant are associated with a more severe phenotype and lower survival compared with EGFr 7-34 pathogenic variant

Predictors of Clinical Worsening in Cerebral Autosomal Dominant Arteriopathy With Subcortical Infarcts and Leukoencephalopathy: Prospective Cohort Study

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