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Selective induction of cancer cell death by VDAC1‐based peptides and their potential use in cancer therapy

Molecular oncology, 2018-06, Vol.12 (7), p.1077-1103

2018

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VDAC1 in the diseased myocardium and the effect of VDAC1-interacting compound on atrial fibrosis induced by hyperaldosteronism

Scientific reports, 2020-12, Vol.10 (1), p.22101-22101, Article 22101

2020

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SMAC/Diablo controls proliferation of cancer cells by regulating phosphatidylethanolamine synthesis

Molecular oncology, 2021-11, Vol.15 (11), p.3037-3061

2021

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VDAC1 functions in Ca2+ homeostasis and cell life and death in health and disease

Cell calcium (Edinburgh), 2018-01, Vol.69, p.81-100

2018

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VDAC oligomers form mitochondrial pores to release mtDNA fragments and promote lupus-like disease

Science (American Association for the Advancement of Science), 2019-12, Vol.366 (6472), p.1531-1536

2019

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Voltage-Dependent Anion Channel 1 As an Emerging Drug Target for Novel Anti-Cancer Therapeutics

Frontiers in oncology, 2017-07, Vol.7, p.154-154

2017

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NCLX is an essential component of mitochondrial Na⁺/Ca²⁺ exchange

Proceedings of the National Academy of Sciences - PNAS, 2010-01, Vol.107 (1), p.436-441

2010

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Adverse Effects of Metformin From Diabetes to COVID-19, Cancer, Neurodegenerative Diseases, and Aging: Is VDAC1 a Common Target?

Frontiers in physiology, 2021-10, Vol.12, p.730048

2021

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Metabolic Reprograming Via Silencing of Mitochondrial VDAC1 Expression Encourages Differentiation of Cancer Cells

Molecular therapy. Nucleic acids, 2019-09, Vol.17, p.24-37

2019

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VDAC1 at the crossroads of cell metabolism, apoptosis and cell stress

Cell Stress, 2017-10, Vol.1 (1), p.11-36

2017

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Mitochondrial VDAC, the Na+/Ca2+ Exchanger, and the Ca2+ Uniporter in Ca2+ Dynamics and Signaling

Membrane Dynamics and Calcium Signaling, p.323-347

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The Expression Level of the Voltage-Dependent Anion Channel Controls Life and Death of the Cell

Proceedings of the National Academy of Sciences - PNAS, 2006-04, Vol.103 (15), p.5787-5792

2006

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Key regions of VDAC1 functioning in apoptosis induction and regulation by hexokinase

Biochimica et biophysica acta, 2009-05, Vol.1787 (5), p.421-430

2009

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Mitochondrial VDAC1 Silencing Leads to Metabolic Rewiring and the Reprogramming of Tumour Cells into Advanced Differentiated States

Cancers, 2018-12, Vol.10 (12), p.499

2018

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Silencing VDAC1 Expression by siRNA Inhibits Cancer Cell Proliferation and Tumor Growth In Vivo

Molecular therapy. Nucleic acids, 2014, Vol.3 (4), p.e159-e159, Article e159

2014

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Targeting the Mitochondrial Protein VDAC1 as a Potential Therapeutic Strategy in ALS

International journal of molecular sciences, 2022-09, Vol.23 (17), p.9946

2022

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Non-apoptotic activity of the mitochondrial protein SMAC/Diablo in lung cancer: Novel target to disrupt survival, inflammation, and immunosuppression

Frontiers in oncology, 2022-09, Vol.12, p.992260-992260

2022

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Overexpression of the mitochondrial anti-viral signaling protein, MAVS, in cancers is associated with cell survival and inflammation

Molecular therapy. Nucleic acids, 2023-09, Vol.33, p.713-732

2023

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The Mitochondrial Protein VDAC1 at the Crossroads of Cancer Cell Metabolism: The Epigenetic Link

Cancers, 2020-04, Vol.12 (4), p.1031

2020

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VDAC1 Silencing in Cancer Cells Leads to Metabolic Reprogramming That Modulates Tumor Microenvironment

Cancers, 2021-06, Vol.13 (11), p.2850

2021

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Targeting the overexpressed mitochondrial protein VDAC1 in a mouse model of Alzheimer's disease protects against mitochondrial dysfunction and mitigates brain pathology

Translational neurodegeneration, 2022-12, Vol.11 (1), p.58-30, Article 58

2022

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Empty mesoporous silica particles significantly delay disease progression and extend survival in a mouse model of ALS

Scientific reports, 2020-11, Vol.10 (1), p.20675-20675, Article 20675

2020

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Silencing VDAC1 to Treat Mesothelioma Cancer: Tumor Reprograming and Altering Tumor Hallmarks

Biomolecules (Basel, Switzerland), 2022-06, Vol.12 (7), p.895

2022

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Editorial: Altered Expression of Proteins in Cancer: Function and Potential Therapeutic Targets

Frontiers in oncology, 2022-06, Vol.12, p.949139-949139

2022

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Calcium binding and translocation by the voltage-dependent anion channel: a possible regulatory mechanism in mitochondrial function

Biochemical journal, 2001-08, Vol.358 (Pt 1), p.147-155

2001

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Selective induction of cancer cell death by VDAC1‐based peptides and their potential use in cancer therapy

VDAC1 in the diseased myocardium and the effect of VDAC1-interacting compound on atrial fibrosis induced by hyperaldosteronism

SMAC/Diablo controls proliferation of cancer cells by regulating phosphatidylethanolamine synthesis

VDAC1 functions in Ca2+ homeostasis and cell life and death in health and disease

VDAC oligomers form mitochondrial pores to release mtDNA fragments and promote lupus-like disease

Voltage-Dependent Anion Channel 1 As an Emerging Drug Target for Novel Anti-Cancer Therapeutics

NCLX is an essential component of mitochondrial Na⁺/Ca²⁺ exchange

Adverse Effects of Metformin From Diabetes to COVID-19, Cancer, Neurodegenerative Diseases, and Aging: Is VDAC1 a Common Target?

Metabolic Reprograming Via Silencing of Mitochondrial VDAC1 Expression Encourages Differentiation of Cancer Cells

VDAC1 at the crossroads of cell metabolism, apoptosis and cell stress

Mitochondrial VDAC, the Na+/Ca2+ Exchanger, and the Ca2+ Uniporter in Ca2+ Dynamics and Signaling

The Expression Level of the Voltage-Dependent Anion Channel Controls Life and Death of the Cell

Key regions of VDAC1 functioning in apoptosis induction and regulation by hexokinase

Mitochondrial VDAC1 Silencing Leads to Metabolic Rewiring and the Reprogramming of Tumour Cells into Advanced Differentiated States

Silencing VDAC1 Expression by siRNA Inhibits Cancer Cell Proliferation and Tumor Growth In Vivo

Targeting the Mitochondrial Protein VDAC1 as a Potential Therapeutic Strategy in ALS

Non-apoptotic activity of the mitochondrial protein SMAC/Diablo in lung cancer: Novel target to disrupt survival, inflammation, and immunosuppression

Overexpression of the mitochondrial anti-viral signaling protein, MAVS, in cancers is associated with cell survival and inflammation

The Mitochondrial Protein VDAC1 at the Crossroads of Cancer Cell Metabolism: The Epigenetic Link

VDAC1 Silencing in Cancer Cells Leads to Metabolic Reprogramming That Modulates Tumor Microenvironment

Targeting the overexpressed mitochondrial protein VDAC1 in a mouse model of Alzheimer's disease protects against mitochondrial dysfunction and mitigates brain pathology

Empty mesoporous silica particles significantly delay disease progression and extend survival in a mouse model of ALS

Silencing VDAC1 to Treat Mesothelioma Cancer: Tumor Reprograming and Altering Tumor Hallmarks

Editorial: Altered Expression of Proteins in Cancer: Function and Potential Therapeutic Targets

Calcium binding and translocation by the voltage-dependent anion channel: a possible regulatory mechanism in mitochondrial function

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