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Lack of skin sensitization hazard potential for alpha-glycosyl isoquercitrin (AGIQ) utilizing the Local Lymph Node Assay

Toxicology reports, 2022-01, Vol.9, p.1291-1296

2022

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Continuous exposure to α-glycosyl isoquercitrin from developmental stages to adulthood is necessary for facilitating fear extinction learning in rats

Journal of Toxicologic Pathology, 2020, Vol.33(4), pp.247-263

2020

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Effects of fixation on RNA extraction and amplification from laser capture microdissected tissue

Molecular carcinogenesis, 1999-06, Vol.25 (2), p.86-91

1999

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Lack of combined effect of continuous exposure to α-glycosyl isoquercitrin from fetal stages to adulthood and voluntary exercise or environmental enrichment on learning and behaviors in rats

Fundamental toxicological sciences, 2020, Vol.7 (5), p.241-248

2020

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Mutations in the ras proto-oncogene: clues to etiology and molecular pathogenesis of mouse liver tumors

Toxicology, 1995-08, Vol.101 (3), p.125-156

1995

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Tracheal Morphologic and Protein Alterations FollowingShort-Term Cigarette Mainstream Smoke Exposure to Rats

Journal of Toxicologic Pathology, 2012, Vol.25(3), pp.201-207

2012

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Applications of Magnetic Resonance Microscopy

Toxicologic pathology, 2004-07, Vol.32 (2_suppl), p.42-48

2004

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Continuous exposure to α-glycosyl isoquercitrin from developmental stages to adulthood is necessary for facilitating fear extinction learning in rats

Journal of Toxicologic Pathology, 2020, Vol.33 (4), p.247-263

2020

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Lack of preventive effect of maternal exposure to α-glycosyl isoquercitrin and α-lipoic acid on developmental hypothyroidism-induced aberrations of hippocampal neurogenesis in rat offspring

Journal of Toxicologic Pathology, 2019, Vol.32 (3), p.165-180

2019

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Relevance of Animal Carcinogenesis Findings to Human Cancer Predictions and Prevention

Toxicologic pathology, 2004-03, Vol.32 (1_suppl), p.40-48

2004

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Best Practices Guideline: Toxicologic Histopathology

Toxicologic pathology, 2004-01, Vol.32 (1), p.126-131

2004

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Spontaneous Lesions in Control B6C3F1 Mice and Recommended Sectioning of Male Accessory Sex Organs

Toxicologic pathology, 2002-02, Vol.30 (2), p.228-234

2002

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High frequency and heterogeneous distribution of p53 mutations in aflatoxin B1-induced mouse lung tumors

Cancer research (Chicago, Ill.), 1999-08, Vol.59 (15), p.3634-3640

1999

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Ten-day and four-week toxicity and toxicokinetics studies of alpha-glycosyl isoquercitrin in juvenile Göttingen minipigs

Toxicology research and application, 2019-01, Vol.3

2019

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Impact of Helicobacter hepaticus Infection in B6C3F1 Mice from Twelve National Toxicology Program Two-Year Carcinogenesis Studies

Toxicologic pathology, 1998-09, Vol.26 (5), p.602-611

1998

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Follicular Epithelial Cell Hypertrophy Induced by Chronic Oral Administration of 2,3,7,8-Tetrachlorodibenzo-p-Dioxin in Female Harlan Sprague—Dawley Rats

Toxicologic pathology, 2004-01, Vol.32 (1), p.41-49

2004

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Comparison of Proliferating Cell Nuclear Antigen to Tritiated Thymidine As a Marker of Proliferating Hepatocytes in Rats

Environmental health perspectives, 1993-12, Vol.101 (suppl 5), p.199-205

1993

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The Potential of Genetically Altered Mice as Animal Models for Carcinogen Identification

Toxicologic pathology, 1998-07, Vol.26 (4), p.579-581

Maronpot, R. R.

1998

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Activation of K-ras in aflatoxin B1-induced lung tumors from AC3F1 (A/J×C3H/HeJ) mice

Carcinogenesis (New York), 1996-08, Vol.17 (8), p.1735-1740

1996

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Failure of catalase to protect against aflatoxin B1 -induced mouse lung tumorigenicity

Toxicology and applied pharmacology, 2008-03, Vol.227 (2), p.179-183

2008

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Registered (1)H and (3)He magnetic resonance microscopy of the lung

Magnetic resonance in medicine, 2001-03, Vol.45 (3), p.365-370

2001

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Role of Proto-Oncogene Activation in Carcinogenesis

Environmental health perspectives, 1992-11, Vol.98, p.13-24

1992

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Ras proto-oncogene activation in liver and lung tumors from B6C3F1 mice exposed chronically to methylene chloride

Carcinogenesis (New York), 1993-05, Vol.14 (5), p.795-801

1993

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Hepatocarcinogenicity of chlordane in B6C3F1 and B6D2F1 male mice: evidence for regression in B6C3F1 mice and carcinogenesis independent of ras proto-oncogene activation

Carcinogenesis (New York), 1995-11, Vol.16 (11), p.2617-2625

1995

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Concepts, Labeling Procedures, and Design of Cell Proliferation Studies Relating to Carcinogenesis

Environmental health perspectives, 1993-12, Vol.101 (Suppl 5), p.59-65

1993

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Lack of skin sensitization hazard potential for alpha-glycosyl isoquercitrin (AGIQ) utilizing the Local Lymph Node Assay

Continuous exposure to α-glycosyl isoquercitrin from developmental stages to adulthood is necessary for facilitating fear extinction learning in rats

Effects of fixation on RNA extraction and amplification from laser capture microdissected tissue

Lack of combined effect of continuous exposure to α-glycosyl isoquercitrin from fetal stages to adulthood and voluntary exercise or environmental enrichment on learning and behaviors in rats

Mutations in the ras proto-oncogene: clues to etiology and molecular pathogenesis of mouse liver tumors

Tracheal Morphologic and Protein Alterations FollowingShort-Term Cigarette Mainstream Smoke Exposure to Rats

Applications of Magnetic Resonance Microscopy

Continuous exposure to α-glycosyl isoquercitrin from developmental stages to adulthood is necessary for facilitating fear extinction learning in rats

Lack of preventive effect of maternal exposure to α-glycosyl isoquercitrin and α-lipoic acid on developmental hypothyroidism-induced aberrations of hippocampal neurogenesis in rat offspring

Relevance of Animal Carcinogenesis Findings to Human Cancer Predictions and Prevention

Best Practices Guideline: Toxicologic Histopathology

Spontaneous Lesions in Control B6C3F1 Mice and Recommended Sectioning of Male Accessory Sex Organs

High frequency and heterogeneous distribution of p53 mutations in aflatoxin B1-induced mouse lung tumors

Ten-day and four-week toxicity and toxicokinetics studies of alpha-glycosyl isoquercitrin in juvenile Göttingen minipigs

Impact of Helicobacter hepaticus Infection in B6C3F1 Mice from Twelve National Toxicology Program Two-Year Carcinogenesis Studies

Follicular Epithelial Cell Hypertrophy Induced by Chronic Oral Administration of 2,3,7,8-Tetrachlorodibenzo-p-Dioxin in Female Harlan Sprague—Dawley Rats

Comparison of Proliferating Cell Nuclear Antigen to Tritiated Thymidine As a Marker of Proliferating Hepatocytes in Rats

The Potential of Genetically Altered Mice as Animal Models for Carcinogen Identification

Activation of K-ras in aflatoxin B1-induced lung tumors from AC3F1 (A/J×C3H/HeJ) mice

Failure of catalase to protect against aflatoxin B1 -induced mouse lung tumorigenicity

Registered (1)H and (3)He magnetic resonance microscopy of the lung

Role of Proto-Oncogene Activation in Carcinogenesis

Ras proto-oncogene activation in liver and lung tumors from B6C3F1 mice exposed chronically to methylene chloride

Hepatocarcinogenicity of chlordane in B6C3F1 and B6D2F1 male mice: evidence for regression in B6C3F1 mice and carcinogenesis independent of ras proto-oncogene activation

Concepts, Labeling Procedures, and Design of Cell Proliferation Studies Relating to Carcinogenesis

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