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The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2020-12, Vol.26 (1), p.1-98

2020

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Correction to: GTS-21, an α7nAChR agonist, increases pulmonary bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2021-08, Vol.27 (1), p.93-93, Article 93

2021

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Retraction Note: The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2020-12, Vol.26 (1), p.132-132, Article 132

2020

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Correction to: 2‑O, 3‑O desulfated heparin (ODSH) increases bacterial clearance and attenuates lung injury in cystic fibrosis by restoring HMGB1‑compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2021-09, Vol.27 (1), p.98-98, Article 98

2021

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Expression of Concern to: The α7 nicotine acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2020-02, Vol.26 (1), p.16-16, Article 16

2020

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Our Fragile Water Planet: An Introduction to the Earth Sciences

Mantell, C

1, 1976

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A32 MICROBIAL INTERACTIONS AND HOST DEFENSE: Host And Pathogen Factors Modulate Antibacterial Properties Of Histones And Non-Histone Protein Hmgb1

American journal of respiratory and critical care medicine, 2015-01, Vol.191, p.1

2015

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GAT107-mediated α7 nicotinic acetylcholine receptor signaling attenuates inflammatory lung injury and mortality in a mouse model of ventilator-associated pneumonia by alleviating macrophage mitochondrial oxidative stress via reducing MnSOD-S-glutathionylation

Redox biology, 2023-04, Vol.60, p.102614-102614, Article 102614

2023

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C73 OXIDANTS: Hyperoxia-Compromised Phagocytic Function In Macrophages Is Improved By Salicylates Via Inhibition Of Hmgb1 Release

American journal of respiratory and critical care medicine, 2016-01, Vol.193, p.1

2016

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Hyperoxia-induced Cell Death in the Lung-the Correlation of Apoptosis, Necrosis, and Inflammation

Annals of the New York Academy of Sciences, 1999-12, Vol.887 (1), p.171-180

1999

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A37 CYSTIC FIBROSIS: IMMUNE REGULATORY PATHWAYS: Cf Airway Epithelia Contribute To The Compromised Innate Immunity Against Bacterial Infection

American journal of respiratory and critical care medicine, 2016-01, Vol.193, p.1

2016

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A57 LUNG INJURY, REPAIR, AND FIBROSIS: THE PLOT THICKENS FOR THREE'S COMPANY: Mnsod Prevents Hyperoxia-Induced Cell Death Via Erk Activation

American journal of respiratory and critical care medicine, 2015-01, Vol.191, p.1

2015

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C59 IT HAPPENED BY OXIDANT! MOLECULAR OXYGEN AND OXIDATION: The [alpha]7 Nicotinic Acetylcholine Receptor Agonist Gts-21 Improves Bacterial Clearance In Mice By Restoring Hyperoxia-Compromised Macrophage Function

American journal of respiratory and critical care medicine, 2014-01, Vol.189, p.1

2014

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B34 LUNG MACROPHAGE ACTIVATION IN HEALTH AND DISEASE: Ethacrynic Acid Enhances Oxygen Toxicity-Compromised Macrophage Functions By Inhibiting Hmgb1 Release Via Blunting Nf-[kappa]B Activation

American journal of respiratory and critical care medicine, 2014-01, Vol.189, p.1

2014

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B34 LUNG MACROPHAGE ACTIVATION IN HEALTH AND DISEASE: Statins Improves Hyperoxia-Compromised Macrophage Function

American journal of respiratory and critical care medicine, 2014-01, Vol.189, p.1

2014

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D40 CRITICAL CARE: DIAGNOSING AND TREATING BACTERIAL AND VIRAL INFECTIONS IN THE ICU: Ascorbic Acid Attenuates Hyperoxia-Compromised Host Defense Against Pulmonary Bacterial Infection

American journal of respiratory and critical care medicine, 2016-01, Vol.193, p.1

2016

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B31 PNEUMONIA, ACUTE RESPIRATORY INFECTION: Partially Desulfated Heparin Enhances Oxygen Toxicity- Compromised Immunity Against Pseudomonas Lung Infection

American journal of respiratory and critical care medicine, 2014-01, Vol.189, p.1

2014

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C37 STRUCTURAL AND HEMATOPOIETIC CELL INTERACTIONS: The Hyperoxia-Compromised Macrophage Functions Are Attenuated By Ethacrynic Acid Via Inhibition Of Nf-[kappa]b-Mediated Release Of Hmgb1

American journal of respiratory and critical care medicine, 2015-01, Vol.191, p.1

2015

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Corrigendum to “Carbon Nanomaterials Stimulate HMGB1 Release From Macrophages and Induce Cell Migration and Invasion”

Toxicological sciences, 2020-05, Vol.175 (1), p.141-142

2020

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B32 REGULATION OF INFLAMMATION AND LUNG INJURY I: Inhibition Of Extracellular Hmgb1 Attenuates Hyperoxia-Induced Inflammatory Acute Lung Injury

American journal of respiratory and critical care medicine, 2015-01, Vol.191, p.1

2015

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GTS-21, an α7nAChR agonist, increases pulmonary bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Molecular medicine (Cambridge, Mass.), 2020-10, Vol.26 (1), Article 98

2020

Volltextzugriff (PDF)

C39 REGULATION OF INFLAMMATION AND LUNG INJURY II: Nf-[kappa]b In Airway Epithelium Protects Against Hyperoxia-Induced Inflammatory Lung Injury

American journal of respiratory and critical care medicine, 2015-01, Vol.191, p.1

2015

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Hyperoxia in Cell Culture: A Non-apoptotic Programmed Cell Death

Annals of the New York Academy of Sciences, 1999-12, Vol.887 (1), p.164-170

1999

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P-149 - 2-O, 3-O desulfated heparin (ODSH) improves bacterial clearance in cystic fibrosis by reducing HMGB1 release and improving phagocytosis of airway macrophages

Free radical biology & medicine, 2018-05, Vol.120, p.S89-S90

2018

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2-O, 3-O desulfated heparin (ODSH) improves bacterial clearance in cystic fibrosis by reducing HMGB1 release and improving phagocytosis of airway macrophages

Free radical biology & medicine, 2018-05, Vol.120, p.S89-S90

2018

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The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Correction to: GTS-21, an α7nAChR agonist, increases pulmonary bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Retraction Note: The α7 nicotinic acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Correction to: 2‑O, 3‑O desulfated heparin (ODSH) increases bacterial clearance and attenuates lung injury in cystic fibrosis by restoring HMGB1‑compromised macrophage function

Expression of Concern to: The α7 nicotine acetylcholine receptor agonist GTS-21 improves bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

Our Fragile Water Planet: An Introduction to the Earth Sciences

A32 MICROBIAL INTERACTIONS AND HOST DEFENSE: Host And Pathogen Factors Modulate Antibacterial Properties Of Histones And Non-Histone Protein Hmgb1

GAT107-mediated α7 nicotinic acetylcholine receptor signaling attenuates inflammatory lung injury and mortality in a mouse model of ventilator-associated pneumonia by alleviating macrophage mitochondrial oxidative stress via reducing MnSOD-S-glutathionylation

C73 OXIDANTS: Hyperoxia-Compromised Phagocytic Function In Macrophages Is Improved By Salicylates Via Inhibition Of Hmgb1 Release

Hyperoxia-induced Cell Death in the Lung-the Correlation of Apoptosis, Necrosis, and Inflammation

A37 CYSTIC FIBROSIS: IMMUNE REGULATORY PATHWAYS: Cf Airway Epithelia Contribute To The Compromised Innate Immunity Against Bacterial Infection

A57 LUNG INJURY, REPAIR, AND FIBROSIS: THE PLOT THICKENS FOR THREE'S COMPANY: Mnsod Prevents Hyperoxia-Induced Cell Death Via Erk Activation

C59 IT HAPPENED BY OXIDANT! MOLECULAR OXYGEN AND OXIDATION: The [alpha]7 Nicotinic Acetylcholine Receptor Agonist Gts-21 Improves Bacterial Clearance In Mice By Restoring Hyperoxia-Compromised Macrophage Function

B34 LUNG MACROPHAGE ACTIVATION IN HEALTH AND DISEASE: Ethacrynic Acid Enhances Oxygen Toxicity-Compromised Macrophage Functions By Inhibiting Hmgb1 Release Via Blunting Nf-[kappa]B Activation

B34 LUNG MACROPHAGE ACTIVATION IN HEALTH AND DISEASE: Statins Improves Hyperoxia-Compromised Macrophage Function

D40 CRITICAL CARE: DIAGNOSING AND TREATING BACTERIAL AND VIRAL INFECTIONS IN THE ICU: Ascorbic Acid Attenuates Hyperoxia-Compromised Host Defense Against Pulmonary Bacterial Infection

B31 PNEUMONIA, ACUTE RESPIRATORY INFECTION: Partially Desulfated Heparin Enhances Oxygen Toxicity- Compromised Immunity Against Pseudomonas Lung Infection

C37 STRUCTURAL AND HEMATOPOIETIC CELL INTERACTIONS: The Hyperoxia-Compromised Macrophage Functions Are Attenuated By Ethacrynic Acid Via Inhibition Of Nf-[kappa]b-Mediated Release Of Hmgb1

Corrigendum to “Carbon Nanomaterials Stimulate HMGB1 Release From Macrophages and Induce Cell Migration and Invasion”

B32 REGULATION OF INFLAMMATION AND LUNG INJURY I: Inhibition Of Extracellular Hmgb1 Attenuates Hyperoxia-Induced Inflammatory Acute Lung Injury

GTS-21, an α7nAChR agonist, increases pulmonary bacterial clearance in mice by restoring hyperoxia-compromised macrophage function

C39 REGULATION OF INFLAMMATION AND LUNG INJURY II: Nf-[kappa]b In Airway Epithelium Protects Against Hyperoxia-Induced Inflammatory Lung Injury

Hyperoxia in Cell Culture: A Non-apoptotic Programmed Cell Death

P-149 - 2-O, 3-O desulfated heparin (ODSH) improves bacterial clearance in cystic fibrosis by reducing HMGB1 release and improving phagocytosis of airway macrophages

2-O, 3-O desulfated heparin (ODSH) improves bacterial clearance in cystic fibrosis by reducing HMGB1 release and improving phagocytosis of airway macrophages

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