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Manganese and Vanadium Co-Exposure Induces Severe Neurotoxicity in the Olfactory System: Relevance to Metal-Induced Parkinsonism

International journal of molecular sciences, 2024-05, Vol.25 (10), p.5285

2024

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Proteolytic activation of proapoptotic kinase protein kinase Cδ by tumor necrosis factor α death receptor signaling in dopaminergic neurons during neuroinflammation

Journal of neuroinflammation, 2012-04, Vol.9 (1), p.82-82, Article 82

2012

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The Peptidyl-prolyl Isomerase Pin1 Up-regulation and Proapoptotic Function in Dopaminergic Neurons

The Journal of biological chemistry, , Vol.288 (30), p.21955-21971

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Emerging Microbiome Genetic Engineering Technology for Stable Levodopa Delivery in Parkinson’s Disease

The FASEB journal, 2022-05, Vol.36 (S1), p.n/a

2022

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Emerging Microbiome Genetic Engineering Technology for Stable Levodopa Delivery in Parkinson’s Disease

Free radical biology & medicine, 2022-02, Vol.180, p.s108

2022

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α-Synuclein RT-QuIC in the Submandibular Glands of Parkinson’s Disease Patients

Movement disorders, 2019-11, Vol.35 (2), p.268-278

2019

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Environmental Neurotoxic Pesticide Exposure Induces Gut Inflammation and Enteric Neuronal Degeneration by Impairing Enteric Glial Mitochondrial Function in Pesticide Models of Parkinson’s Disease: Potential Relevance to Gut-Brain Axis Inflammation in Disease Pathogenesis

The international journal of biochemistry & cell biology, 2022-05, Vol.147, p.106225-106225

2022

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Methamphetamine Induces Autophagy and Apoptosis in a Mesencephalic Dopaminergic Neuronal Culture Model

Annals of the New York Academy of Sciences, 2006-08, Vol.1074 (1), p.234-244

2006

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Novel NOX2 inhibitor Mito‐Apocynin Protects Against LPS‐induced Endotoxemia Pre‐Clinical Animal Model

The FASEB journal, 2019-04, Vol.33 (S1), p.lb39-lb39

2019

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Mitochondrial Impairment Upregulates MICOS Expression in a Human Microglial Cell Model

The FASEB journal, 2019-04, Vol.33 (S1), p.lb19-lb19

2019

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The Role of ZBP1 in the Neuroinflammatory Response in Glia Cell Models of Parkinson's Disease

The FASEB journal, 2019-04, Vol.33 (S1), p.lb16-lb16

2019

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Manganese exposure augments misfolded α‐synuclein‐induced proinflammatory M1 microglial phenotype and inflammasome activation

The FASEB journal, 2019-04, Vol.33 (S1), p.505.7-505.7

2019

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Calcium‐activated K+ channel KCa3.1 Plays a Pro‐inflammatory Role in α‐Synuclein Models of Parkinson's Disease

The FASEB journal, 2019-04, Vol.33 (S1), p.500.20-500.20

2019

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Mitochondrial Stress Modulates Elongator Protein 3 (ELP3) Function to Enhance Dopaminergic Neurotoxicity in Cell Culture and Animal Models of Parkinson's Disease

The FASEB journal, 2019-04, Vol.33 (S1), p.461.1-461.1

2019

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Ultra‐sensitive detection of pathological α‐synuclein in human tissues and biofluids using the RT‐QuIC Assay: Relevance to development of circulating and peripheral biomarkers for diagnosing α‐synucleinopathies and other protein misfolding diseases

The FASEB journal, 2019-04, Vol.33 (S1), p.lb88-lb88

2019

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Calcium‐activated K + channel K Ca 3.1 Plays a Pro‐inflammatory Role in α‐Synuclein Models of Parkinson's Disease

The FASEB journal, 2019-04, Vol.33 (S1)

2019

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Manganese Exposure Induces Neuroinflammation by Impairing Mitochondrial Dynamics in Astrocytes

The FASEB journal, 2017-04, Vol.31 (S1)

2017

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Manganese Exposure Activates NLRP3 Inflammasome Signaling and Propagates Exosomal Release of ASC in Microglial Cells

The FASEB journal, 2018-04, Vol.32 (S1), p.823.8-823.8

2018

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The Fyn‐dependent voltage‐gated potassium channel Kv1.3 modulates neuroinflammation and neurodegeneration in Parkinson's disease models

The FASEB journal, 2018-04, Vol.32 (S1), p.553.1-553.1

2018

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Environmental neurotoxicant manganese regulates exosome-mediated extracellular miRNAs in cell culture model of Parkinson's disease: Relevance to a-synuclein misfolding in metal neurotoxicity

Neurotoxicology (Park Forest South), 2018-01, Vol.64, p.267

2018

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Pesticide‐induced Mitochondrial Dysfunction Augments NLRP3 Inflammasome Signaling Pathway in Primary Microglia

The FASEB journal, 2015-04, Vol.29 (S1)

2015

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MITO‐APOCYNIN, A NOVEL MITOCHONDRIA‐TARGETED DERIVATIVE OF APOCYNIN, INHIBITS NLRP3 INFLAMMASOME ACTIVATION IN PRIMARY MURINE MICROGLIA IN DOPAMINERGIC NEUROTOXIC MODELS

The FASEB journal, 2016-04, Vol.30 (S1)

2016

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Microglial Kv1.3, a voltage‐gated potassium channel, mediates neuroinflammation and neurodegeneration in cell culture and animal models of Parkinson's disease

The FASEB journal, 2017-04, Vol.31 (S1)

2017

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Fyn kinase regulates microglial neuroinflammatory responses in cell culture and animal models of Parkinson’s disease (1055.7)

The FASEB journal, 2014-04, Vol.28 (S1), p.n/a

2014

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Chronic Low-Dose Oxidative Stress Induces Caspase-3-Dependent PKCδ Proteolytic Activation and Apoptosis in a Cell Culture Model of Dopaminergic Neurodegeneration

Annals of the New York Academy of Sciences, 2008-10, Vol.1139 (1), p.197-205

2008

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Manganese and Vanadium Co-Exposure Induces Severe Neurotoxicity in the Olfactory System: Relevance to Metal-Induced Parkinsonism

Proteolytic activation of proapoptotic kinase protein kinase Cδ by tumor necrosis factor α death receptor signaling in dopaminergic neurons during neuroinflammation

The Peptidyl-prolyl Isomerase Pin1 Up-regulation and Proapoptotic Function in Dopaminergic Neurons

Emerging Microbiome Genetic Engineering Technology for Stable Levodopa Delivery in Parkinson’s Disease

Emerging Microbiome Genetic Engineering Technology for Stable Levodopa Delivery in Parkinson’s Disease

α-Synuclein RT-QuIC in the Submandibular Glands of Parkinson’s Disease Patients

Environmental Neurotoxic Pesticide Exposure Induces Gut Inflammation and Enteric Neuronal Degeneration by Impairing Enteric Glial Mitochondrial Function in Pesticide Models of Parkinson’s Disease: Potential Relevance to Gut-Brain Axis Inflammation in Disease Pathogenesis

Methamphetamine Induces Autophagy and Apoptosis in a Mesencephalic Dopaminergic Neuronal Culture Model

Novel NOX2 inhibitor Mito‐Apocynin Protects Against LPS‐induced Endotoxemia Pre‐Clinical Animal Model

Mitochondrial Impairment Upregulates MICOS Expression in a Human Microglial Cell Model

The Role of ZBP1 in the Neuroinflammatory Response in Glia Cell Models of Parkinson's Disease

Manganese exposure augments misfolded α‐synuclein‐induced proinflammatory M1 microglial phenotype and inflammasome activation

Calcium‐activated K+ channel KCa3.1 Plays a Pro‐inflammatory Role in α‐Synuclein Models of Parkinson's Disease

Mitochondrial Stress Modulates Elongator Protein 3 (ELP3) Function to Enhance Dopaminergic Neurotoxicity in Cell Culture and Animal Models of Parkinson's Disease

Ultra‐sensitive detection of pathological α‐synuclein in human tissues and biofluids using the RT‐QuIC Assay: Relevance to development of circulating and peripheral biomarkers for diagnosing α‐synucleinopathies and other protein misfolding diseases

Calcium‐activated K + channel K Ca 3.1 Plays a Pro‐inflammatory Role in α‐Synuclein Models of Parkinson's Disease

Manganese Exposure Induces Neuroinflammation by Impairing Mitochondrial Dynamics in Astrocytes

Manganese Exposure Activates NLRP3 Inflammasome Signaling and Propagates Exosomal Release of ASC in Microglial Cells

The Fyn‐dependent voltage‐gated potassium channel Kv1.3 modulates neuroinflammation and neurodegeneration in Parkinson's disease models

Environmental neurotoxicant manganese regulates exosome-mediated extracellular miRNAs in cell culture model of Parkinson's disease: Relevance to a-synuclein misfolding in metal neurotoxicity

Pesticide‐induced Mitochondrial Dysfunction Augments NLRP3 Inflammasome Signaling Pathway in Primary Microglia

MITO‐APOCYNIN, A NOVEL MITOCHONDRIA‐TARGETED DERIVATIVE OF APOCYNIN, INHIBITS NLRP3 INFLAMMASOME ACTIVATION IN PRIMARY MURINE MICROGLIA IN DOPAMINERGIC NEUROTOXIC MODELS

Microglial Kv1.3, a voltage‐gated potassium channel, mediates neuroinflammation and neurodegeneration in cell culture and animal models of Parkinson's disease

Fyn kinase regulates microglial neuroinflammatory responses in cell culture and animal models of Parkinson’s disease (1055.7)

Chronic Low-Dose Oxidative Stress Induces Caspase-3-Dependent PKCδ Proteolytic Activation and Apoptosis in a Cell Culture Model of Dopaminergic Neurodegeneration

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