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Selective targeting of CREB‐binding protein/β‐catenin inhibits growth of and extracellular matrix remodelling by airway smooth muscle

British journal of pharmacology, 2016-12, Vol.173 (23), p.3327-3341

2016

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Dynamics of pro-inflammatory and anti-inflammatory cytokine release during acute inflammation in chronic obstructive pulmonary disease: an ex vivo study

Respiratory research, 2008-05, Vol.9 (1), p.47-47, Article 47

2008

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IL-13 and TH2 cytokine exposure triggers matrix metalloproteinase 7-mediated Fas ligand cleavage from bronchial epithelial cells

Journal of allergy and clinical immunology, 2010-08, Vol.126 (2), p.366-374.e8

2010

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Induction of Epithelial―Mesenchymal Transition in Primary Airway Epithelial Cells from Patients with Asthma by Transforming Growth Factor-β1

American journal of respiratory and critical care medicine, 2009-07, Vol.180 (2), p.122-133

2009

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The molecular and cellular mechanisms associated with the destruction of terminal bronchioles in COPD

The European respiratory journal, 2022-05, Vol.59 (5), p.2101411

2022

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Protocadherin-1 binds to SMAD3 and suppresses TGF-β1-induced gene transcription

American journal of physiology. Lung cellular and molecular physiology, 2015-10, Vol.309 (7), p.L725-L735

2015

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Mesenchymal stem cells for repair of the airway epithelium in asthma

Expert review of respiratory medicine, 2010-12, Vol.4 (6), p.747-758

2010

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Small Airways in Pulmonary Fibrosis: Revisiting an Old Question with New Tools / Reply

American journal of respiratory and critical care medicine, 2022-08, Vol.206 (4), p.517

2022

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Reply to Cottin: Small Airways in Pulmonary Fibrosis: Revisiting an Old Question with New Tools

American journal of respiratory and critical care medicine, 2022-08, Vol.206 (4), p.517-517

2022

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Reply to Janssen and Wouters: Loss of Alveolar Attachments as a Pathomechanistic Link between Small Airway Disease and Emphysema

American journal of respiratory and critical care medicine, 2020-04, Vol.201 (7), p.879-880

2020

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Reply to: Small Airways in Pulmonary Fibrosis: Revisiting an Old Question with New Tools

American journal of respiratory and critical care medicine, 2022-05

2022

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Small Airways Disease in Chronic Obstructive Pulmonary Disease: A 50 Years History

Barcelona respiratory network reviews, 2017-10, Vol.3 (4)

2017

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TGF-β1, pSmad-2/3, Smad-7, and β-Catenin Are Augmented in the Pulmonary Arteries from Patients with Idiopathic Pulmonary Fibrosis

Journal of clinical medicine, 2024-02, Vol.13 (4)

2024

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A31 MECHANISMS IN CYSTIC FIBROSIS AND OTHER BRONCHIECTATIC DISEASES: A Comparative Study of Tissue Remodeling and Inflammatory Immune Cell Infiltration in Patients Treated by Lung Transplantation for Severe COPD Who Have Either the ZZ Phenotype of Alpha-One Antitrypsin Deficiency or the Centrilobular Phenotype of Emphysematous Destruction Commonly Observed in Smokers

American journal of respiratory and critical care medicine, 2017-01, Vol.195

2017

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Vascular remodelling in IPF patients and its detrimental effect on lung physiology: potential role of endothelial to mesenchymal transition (EndMT)

ERJ open research, 2022-02, p.571

2022

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Protocadherin-1 binds to SMAD3 and suppresses TGF-[Beta]1-induced gene transcription

American journal of physiology. Lung cellular and molecular physiology, 2015-10, Vol.309 (7), p.L725

2015

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IL-13 and T sub(H)2 cytokine exposure triggers matrix metalloproteinase 7-mediated Fas ligand cleavage from bronchial epithelial cells

Journal of allergy and clinical immunology, 2010-08, Vol.126 (2), p.366-374.e8

2010

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IL-13 and T H2 cytokine exposure triggers matrix metalloproteinase 7–mediated Fas ligand cleavage from bronchial epithelial cells

Journal of allergy and clinical immunology, 2010, Vol.126 (2), p.366-374.e8

2010

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IL-13 and TH 2 cytokine exposure triggers matrix metalloproteinase 7–mediated Fas ligand cleavage from bronchial epithelial cells

Journal of allergy and clinical immunology, 2010, Vol.126 (2), p.366-374.e8

2010

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Induction of Epithelial-Mesenchymal Transition in Primary Airway Epithelial Cells from Patients with Asthma by Transforming Growth Factor-{beta}1

American journal of respiratory and critical care medicine, 2009-07, Vol.180 (2), p.122

2009

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Three Dimensional Imaging of Paraffin Embedded Human Lung Tissue Samples by Micro-Computed Tomography: e0126230

PloS one, 2015-06, Vol.10 (6)

2015

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73 - Chronic Obstructive Pulmonary Disease and Emphysema

Clinical Immunology, 2023, p.936-942

Sixth Edition, 2023

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Induction of epithelial-mesenchymal transition in primary airway epithelial cells from patients with asthma by transforming growth factor-beta1

American journal of respiratory and critical care medicine, 2009-07, Vol.180 (2), p.122-133

2009

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The role of tumour necrosis factor alpha in acute inflammation of human lung tissue

Hackett, Tillie-Louise

2005

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Selective targeting of CREB‐binding protein/β‐catenin inhibits growth of and extracellular matrix remodelling by airway smooth muscle

Dynamics of pro-inflammatory and anti-inflammatory cytokine release during acute inflammation in chronic obstructive pulmonary disease: an ex vivo study

IL-13 and TH2 cytokine exposure triggers matrix metalloproteinase 7-mediated Fas ligand cleavage from bronchial epithelial cells

Induction of Epithelial―Mesenchymal Transition in Primary Airway Epithelial Cells from Patients with Asthma by Transforming Growth Factor-β1

The molecular and cellular mechanisms associated with the destruction of terminal bronchioles in COPD

Protocadherin-1 binds to SMAD3 and suppresses TGF-β1-induced gene transcription

Mesenchymal stem cells for repair of the airway epithelium in asthma

Small Airways in Pulmonary Fibrosis: Revisiting an Old Question with New Tools / Reply

Reply to Cottin: Small Airways in Pulmonary Fibrosis: Revisiting an Old Question with New Tools

Reply to Janssen and Wouters: Loss of Alveolar Attachments as a Pathomechanistic Link between Small Airway Disease and Emphysema

Reply to: Small Airways in Pulmonary Fibrosis: Revisiting an Old Question with New Tools

Small Airways Disease in Chronic Obstructive Pulmonary Disease: A 50 Years History

TGF-β1, pSmad-2/3, Smad-7, and β-Catenin Are Augmented in the Pulmonary Arteries from Patients with Idiopathic Pulmonary Fibrosis

Vascular remodelling in IPF patients and its detrimental effect on lung physiology: potential role of endothelial to mesenchymal transition (EndMT)

Protocadherin-1 binds to SMAD3 and suppresses TGF-[Beta]1-induced gene transcription

IL-13 and T sub(H)2 cytokine exposure triggers matrix metalloproteinase 7-mediated Fas ligand cleavage from bronchial epithelial cells

IL-13 and T H2 cytokine exposure triggers matrix metalloproteinase 7–mediated Fas ligand cleavage from bronchial epithelial cells

IL-13 and TH 2 cytokine exposure triggers matrix metalloproteinase 7–mediated Fas ligand cleavage from bronchial epithelial cells

Induction of Epithelial-Mesenchymal Transition in Primary Airway Epithelial Cells from Patients with Asthma by Transforming Growth Factor-{beta}1

Three Dimensional Imaging of Paraffin Embedded Human Lung Tissue Samples by Micro-Computed Tomography: e0126230

73 - Chronic Obstructive Pulmonary Disease and Emphysema

Induction of epithelial-mesenchymal transition in primary airway epithelial cells from patients with asthma by transforming growth factor-beta1

The role of tumour necrosis factor alpha in acute inflammation of human lung tissue

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