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Prostaglandin E2 amplifies IL-17 production by γδ T cells during barrier inflammation

Cell reports (Cambridge), 2021-07, Vol.36 (4), p.109456, Article 109456

2021

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CD109 Restrains Activation of Cutaneous IL-17-Producing γδ T Cells by Commensal Microbiota

Cell reports (Cambridge), 2019-10, Vol.29 (2), p.391-405.e5

2019

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Understanding mechanisms of disease tolerance during intestinal helminth infection

The Journal of immunology (1950), 2019-05, Vol.202 (1_Supplement), p.190-190.27

2019

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Lipopolysaccharides Impair Insulin Gene Expression in Isolated Islets of Langerhans via Toll-Like Receptor-4 and NF-[kappa]B Signalling

PloS one, 2012-04, Vol.7 (4)

2012

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Dual-Reporter [beta]-Cell-Specific Male Transgenic Rats for the Analysis of [beta]-Cell Functional Mass and Enrichment by Flow Cytometry

Endocrinology (Philadelphia), 2016-03, Vol.157 (3), p.1299-1306

2016

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Prostaglandin E 2 amplifies IL-17 production by γδ T cells during barrier inflammation

Cell reports (Cambridge), 2021-07, Vol.36 (4), p.109456

2021

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Involvement of Per-Arnt-Sim kinase and extracellular-regulated kinases-1/2 in palmitate inhibition of insulin gene expression in pancreatic [beta]-cells

Diabetes (New York, N.Y.), 2009-09, Vol.58 (9), p.2048

2009

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Lack of TXNIP Protects against mitochondria-mediated apoptosis but not against fatty acid-induced ER stress-mediated [beta]-cell death

Diabetes (New York, N.Y.), 2010-02, Vol.59 (2), p.440

2010

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The ΔF508 Gene Mutation of Cystic Fibrosis Transmembrane Regulator Protein Leads to a Progressive Decline of Beta-Cell Function in Mice Carrying This Mutation

Canadian journal of diabetes, 2013-10, Vol.37, p.S57-S57

2013

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The [Delta]F508 Mutation in the Cystic Fibrosis Transmembrane Conductance Regulator Is Associated With Progressive Insulin Resistance and Decreased Functional [Beta]-Cell Mass in Mice

Diabetes (New York, N.Y.), 2015-12, Vol.64 (12), p.4112

2015

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Epidermal Growth Factor Receptor Signaling Promotes Pancreatic [Beta]-Cell Proliferation in Response to Nutrient Excess in Rats Through mTOR and FOXM1

Diabetes (New York, N.Y.), 2014-03, Vol.63 (3), p.982

2014

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A Model of Chronic Nutrient Infusion in the Rat

Journal of visualized experiments, 2013-08 (78)

2013

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Miniglucagon (Glucagon 19-29)

Diabetes (New York, N.Y.), 2002-02, Vol.51 (2), p.406-412

2002

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Epidermal Growth Factor Signalling Promotes Pancreatic Beta-Cell Proliferation in Response to Nutrient Excess in Rats Through MTOR And FOXM1

Canadian journal of diabetes, 2013-12, Vol.37 (6), p.433-433

2013

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PAS Kinase Regulates PDX-1 Protein Stability Via Phosphorylation of GSK3β in Pancreatic Beta Cells

Canadian journal of diabetes, 2013-10, Vol.37, p.S59-S59

2013

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Epidermal Growth Factor Signalling Promotes Pancreatic Beta-Cell Proliferation In Response to Nutrient Excess tn Rats Through MTOR And FOXM1

Canadian journal of diabetes, 2013-10, Vol.37, p.S8-S8

2013

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The Glucagon-Miniglucagon Interplay

Annals of the New York Academy of Sciences, 2006-07, Vol.1070 (1), p.161-166

2006

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2 - A role for ER stress and JNK in fatty acid inhibition of the insulin gene

Canadian journal of diabetes, 2009, Vol.33 (1), p.55-56

2009

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A role for ER stress and JNK in fatty acid inhibition of the insulin gene

Canadian journal of diabetes, 2009, Vol.33 (1), p.55-56

2009

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PAS Kinase Mediates Palmitate Inhibition of Insulin Gene Expression in Pancreatic Beta-Cells

Canadian journal of diabetes, 2008, Vol.32 (4), p.303-303

2008

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A Role for ER Stress and JNK in Fatty Acid Inhibition of the Insulin Gene

Canadian journal of diabetes, 2008, Vol.32 (4), p.338-338

2008

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Miniglucagon (MG)-generating endopeptidase, which processes glucagon into MG, is composed of N-arginine dibasic convertase and aminopeptidase B

Endocrinology (Philadelphia), 2005-02, Vol.146 (2), p.702-12

2005

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Prostaglandin E2 amplifies IL-17 production by γδ T cells during barrier inflammation

CD109 Restrains Activation of Cutaneous IL-17-Producing γδ T Cells by Commensal Microbiota

Understanding mechanisms of disease tolerance during intestinal helminth infection

Lipopolysaccharides Impair Insulin Gene Expression in Isolated Islets of Langerhans via Toll-Like Receptor-4 and NF-[kappa]B Signalling

Dual-Reporter [beta]-Cell-Specific Male Transgenic Rats for the Analysis of [beta]-Cell Functional Mass and Enrichment by Flow Cytometry

Prostaglandin E 2 amplifies IL-17 production by γδ T cells during barrier inflammation

Involvement of Per-Arnt-Sim kinase and extracellular-regulated kinases-1/2 in palmitate inhibition of insulin gene expression in pancreatic [beta]-cells

Lack of TXNIP Protects against mitochondria-mediated apoptosis but not against fatty acid-induced ER stress-mediated [beta]-cell death

The ΔF508 Gene Mutation of Cystic Fibrosis Transmembrane Regulator Protein Leads to a Progressive Decline of Beta-Cell Function in Mice Carrying This Mutation

The [Delta]F508 Mutation in the Cystic Fibrosis Transmembrane Conductance Regulator Is Associated With Progressive Insulin Resistance and Decreased Functional [Beta]-Cell Mass in Mice

Epidermal Growth Factor Receptor Signaling Promotes Pancreatic [Beta]-Cell Proliferation in Response to Nutrient Excess in Rats Through mTOR and FOXM1

A Model of Chronic Nutrient Infusion in the Rat

Miniglucagon (Glucagon 19-29)

Epidermal Growth Factor Signalling Promotes Pancreatic Beta-Cell Proliferation in Response to Nutrient Excess in Rats Through MTOR And FOXM1

PAS Kinase Regulates PDX-1 Protein Stability Via Phosphorylation of GSK3β in Pancreatic Beta Cells

Epidermal Growth Factor Signalling Promotes Pancreatic Beta-Cell Proliferation In Response to Nutrient Excess tn Rats Through MTOR And FOXM1

The Glucagon-Miniglucagon Interplay

2 - A role for ER stress and JNK in fatty acid inhibition of the insulin gene

A role for ER stress and JNK in fatty acid inhibition of the insulin gene

PAS Kinase Mediates Palmitate Inhibition of Insulin Gene Expression in Pancreatic Beta-Cells

A Role for ER Stress and JNK in Fatty Acid Inhibition of the Insulin Gene

Miniglucagon (MG)-generating endopeptidase, which processes glucagon into MG, is composed of N-arginine dibasic convertase and aminopeptidase B

Contraintes et spécificités de l’évaluation clinique des dispositifs médicaux

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