UB Paderborn / Katalog / Suche / in CDI

1 – 25 von 37

Sortiert nach

A unique subset of low-risk Wilms tumors is characterized by loss of function of TRIM28

PloS one, 2018-12, Vol.13 (12), p.e0208936

2018

Volltextzugriff (PDF)

Knock-in of the Wt1 R394W mutation causes MDS and cooperates with Flt3/ITD to drive aggressive myeloid neoplasms in mice

Oncotarget, 2018-10, Vol.9 (82), p.35313-35326

2018

Volltextzugriff (PDF)

Abstract LB-180: The genetic landscape of Wilms tumor

Cancer research (Chicago, Ill.), 2016-07, Vol.76 (14_Supplement), p.LB-180-LB-180

2016

Volltextzugriff (PDF)

Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with β-Catenin Activation or Wt1 Ablation and Igf2 Upregulation1

Neoplasia (New York, N.Y.), 2016-02, Vol.18 (2), p.71-81

2016

Volltextzugriff (PDF)

Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with [beta]-Catenin Activation or Wt1 Ablation and Igf2 Upregulation

Neoplasia (New York, N.Y.), 2016-02, Vol.18 (2), p.71-81

2016

Volltextzugriff (PDF)

A Wilms Tumor 1 (WT1) Mutation Causes Myelodysplastic Syndrome in a Knock-in Mouse Model, and a Mixed Myelodysplastic/Myeloproliferative Neoplam in Double Knock-in Mice with WT1 and FLT3/ITD Mutations

Blood, 2015-12, Vol.126 (23), p.312-312

2015

Volltextzugriff (PDF)

Identification of germline DICER1 mutations and loss of heterozygosity in familial Wilms tumor using whole genome sequencing

Journal of medical genetics, 2015-11, Vol.53 (6), p.385-388

2015

Volltextzugriff (PDF)

Clinical outcome and biological predictors of relapse following nephrectomy only for very low risk Wilms tumor (VLR WT): A report from Children’s Oncology Group AREN0532

Journal of clinical oncology, 2015-05, Vol.33 (15_suppl), p.10023-10023

2015

Volltextzugriff (PDF)

Reprogramming of Sertoli cells to fetal-like Leydig cells byWt1ablation

Proceedings of the National Academy of Sciences - PNAS, 2015-03, Vol.112 (13), p.4003-4008

2015

Volltextzugriff (PDF)

Characterization of the Inflammatory Microenvironment and Identification of Potential Therapeutic Targets in Wilms Tumors12

Translational oncology, 2014-06, Vol.7 (4), p.484-492

2014

Volltextzugriff (PDF)

The Wilms Tumor Gene, Wt1, Is Critical for Mouse Spermatogenesis via Regulation of Sertoli Cell Polarity and Is Associated with Non-Obstructive Azoospermia in Humans: e1003645

PLoS genetics, 2013-08, Vol.9 (8)

2013

Volltextzugriff (PDF)

Abstract 5047: Impact of kidney progenitor cell differentiation status in Wilms tumorigenesis

Cancer research (Chicago, Ill.), 2013-04, Vol.73 (8_Supplement), p.5047-5047

2013

Volltextzugriff (PDF)

Monitoring Therapy with MEK Inhibitor U0126 in a Novel Wilms Tumor Model in Wt1 Knockout Igf2 Transgenic Mice Using ^sup 18^F-FDG PET with Dual-Contrast Enhanced CT and MRI: Early Metabolic Response Without Inhibition of Tumor Growth

Molecular imaging and biology, 2013-04, Vol.15 (2), p.175

2013

Volltextzugriff (PDF)

Monitoring Therapy with MEK Inhibitor U0126 in a Novel Wilms Tumor Model in Wt1 Knockout Igf2 Transgenic Mice Using super(18)F-FDG PET with Dual-Contrast Enhanced CT and MRI: Early Metabolic Response Without Inhibition of Tumor Growth

Molecular imaging and biology, 2013-04, Vol.15 (2), p.175-185

2013

Volltextzugriff (PDF)

Monitoring Therapy with MEK Inhibitor U0126 in a Novel Wilms Tumor Model in Wt1 Knockout Igf2 Transgenic Mice Using 18F-FDG PET with Dual-Contrast Enhanced CT and MRI: Early Metabolic Response Without Inhibition of Tumor Growth

Molecular imaging and biology, 2013-04, Vol.15 (2), p.175-185

2013

Volltextzugriff (PDF)

Clinically Relevant Subsets Identified by Gene Expression Patterns Support a Revised Ontogenic Model of Wilms Tumor: A Children's Oncology Group Study12

Neoplasia (New York, N.Y.), 2012-08, Vol.14 (8), p.742-756

2012

Volltextzugriff (PDF)

Abstract 1439: Loss of expression of SWI/SNF subunits in high risk Wilms tumor is accompanied by global gene expression changes

Cancer research (Chicago, Ill.), 2012-04, Vol.72 (8_Supplement), p.1439-1439

2012

Volltextzugriff (PDF)

Reply to B. Royer-Pokora et al

Journal of clinical oncology, 2011-06, Vol.29 (16), p.e487-e488

2011

Volltextzugriff (PDF)

Abstract 5353: Comprehensive Ggnomic analysis of diffuse hyperplastic perilobar nephroblastomatosis (DHPLN)

Cancer research (Chicago, Ill.), 2011-04, Vol.71 (8_Supplement), p.5353-5353

2011

Volltextzugriff (PDF)

Wt1 negatively regulates Î²-catenin signaling during testis development

Development (Cambridge), 2008-05, Vol.135 (10), p.1875

2008

Volltextzugriff (PDF)

UNIVERSAL NEPHROBLASTOMATOSIS WITH BILATERAL HYPERPLASTIC NEPHROMEGALY IN SIBLINGS

Pediatric and developmental pathology, 2006, Vol.preprint (2008), p.1

2006

Volltextzugriff (PDF)

Genome-wide loss of heterozygosity analysis of WT1-wild-type and WT1-mutant Wilms tumors

Genes chromosomes & cancer, 2005-06, Vol.43 (2), p.172-180

2005

Volltextzugriff (PDF)

Genome-wide loss of heterozygosity analysis ofWT1-wild-type andWT1-mutant Wilms tumors

Genes chromosomes & cancer, 2005, Vol.43 (2), p.172-180

2005

Volltextzugriff (PDF)

Loss of heterozygosity for chromosomes 1p and 16q is an adverse prognostic factor in favorable-histology wilms tumor : A report from the national wilms tumor study group: Molecular oncology: genomics

Journal of clinical oncology, 2005, Vol.23 (29), p.7312-7321

2005

Volltextzugriff (PDF)

The Wt1+/R394W Mouse Displays Glomerulosclerosis and Early-Onset Renal Failure Characteristic of Human Denys-Drash Syndrome

Molecular and Cellular Biology, 2004-11, Vol.24 (22), p.9899-9910

2004

Volltextzugriff (PDF)

1 – 25 von 37

Suche als RSS-Feed

Aktive Filter

ThemaScience & Technology

SpracheEnglisch

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(29)
Mit Volltextzugriff
(34)

Publikationsform

Sprache

Japanisch
(2)

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Zeitschrift / Serie

5 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

A unique subset of low-risk Wilms tumors is characterized by loss of function of TRIM28

Knock-in of the Wt1 R394W mutation causes MDS and cooperates with Flt3/ITD to drive aggressive myeloid neoplasms in mice

Abstract LB-180: The genetic landscape of Wilms tumor

Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with β-Catenin Activation or Wt1 Ablation and Igf2 Upregulation1

Nephron Progenitor But Not Stromal Progenitor Cells Give Rise to Wilms Tumors in Mouse Models with [beta]-Catenin Activation or Wt1 Ablation and Igf2 Upregulation

A Wilms Tumor 1 (WT1) Mutation Causes Myelodysplastic Syndrome in a Knock-in Mouse Model, and a Mixed Myelodysplastic/Myeloproliferative Neoplam in Double Knock-in Mice with WT1 and FLT3/ITD Mutations

Identification of germline DICER1 mutations and loss of heterozygosity in familial Wilms tumor using whole genome sequencing

Clinical outcome and biological predictors of relapse following nephrectomy only for very low risk Wilms tumor (VLR WT): A report from Children’s Oncology Group AREN0532

Reprogramming of Sertoli cells to fetal-like Leydig cells byWt1ablation

Characterization of the Inflammatory Microenvironment and Identification of Potential Therapeutic Targets in Wilms Tumors12

The Wilms Tumor Gene, Wt1, Is Critical for Mouse Spermatogenesis via Regulation of Sertoli Cell Polarity and Is Associated with Non-Obstructive Azoospermia in Humans: e1003645

Abstract 5047: Impact of kidney progenitor cell differentiation status in Wilms tumorigenesis

Monitoring Therapy with MEK Inhibitor U0126 in a Novel Wilms Tumor Model in Wt1 Knockout Igf2 Transgenic Mice Using ^sup 18^F-FDG PET with Dual-Contrast Enhanced CT and MRI: Early Metabolic Response Without Inhibition of Tumor Growth

Monitoring Therapy with MEK Inhibitor U0126 in a Novel Wilms Tumor Model in Wt1 Knockout Igf2 Transgenic Mice Using super(18)F-FDG PET with Dual-Contrast Enhanced CT and MRI: Early Metabolic Response Without Inhibition of Tumor Growth

Monitoring Therapy with MEK Inhibitor U0126 in a Novel Wilms Tumor Model in Wt1 Knockout Igf2 Transgenic Mice Using 18F-FDG PET with Dual-Contrast Enhanced CT and MRI: Early Metabolic Response Without Inhibition of Tumor Growth

Clinically Relevant Subsets Identified by Gene Expression Patterns Support a Revised Ontogenic Model of Wilms Tumor: A Children's Oncology Group Study12

Abstract 1439: Loss of expression of SWI/SNF subunits in high risk Wilms tumor is accompanied by global gene expression changes

Reply to B. Royer-Pokora et al

Abstract 5353: Comprehensive Ggnomic analysis of diffuse hyperplastic perilobar nephroblastomatosis (DHPLN)

Wt1 negatively regulates Î²-catenin signaling during testis development

UNIVERSAL NEPHROBLASTOMATOSIS WITH BILATERAL HYPERPLASTIC NEPHROMEGALY IN SIBLINGS

Genome-wide loss of heterozygosity analysis of WT1-wild-type and WT1-mutant Wilms tumors

Genome-wide loss of heterozygosity analysis ofWT1-wild-type andWT1-mutant Wilms tumors

Loss of heterozygosity for chromosomes 1p and 16q is an adverse prognostic factor in favorable-histology wilms tumor : A report from the national wilms tumor study group: Molecular oncology: genomics

The Wt1+/R394W Mouse Displays Glomerulosclerosis and Early-Onset Renal Failure Characteristic of Human Denys-Drash Syndrome

Aktive Filter

Suchergebnisse filtern