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The Success and Failure of the Schwann Cell Response to Nerve Injury

Frontiers in cellular neuroscience, 2019-02, Vol.13, p.33-33

2019

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Schwann Cell Precursors; Multipotent Glial Cells in Embryonic Nerves

Frontiers in molecular neuroscience, 2019-03, Vol.12, p.69-69

2019

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The Role of Cell Plasticity in Tissue Repair: Adaptive Cellular Reprogramming

Developmental cell, 2015-09, Vol.34 (6), p.613-620

2015

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The Role of c-Jun and Autocrine Signaling Loops in the Control of Repair Schwann Cells and Regeneration

Frontiers in cellular neuroscience, 2022-02, Vol.15, p.820216-820216

2022

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STAT3 Controls the Long-Term Survival and Phenotype of Repair Schwann Cells during Nerve Regeneration

The Journal of neuroscience, 2017-04, Vol.37 (16), p.4255-4269

2017

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Changes in the Coding and Non-coding Transcriptome and DNA Methylome that Define the Schwann Cell Repair Phenotype after Nerve Injury

Cell reports (Cambridge), 2017-09, Vol.20 (11), p.2719-2734

2017

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Failures of nerve regeneration caused by aging or chronic denervation are rescued by restoring Schwann cell c-Jun

eLife, 2021-01, Vol.10

2021

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Isolation of Schwann Cell Precursors from Rodents

Methods in molecular biology (Clifton, N.J.), 2018-01, Vol.1739, p.3

2018

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Characterising cellular and molecular features of human peripheral nerve degeneration

Acta neuropathologica communications, 2020-04, Vol.8 (1), p.51-17, Article 51

2020

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c-Jun Reprograms Schwann Cells of Injured Nerves to Generate a Repair Cell Essential for Regeneration

Neuron (Cambridge, Mass.), 2012-08, Vol.75 (4), p.633-647

2012

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Negative regulation of myelination: Relevance for development, injury, and demyelinating disease

Glia, 2008-11, Vol.56 (14), p.1552-1565

2008

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Denervated Schwann Cells Attract Macrophages by Secretion of Leukemia Inhibitory Factor (LIF) and Monocyte Chemoattractant Protein-1 in a Process Regulated by Interleukin-6 and LIF

The Journal of neuroscience, 2002-08, Vol.22 (15), p.6696-6703

2002

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The origin and development of glial cells in peripheral nerves

Nature reviews. Neuroscience, 2005-09, Vol.6 (9), p.671-682

2005

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Regulation of Schwann cell differentiation and proliferation by the Pax-3 transcription factor

Glia, 2012-09, Vol.60 (9), p.1269-1278

2012

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The molecular profile of nerve repair: humans mirror rodents

Neural regeneration research, 2021-07, Vol.16 (7), p.1440-1441

2021

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Schwann cell autophagy, myelinophagy, initiates myelin clearance from injured nerves

The Journal of cell biology, 2015-07, Vol.210 (1), p.153-168

2015

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After Nerve Injury, Lineage Tracing Shows That Myelin and Remak Schwann Cells Elongate Extensively and Branch to Form Repair Schwann Cells, Which Shorten Radically on Remyelination

The Journal of neuroscience, 2017-09, Vol.37 (37), p.9086-9099

2017

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c-Jun is a negative regulator of myelination

The Journal of cell biology, 2008-05, Vol.181 (4), p.625-637

2008

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Reprogrammed Schwann Cells Organize into Dynamic Tracks that Promote Pancreatic Cancer Invasion

Cancer discovery, 2022-10, Vol.12 (10), p.2454-2473

2022

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Glial cells

The international journal of biochemistry & cell biology, 2004-10, Vol.36 (10), p.1861-1867

Jessen, Kristjan R

2004

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S-adenosylmethionine Levels Regulate the Schwann Cell DNA Methylome

Neuron (Cambridge, Mass.), 2014-03, Vol.81 (5), p.1024-1039

2014

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Graded Elevation of c-Jun in Schwann Cells In Vivo : Gene Dosage Determines Effects on Development, Remyelination, Tumorigenesis, and Hypomyelination

The Journal of neuroscience, 2017-12, Vol.37 (50), p.12297-12313

2017

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Mouse schwann cells need both NRG1 and cyclic AMP to myelinate

Glia, 2011-05, Vol.59 (5), p.720-733

2011

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c-Jun activation in Schwann cells protects against loss of sensory axons in inherited neuropathy

Brain (London, England : 1878), 2014-11, Vol.137 (Pt 11), p.2922-2937

2014

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A double point mutation in the DNA-binding region of Egr2 switches its function from inhibition to induction of proliferation: A potential contribution to the development of congenital hypomyelinating neuropathy

Neurobiology of disease, 2006-10, Vol.24 (1), p.159-169

2006

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The Success and Failure of the Schwann Cell Response to Nerve Injury

Schwann Cell Precursors; Multipotent Glial Cells in Embryonic Nerves

The Role of Cell Plasticity in Tissue Repair: Adaptive Cellular Reprogramming

The Role of c-Jun and Autocrine Signaling Loops in the Control of Repair Schwann Cells and Regeneration

STAT3 Controls the Long-Term Survival and Phenotype of Repair Schwann Cells during Nerve Regeneration

Changes in the Coding and Non-coding Transcriptome and DNA Methylome that Define the Schwann Cell Repair Phenotype after Nerve Injury

Failures of nerve regeneration caused by aging or chronic denervation are rescued by restoring Schwann cell c-Jun

Isolation of Schwann Cell Precursors from Rodents

Characterising cellular and molecular features of human peripheral nerve degeneration

c-Jun Reprograms Schwann Cells of Injured Nerves to Generate a Repair Cell Essential for Regeneration

Negative regulation of myelination: Relevance for development, injury, and demyelinating disease

Denervated Schwann Cells Attract Macrophages by Secretion of Leukemia Inhibitory Factor (LIF) and Monocyte Chemoattractant Protein-1 in a Process Regulated by Interleukin-6 and LIF

The origin and development of glial cells in peripheral nerves

Regulation of Schwann cell differentiation and proliferation by the Pax-3 transcription factor

The molecular profile of nerve repair: humans mirror rodents

Schwann cell autophagy, myelinophagy, initiates myelin clearance from injured nerves

After Nerve Injury, Lineage Tracing Shows That Myelin and Remak Schwann Cells Elongate Extensively and Branch to Form Repair Schwann Cells, Which Shorten Radically on Remyelination

c-Jun is a negative regulator of myelination

Reprogrammed Schwann Cells Organize into Dynamic Tracks that Promote Pancreatic Cancer Invasion

Glial cells

S-adenosylmethionine Levels Regulate the Schwann Cell DNA Methylome

Graded Elevation of c-Jun in Schwann Cells In Vivo : Gene Dosage Determines Effects on Development, Remyelination, Tumorigenesis, and Hypomyelination

Mouse schwann cells need both NRG1 and cyclic AMP to myelinate

c-Jun activation in Schwann cells protects against loss of sensory axons in inherited neuropathy

A double point mutation in the DNA-binding region of Egr2 switches its function from inhibition to induction of proliferation: A potential contribution to the development of congenital hypomyelinating neuropathy

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