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Enhancement of lung tumorigenesis in a Gprc5a Knockout mouse by chronic extrinsic airway inflammation

Molecular cancer, 2012-01, Vol.11 (1), p.4-4, Article 4

2012

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Disruption of the Caveolin-1 Gene Impairs Renal Calcium Reabsorption and Leads to Hypercalciuria and Urolithiasis

The American journal of pathology, 2003-04, Vol.162 (4), p.1241-1248

2003

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Comparable molecular alterations in 4-nitroquinoline 1-oxide-induced oral and esophageal cancer in mice and in human esophageal cancer, associated with poor prognosis of patients

In vivo (Athens), 2013-07, Vol.27 (4), p.473-484

2013

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Porcine leptin receptor: Molecular structure and expression in the ovary

Molecular reproduction and development, 2000-08, Vol.56 (4), p.465-474

2000

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Abstract A38: Aberrant expression of cell growth-critical genes in 4-nitroquinoline 1-oxide-induced mouse tumors and in human esophageal cancer tissues

Cancer prevention research (Philadelphia, Pa.), 2012-11, Vol.5 (11_Supplement), p.A38-A38

2012

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Identification of the Retinoic Acid–Inducible Gprc5a As a New Lung Tumor Suppressor Gene

JNCI : Journal of the National Cancer Institute, 2007-11, Vol.99 (22), p.1668-1682

2007

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Three-year survival in stage IV non-small cell lung cancer (NSCLC): The importance of metastatic distribution

Journal of clinical oncology, 2012-05, Vol.30 (15_suppl), p.7608-7608

2012

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Gprc5a deletion enhances the transformed phenotype in normal and malignant lung epithelial cells by eliciting persistent Stat3 signaling induced by autocrine Lif

Cancer research (Chicago, Ill.), 2010-10, Vol.70 (21), p.8917-8926

2010

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Abstract 4995: Loss of the lung-specific tumor suppressor Gprc5a causes persistent activation of Stat3 leading to increased survival signaling in normal and malignant lung epithelial cells

Cancer research (Chicago, Ill.), 2010-04, Vol.70 (8_Supplement), p.4995-4995

2010

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Abstract 2466: Tobacco carcinogen-enhanced lung tumorigenesis in Gprc5a knockout mice recapitulates human lung cancer development

Cancer research (Chicago, Ill.), 2010-04, Vol.70 (8_Supplement), p.2466-2466

2010

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A Gprc5a Tumor Suppressor Loss of Expression Signature Is Conserved, Prevalent, and Associated with Survival in Human Lung Adenocarcinomas12

Neoplasia (New York, N.Y.), 2010-06, Vol.12 (6), p.499-505

2010

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Antitumor Effect of Retinoic Acid Receptor-β2 Associated with Suppression of Cyclooxygenase-2

Cancer prevention research (Philadelphia, Pa.), 2009-03, Vol.2 (3), p.274-280

2009

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Three-Year Survival in Stage IV Non-Small Cell Lung Cancer (NSCLC): The Importance of Metastatic Distribution

Open journal of thoracic surgery, 2013, Vol.3 (2), p.42-46

2013

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Porcine Leptin Receptor: Molecular Structure and Expression in the Ovary

Molecular reproduction and development, 2000-08, Vol.56 (4), p.465-474

2000

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Identification of the Retinoic Acid-lnducible Gprc5a As a New Lung Tumor Suppressor Gene

JNCI : Journal of the National Cancer Institute, 2007, Vol.99 (22), p.1668-1682

2007

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Antitumor effect of retinoic acid receptor-beta2 associated with suppression of cyclooxygenase-2

Cancer prevention research (Philadelphia, Pa.), 2009-03, Vol.2 (3), p.274-280

2009

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Studies on changes of serum progesterone level of sables in the pregnancy period

Shou lei xuebao, 1993-02, Vol.13 (1)

1993

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Enhancement of lung tumorigenesis in a Gprc5a Knockout mouse by chronic extrinsic airway inflammation

Disruption of the Caveolin-1 Gene Impairs Renal Calcium Reabsorption and Leads to Hypercalciuria and Urolithiasis

Comparable molecular alterations in 4-nitroquinoline 1-oxide-induced oral and esophageal cancer in mice and in human esophageal cancer, associated with poor prognosis of patients

Porcine leptin receptor: Molecular structure and expression in the ovary

Abstract A38: Aberrant expression of cell growth-critical genes in 4-nitroquinoline 1-oxide-induced mouse tumors and in human esophageal cancer tissues

Identification of the Retinoic Acid–Inducible Gprc5a As a New Lung Tumor Suppressor Gene

Three-year survival in stage IV non-small cell lung cancer (NSCLC): The importance of metastatic distribution

Gprc5a deletion enhances the transformed phenotype in normal and malignant lung epithelial cells by eliciting persistent Stat3 signaling induced by autocrine Lif

Abstract 4995: Loss of the lung-specific tumor suppressor Gprc5a causes persistent activation of Stat3 leading to increased survival signaling in normal and malignant lung epithelial cells

Abstract 2466: Tobacco carcinogen-enhanced lung tumorigenesis in Gprc5a knockout mice recapitulates human lung cancer development

A Gprc5a Tumor Suppressor Loss of Expression Signature Is Conserved, Prevalent, and Associated with Survival in Human Lung Adenocarcinomas12

Antitumor Effect of Retinoic Acid Receptor-β2 Associated with Suppression of Cyclooxygenase-2

Three-Year Survival in Stage IV Non-Small Cell Lung Cancer (NSCLC): The Importance of Metastatic Distribution

Porcine Leptin Receptor: Molecular Structure and Expression in the Ovary

Identification of the Retinoic Acid-lnducible Gprc5a As a New Lung Tumor Suppressor Gene

Antitumor effect of retinoic acid receptor-beta2 associated with suppression of cyclooxygenase-2

Studies on changes of serum progesterone level of sables in the pregnancy period

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