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TRPM8 is the principal mediator of menthol-induced analgesia of acute and inflammatory pain

Pain (Amsterdam), 2013-10, Vol.154 (10), p.2169-2177

2013

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Targeting the nitric oxide/cGMP signaling pathway to treat chronic pain

Neural regeneration research, 2023-05, Vol.18 (5), p.996-1003

2023

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An Introduction to Pain Pathways and Pain ``Targets

Molecular and Cell Biology of Pain, 2015, Vol.131, p.1-30

Das, Vaskar

2015

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AMPK activation attenuates inflammatory pain through inhibiting NF-κB activation and IL-1β expression

Journal of neuroinflammation, 2019-02, Vol.16 (1), p.34-34, Article 34

2019

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GABAB receptors and pain

Neuropharmacology, 2018-07, Vol.136, p.102-105

Malcangio, Marzia

2018

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Central and peripheral sites of action for CB 2 receptor mediated analgesic activity in chronic inflammatory and neuropathic pain models in rats

British journal of pharmacology, 2011-01, Vol.162 (2), p.428-440

2011

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Macrophages transfer mitochondria to sensory neurons to resolve inflammatory pain

Neuron (Cambridge, Mass.), 2022-02, Vol.110 (4), p.613-626.e9

2022

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Kinins and their B1 and B2 receptors as potential therapeutic targets for pain relief

Life sciences (1973), 2023-02, Vol.314, p.121302, Article 121302

2023

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CircRNA expression profiling of the rat thalamus in temporomandibular joint chronic inflammatory pain

Gene, 2025-01, Vol.934, p.149024, Article 149024

2025

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Cannabinoids, the endocannabinoid system, and pain: a review of preclinical studies

Pain (Amsterdam), 2021-07, Vol.162 (Suppl 1), p.S5-S25

2021

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In vitro and in vivo characterization of A‐796260: a selective cannabinoid CB 2 receptor agonist exhibiting analgesic activity in rodent pain models

British journal of pharmacology, 2009-01, Vol.153 (2), p.390-401

2009

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MEL endomorphins act as potent inflammatory analgesics with the inhibition of activated non-neuronal cells and modulation of pro-inflammatory cytokines

Neuropharmacology, 2020-05, Vol.168, p.107992-107992, Article 107992

2020

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Epigenetic mechanisms of chronic pain

Trends in neurosciences (Regular ed.), 2015-04, Vol.38 (4), p.237-246

2015

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TRPA1 as a therapeutic target for nociceptive pain

Expert opinion on therapeutic targets, 2020-10, Vol.24 (10), p.997-1008

2020

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Soluble epoxide hydrolase as a therapeutic target for pain, inflammatory and neurodegenerative diseases

Pharmacology & therapeutics (Oxford), 2017-12, Vol.180, p.62-76

2017

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Interleukin-17 as a potential therapeutic target for chronic pain

Frontiers in immunology, 2022-09, Vol.13, p.999407-999407

2022

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TRP Channels and Pain

Annual review of cell and developmental biology, 2013-10, Vol.29 (1), p.355-384

Julius, David

2013

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PI3K/Akt Pathway: A Potential Therapeutic Target for Chronic Pain

Current pharmaceutical design, 2017-01, Vol.23 (12), p.1860-1868

2017

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3,5-Dicaffeoylquinic acid attenuates microglial activation-mediated inflammatory pain by enhancing autophagy through the suppression of MCP3/JAK2/STAT3 signaling

Biomedicine & pharmacotherapy, 2022-09, Vol.153, p.113549-113549, Article 113549

2022

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Central Sensitization: A Generator of Pain Hypersensitivity by Central Neural Plasticity

The journal of pain, 2009-09, Vol.10 (9), p.895-926

2009

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The emerging role of the BDNF-TrkB signaling pathway in the modulation of pain perception

Journal of neuroimmunology, 2020-12, Vol.349, p.577406-577406, Article 577406

2020

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CXCL1/CXCR2 signaling in pathological pain: Role in peripheral and central sensitization

Neurobiology of disease, 2017-09, Vol.105, p.109-116

2017

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The crucial role of NR2A mediating the activation of satellite glial cells in the trigeminal ganglion contributes to orofacial inflammatory pain during TMJ inflammation

Neuropharmacology, 2024-12, Vol.261, p.110173, Article 110173

2024

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Peripheral nitric oxide signaling directly blocks inflammatory pain

Biochemical pharmacology, 2020-06, Vol.176, p.113862-113862, Article 113862

2020

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TRPM8 is the principal mediator of menthol-induced analgesia of acute and inflammatory pain

Targeting the nitric oxide/cGMP signaling pathway to treat chronic pain

An Introduction to Pain Pathways and Pain ``Targets

AMPK activation attenuates inflammatory pain through inhibiting NF-κB activation and IL-1β expression

GABAB receptors and pain

Central and peripheral sites of action for CB 2 receptor mediated analgesic activity in chronic inflammatory and neuropathic pain models in rats

Macrophages transfer mitochondria to sensory neurons to resolve inflammatory pain

Kinins and their B1 and B2 receptors as potential therapeutic targets for pain relief

CircRNA expression profiling of the rat thalamus in temporomandibular joint chronic inflammatory pain

Cannabinoids, the endocannabinoid system, and pain: a review of preclinical studies

In vitro and in vivo characterization of A‐796260: a selective cannabinoid CB 2 receptor agonist exhibiting analgesic activity in rodent pain models

MEL endomorphins act as potent inflammatory analgesics with the inhibition of activated non-neuronal cells and modulation of pro-inflammatory cytokines

Epigenetic mechanisms of chronic pain

Cross-centre replication of suppressed burrowing behaviour as an ethologically relevant pain outcome measure in the rat: a prospective multicentre study

TRPA1 as a therapeutic target for nociceptive pain

Soluble epoxide hydrolase as a therapeutic target for pain, inflammatory and neurodegenerative diseases

Interleukin-17 as a potential therapeutic target for chronic pain

TRP Channels and Pain

PI3K/Akt Pathway: A Potential Therapeutic Target for Chronic Pain

3,5-Dicaffeoylquinic acid attenuates microglial activation-mediated inflammatory pain by enhancing autophagy through the suppression of MCP3/JAK2/STAT3 signaling

Central Sensitization: A Generator of Pain Hypersensitivity by Central Neural Plasticity

The emerging role of the BDNF-TrkB signaling pathway in the modulation of pain perception

CXCL1/CXCR2 signaling in pathological pain: Role in peripheral and central sensitization

The crucial role of NR2A mediating the activation of satellite glial cells in the trigeminal ganglion contributes to orofacial inflammatory pain during TMJ inflammation

Peripheral nitric oxide signaling directly blocks inflammatory pain

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