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PRRT2 Is a Key Component of the Ca2+-Dependent Neurotransmitter Release Machinery

Cell reports (Cambridge), 2016-04, Vol.15 (1), p.117-131

2016

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SYN1 loss-of-function mutations in autism and partial epilepsy cause impaired synaptic function

Human molecular genetics, 2011-06, Vol.20 (12), p.2297-2307

2011

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TBC1D24 regulates neuronal migration and maturation through modulation of the ARF6-dependent pathway

Proceedings of the National Academy of Sciences - PNAS, 2014-02, Vol.111 (6), p.2337-2342

2014

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The epilepsy-associated protein TBC1D24 is required for normal development, survival and vesicle trafficking in mammalian neurons

Human molecular genetics, 2019-02, Vol.28 (4), p.584-597

2019

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TBC1D24 regulates axonal outgrowth and membrane trafficking at the growth cone in rodent and human neurons

Cell death and differentiation, 2019-11, Vol.26 (11), p.2464-2478

2019

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Arf6 regulates the cycling and the readily releasable pool of synaptic vesicles at hippocampal synapse

eLife, 2016-01, Vol.5

2016

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SYN2 is an autism predisposing gene: loss-of-function mutations alter synaptic vesicle cycling and axon outgrowth

Human molecular genetics, 2014-01, Vol.23 (1), p.90-103

2014

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V-ATPase Dysfunction in the Brain: Genetic Insights and Therapeutic Opportunities

Cells (Basel, Switzerland), 2024-08, Vol.13 (17), p.1441

2024

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An interaction between PRRT2 and Na + /K + ATPase contributes to the control of neuronal excitability

Cell death & disease, 2021-03, Vol.12 (4), p.292-292, Article 292

2021

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TBC1D24, an ARF6-Interacting Protein, Is Mutated in Familial Infantile Myoclonic Epilepsy

American journal of human genetics, 2010-09, Vol.87 (3), p.365-370

2010

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Glutathione Limits Ero1-dependent Oxidation in the Endoplasmic Reticulum

The Journal of biological chemistry, 2004-07, Vol.279 (31), p.32667-32673

2004

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APache Is an AP2-Interacting Protein Involved in Synaptic Vesicle Trafficking and Neuronal Development

Cell reports (Cambridge), 2017-12, Vol.21 (12), p.3596-3611

2017

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Atmospheres: Feeling Architecture by Emotions

Ambiances (En ligne), 2019-12, Vol.5

2019

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PRRT2 Is a Key Component of the Ca 2+ -Dependent Neurotransmitter Release Machinery

Cell reports (Cambridge), 2016-04, Vol.15 (1), p.117-131

2016

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Formation, isomerisation and reduction of disulphide bonds during protein quality control in the endoplasmic reticulum

Histochemistry and cell biology, 2002-02, Vol.117 (2), p.151-157

2002

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On the way back from 3D to 2D: Chitosan promotes adhesion and development of neuronal networks onto culture supports

Carbohydrate polymers, 2022-12, Vol.297, p.120049-120049, Article 120049

2022

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Lesional and non-lesional epilepsies: A blurring genetic boundary

European journal of paediatric neurology, 2020-01, Vol.24, p.24-29

2020

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ATP6V1A is required for synaptic rearrangements and plasticity in murine hippocampal neurons

Acta Physiologica, 2024-08, Vol.240 (8), p.e14186-n/a

2024

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A micropatterned thermoplasmonic substrate for neuromodulation of in vitro neuronal networks

Acta biomaterialia, 2023-03, Vol.158, p.281-291

2023

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Ca2+ binding to synapsin I regulates resting Ca2+ and recovery from synaptic depression in nerve terminals

Cellular and molecular life sciences : CMLS, 2022-12, Vol.79 (12), Article 600

2022

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PRRT2 Is a Key Component of the Ca(2+)-Dependent Neurotransmitter Release Machinery

Cell reports (Cambridge), 2016-04, Vol.15 (1), p.117-131

2016

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REST/NRSF-mediated intrinsic homeostasis protects neuronal networks from hyperexcitability

The EMBO journal, 2013-11, Vol.32 (22), p.2994-3007

2013

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Synapsins: From synapse to network hyperexcitability and epilepsy

Seminars in cell & developmental biology, 2011-06, Vol.22 (4), p.408-415

2011

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Novel Compound Heterozygous Mutations in TBC1D24 Cause Familial Malignant Migrating Partial Seizures of Infancy

Human mutation, 2013-06, Vol.34 (6), p.869-872

2013

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TBC1D24 genotype-phenotype correlation: Epilepsies and other neurologic features

Neurology, 2016-07, Vol.87 (1), p.77-85

2016

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PRRT2 Is a Key Component of the Ca2+-Dependent Neurotransmitter Release Machinery

SYN1 loss-of-function mutations in autism and partial epilepsy cause impaired synaptic function

TBC1D24 regulates neuronal migration and maturation through modulation of the ARF6-dependent pathway

The epilepsy-associated protein TBC1D24 is required for normal development, survival and vesicle trafficking in mammalian neurons

TBC1D24 regulates axonal outgrowth and membrane trafficking at the growth cone in rodent and human neurons

Arf6 regulates the cycling and the readily releasable pool of synaptic vesicles at hippocampal synapse

SYN2 is an autism predisposing gene: loss-of-function mutations alter synaptic vesicle cycling and axon outgrowth

V-ATPase Dysfunction in the Brain: Genetic Insights and Therapeutic Opportunities

An interaction between PRRT2 and Na + /K + ATPase contributes to the control of neuronal excitability

TBC1D24, an ARF6-Interacting Protein, Is Mutated in Familial Infantile Myoclonic Epilepsy

Glutathione Limits Ero1-dependent Oxidation in the Endoplasmic Reticulum

APache Is an AP2-Interacting Protein Involved in Synaptic Vesicle Trafficking and Neuronal Development

Atmospheres: Feeling Architecture by Emotions

PRRT2 Is a Key Component of the Ca 2+ -Dependent Neurotransmitter Release Machinery

Formation, isomerisation and reduction of disulphide bonds during protein quality control in the endoplasmic reticulum

On the way back from 3D to 2D: Chitosan promotes adhesion and development of neuronal networks onto culture supports

Lesional and non-lesional epilepsies: A blurring genetic boundary

ATP6V1A is required for synaptic rearrangements and plasticity in murine hippocampal neurons

A micropatterned thermoplasmonic substrate for neuromodulation of in vitro neuronal networks

Ca2+ binding to synapsin I regulates resting Ca2+ and recovery from synaptic depression in nerve terminals

PRRT2 Is a Key Component of the Ca(2+)-Dependent Neurotransmitter Release Machinery

REST/NRSF-mediated intrinsic homeostasis protects neuronal networks from hyperexcitability

Synapsins: From synapse to network hyperexcitability and epilepsy

Novel Compound Heterozygous Mutations in TBC1D24 Cause Familial Malignant Migrating Partial Seizures of Infancy

TBC1D24 genotype-phenotype correlation: Epilepsies and other neurologic features

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