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Microbial Persistence and the Road to Drug Resistance

Cell host & microbe, 2013-06, Vol.13 (6), p.632-642

2013

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Bactericidal Antibiotics Induce Toxic Metabolic Perturbations that Lead to Cellular Damage

Cell reports (Cambridge), 2015-11, Vol.13 (5), p.968-980

2015

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Carbon Sources Tune Antibiotic Susceptibility in Pseudomonas aeruginosa via Tricarboxylic Acid Cycle Control

Cell chemical biology, 2017-02, Vol.24 (2), p.195-206

2017

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Antibiotics induce redox-related physiological alterations as part of their lethality

Proceedings of the National Academy of Sciences - PNAS, 2014-05, Vol.111 (20), p.E2100-E2109

2014

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A flexible model of HIV-1 latency permitting evaluation of many primary CD4 T-cell reservoirs

PloS one, 2012-01, Vol.7 (1), p.e30176-e30176

2012

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Bacterial Metabolism and Antibiotic Efficacy

Cell metabolism, 2019-08, Vol.30 (2), p.251-259

2019

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Understanding and Sensitizing Density-Dependent Persistence to Quinolone Antibiotics

Molecular cell, 2017-12, Vol.68 (6), p.1147-1154.e3

2017

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Inhibition of both HIV-1 reverse transcription and gene expression by a cyclic peptide that binds the Tat-transactivating response element (TAR) RNA

PLoS pathogens, 2011-05, Vol.7 (5), p.e1002038

2011

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A Quantitative Affinity-Profiling System That Reveals Distinct CD4/CCR5 Usage Patterns among Human Immunodeficiency Virus Type 1 and Simian Immunodeficiency Virus Strains

Journal of Virology, 2009-11, Vol.83 (21), p.11016-11026

2009

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SARS-CoV-2 seroprevalence in healthcare workers of a Swiss tertiary care centre at the end of the first wave: a cross-sectional study

BMJ open, 2021-07, Vol.11 (7), p.e049232

2021

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Engineered Phagemids for Nonlytic, Targeted Antibacterial Therapies

Nano letters, 2015-07, Vol.15 (7), p.4806-4811

2015

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HIV-1 Entry, Inhibitors, and Resistance

Viruses, 2010-05, Vol.2 (5), p.1069-1105

2010

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HIV-1 replicative fitness in elite controllers

Current opinion in HIV & AIDS, 2011-05, Vol.6 (3), p.214-220

2011

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Multifaceted Mechanisms of HIV Inhibition and Resistance to CCR5 Inhibitors PSC-RANTES and Maraviroc

Antimicrobial Agents and Chemotherapy, 2013-06, Vol.57 (6), p.2640-2650

2013

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Differences in the Fitness of Two Diverse Wild-Type Human Immunodeficiency Virus Type 1 Isolates Are Related to the Efficiency of Cell Binding and Entry

Journal of Virology, 2005-06, Vol.79 (11), p.7121-7134

2005

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Natural Variation in the V3 Crown of Human Immunodeficiency Virus Type 1 Affects Replicative Fitness and Entry Inhibitor Sensitivity

Journal of Virology, 2007-08, Vol.81 (15), p.8258-8269

2007

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Identification of Two APOBEC3F Splice Variants Displaying HIV-1 Antiviral Activity and Contrasting Sensitivity to Vif[S]

The Journal of biological chemistry, 2010-09, Vol.285 (38), p.29326-29335

2010

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Increased energy demand from anabolic-catabolic processes drives β-lactam antibiotic lethality

Cell chemical biology, 2022-02, Vol.29 (2), p.276-286.e4

2022

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Author Correction: A novel antibiotic class targeting the lipopolysaccharide transporter

Nature (London), 2024-07, Vol.631 (8022), p.E17-E17

2024

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Targets of Small Interfering RNA Restriction during Human Immunodeficiency Virus Type 1 Replication

Journal of Virology, 2008-03, Vol.82 (6), p.2938-2951

2008

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Deep Gene Sequence Cluster Analyses of Multi-Virus-Infected Mucosal Tissue Reveal Enhanced Transmission of Acute HIV-1

Journal of virology, 2021-01, Vol.95 (3)

2021

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Elite suppressor-derived HIV-1 envelope glycoproteins exhibit reduced entry efficiency and kinetics

PLoS pathogens, 2009-04, Vol.5 (4), p.e1000377-e1000377

2009

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Antibiotics induce redox-related physiological alterations as part of their lethality

Proceedings of the National Academy of Sciences - PNAS, 2014-05, Vol.111 (20), p.7174-7174

2014

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Elite SuppressoraDerived HIV-1 Envelope Glycoproteins Exhibit Reduced Entry Efficiency and Kinetics

PLoS pathogens, 2009-04, Vol.5 (4), p.e1000377-e1000377

2009

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Elite Suppressor-Derived HIV-1 Envelope Glycoproteins Exhibit Reduced Entry Efficiency and Kinetics: e1000377

PLoS pathogens, 2009-04, Vol.5 (4)

2009

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Microbial Persistence and the Road to Drug Resistance

Bactericidal Antibiotics Induce Toxic Metabolic Perturbations that Lead to Cellular Damage

Carbon Sources Tune Antibiotic Susceptibility in Pseudomonas aeruginosa via Tricarboxylic Acid Cycle Control

Antibiotics induce redox-related physiological alterations as part of their lethality

A flexible model of HIV-1 latency permitting evaluation of many primary CD4 T-cell reservoirs

Bacterial Metabolism and Antibiotic Efficacy

Understanding and Sensitizing Density-Dependent Persistence to Quinolone Antibiotics

Inhibition of both HIV-1 reverse transcription and gene expression by a cyclic peptide that binds the Tat-transactivating response element (TAR) RNA

A Quantitative Affinity-Profiling System That Reveals Distinct CD4/CCR5 Usage Patterns among Human Immunodeficiency Virus Type 1 and Simian Immunodeficiency Virus Strains

SARS-CoV-2 seroprevalence in healthcare workers of a Swiss tertiary care centre at the end of the first wave: a cross-sectional study

Engineered Phagemids for Nonlytic, Targeted Antibacterial Therapies

HIV-1 Entry, Inhibitors, and Resistance

HIV-1 replicative fitness in elite controllers

Multifaceted Mechanisms of HIV Inhibition and Resistance to CCR5 Inhibitors PSC-RANTES and Maraviroc

Differences in the Fitness of Two Diverse Wild-Type Human Immunodeficiency Virus Type 1 Isolates Are Related to the Efficiency of Cell Binding and Entry

Natural Variation in the V3 Crown of Human Immunodeficiency Virus Type 1 Affects Replicative Fitness and Entry Inhibitor Sensitivity

Identification of Two APOBEC3F Splice Variants Displaying HIV-1 Antiviral Activity and Contrasting Sensitivity to Vif[S]

Increased energy demand from anabolic-catabolic processes drives β-lactam antibiotic lethality

Author Correction: A novel antibiotic class targeting the lipopolysaccharide transporter

Targets of Small Interfering RNA Restriction during Human Immunodeficiency Virus Type 1 Replication

Deep Gene Sequence Cluster Analyses of Multi-Virus-Infected Mucosal Tissue Reveal Enhanced Transmission of Acute HIV-1

Elite suppressor-derived HIV-1 envelope glycoproteins exhibit reduced entry efficiency and kinetics

Antibiotics induce redox-related physiological alterations as part of their lethality

Elite SuppressoraDerived HIV-1 Envelope Glycoproteins Exhibit Reduced Entry Efficiency and Kinetics

Elite Suppressor-Derived HIV-1 Envelope Glycoproteins Exhibit Reduced Entry Efficiency and Kinetics: e1000377

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